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Fear Of Crime
Wikipedia
Journal of Quantitative Criminology, 3, 29-46. ^ Perkins, D. & Taylor, R. (1996). Ecological assessments of community disorder: Their relationship to fear of crime and theoretical implications. American Journal of Community Psychology, 24, 63-107. ^ Wyant, B.R. (2008). ... Aldershot: Avebury. ^ Innes, M. (2004) Signal crimes and signal disorders: Notes on deviance as communicative action. British Journal of Sociology, 55, 317-334. ^ Tulloch, M. (2003). ... option=com_content&view=article&id=59%3Aexperience-and-communication-as-factors-explaining-criminal-risk-perception&catid=56%3Acat-crime-perception&Itemid=92&lang=de [ permanent dead link ] ^ Winkel, F. ... Other references [ edit ] Vilalta, C. (2010). "Fear of crime in gated communities and apartment buildings: a comparison of housing types and a test of theories".
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Hyperlexia
Omim
Silberberg and Silberberg (1967) defined the term 'hyperlexia' to describe children who read at levels beyond those expected for their mental age in the face of disordered oral communication. Turkeltaub et al. (2004) stated that there are 3 consistent features of hyperlexia: the presence of a developmental disorder of communication, most often an autistic spectrum disorder (209850); acquisition of reading skills prior to age 5 years without explicit instruction; and advanced word recognition ability relative to mental age, with reading comprehension on par with verbal ability. ... INHERITANCE - Autosomal recessive - Isolated cases - Multifactorial NEUROLOGIC Behavioral Psychiatric Manifestations - Developmental disorder of communication - Autism ( 209850 ) - Pervasive developmental disorder - Precocious reading - Reading at level beyond mental age - Advanced word recognition - Increased phonologic processing - Reading comprehension on par with verbal abilities - Functional MRI (fMRI) shows increased activity in the left inferior frontal cortex - Increased activity in the left posterior superior temporal cortex - Increased activity in the right inferior temporal cortex MISCELLANEOUS - Onset before age 5 years in the absence of instruction ▲ Close
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Autism Spectrum Disorder
Medlineplus
Autism spectrum disorder (ASD) is a condition that appears very early in childhood development, varies in severity, and is characterized by impaired social skills, communication problems, and repetitive behaviors. ... Inability to recognize and use these cues makes it hard for affected individuals to understand the feelings of others or communicate their own feelings appropriately. ... However, the condition is usually diagnosed between ages 2 and 4, when more advanced communication and social skills, such as learning to play with others, typically begin to develop. ... For example, autistic disorder was a term that was used when affected individuals had limited or absent verbal communication, often in combination with intellectual disability. ... These abnormalities are thought to underlie the differences in socialization, communication, and cognitive functioning characteristic of ASD.CHD8, NRXN1, TBR1, SHANK1, PAX6, MECP2, NLGN3, ADNP, UBE3A, NLGN4X, FMR1, MAOA, SHANK3, PTEN, CNTNAP2, IL1RAPL1, TMLHE, CACNA1C, TSC2, KDM5C, SYNGAP1, CHRNA7, PTCHD1, GRIN2B, MACROD2, COMT, ADSL, JMJD1C, IFNG, CSDE1, NTRK2, NSD1, SNRPN, GABRB3, OXTR, MTHFR, BDNF, EN2, SLC25A12, DPYD, SLC6A4, TAF1C, RELN, AVP, FOXP2, DHCR7, BRSK2, MET, RBFOX1, HOXA1, IL6, AVPR1A, GRIK2, DISC1, ITGB3, WNT2, PRKN, NLGN1, GAD1, GLO1, HTR2A, HLA-DRB1, NBEA, NRCAM, NF1, DOCK4, CADPS2, NOS2, ATP10A, AR, ADA, MAPK3, AHI1, CNTN4, ASMT, SHANK2, TPH2, SEMA5A, POMC, LEP, ST8SIA2, ITGA4, PRKCB, MAGEL2, DAB1, GABRA5, NRP2, GABRA4, GABRB1, NRXN3, IMMP2L, LAMB1, DRD3, OXT, SCT, BCL2, TSC1, NRXN2, IL10, SLC9A9, EIF4E, SND1, DLX2, PIK3CG, DRD1, DLGAP2, SERPINE1, IGF1, EGF, HRAS, MEF2C, DIPK2A, TBL1XR1, C4B, RPL10, STK39, PTGS2, AGAP1, MARK1, PRL, STX1A, PON1, EXT1, CACNA1H, GRIA1, GPX1, GRM8, KDM5B, VLDLR, GABBR1, VIP, CADM1, FARP2, GRIN2A, GSTM1, UBE2H, RIMS1, SEZ6L2, HLA-A, VWA8, TRRAP, HOXD11, PASK, EGR2, HDLBP, ZMYND11, GJA1, ROBO3, AQP4, ABAT, KCND2, PCDH10, PITX1, MYO1D, SLC19A1, CDH10, PLA2G4A, CDH9, STXBP6, SCAMP5, SLC1A3, LASP1, MAOB, HTR3C, APC, MIF, RORA, DIXDC1, ROBO2, ROBO1, CSMD3, RAB39B, CHRNA4, CHRNB2, CYP19A1, ROBO4, TNFRSF1B, DHFR, NPAS2, ACE, ASIC2, SNTG2, REEP3, HTR3A, DLX1, ADRB2, PDE4A, IL4, IL5, TDO2, GABBR2, CP, PDE4B, PECAM1, PER1, HTR5A, PRF1, NTF4, RNF8, NR4A2, CNTN3, PLAUR, MBD4, HTR1D, PRLR, MTF1, HTR1B, HTN1, IGF2, IGFBP3, TF, IL1RN, IL2, IL13, IL15, XDH, XPC, INPP1, KCNMA1, LZTR1, GZMB, GSTP1, SLC6A8, SCN7A, MCC, TCN2, STATH, SLC40A1, KIF1A, GABRA2, GABRA1, COP1, JAKMIP1, GPR155, POU6F2, DAO, BTD, NTF3, ZNF292, KAT8, KMT2E, ASTN2, CALCA, CAT, CHD4, GABRA3, RAB11FIP5, AGO1, ZNF713, GHR, DLL1, ACADSB, MIR23A, MIR106B, ADM, CUL7, WDFY4, PLD5, MBD3, UPB1, FBXO40, AUTS2, PIANP, FOXP1, SCN2A, CTNND2, CDKL5, EHMT1, MED12, ANKRD11, STS, STXBP1, CREBBP, GTF2I, UPF3B, SLC1A2, AKT1, BCKDK, PIK3CA, GABRD, RAI1, RAC1, CHD7, TRIM33, CACNA1A, HNF1B, ATRX, SCN1A, DHDDS, SCN8A, LIMK1, SOX2, PAH, OPHN1, MACROD2-AS1, HDAC8, GABRB2, GABRG2, SATB2, CLIP2, EP300, ELN, SLC9A6, CYFIP2, IQSEC2, PPP3CA, MAN1B1, PPL, PRKAR1A, HESX1, IRAK1BP1, PROKR2, PARD3B, ZNF827, NUS1, RLIM, SYNJ1, CNKSR2, TRAK1, TMEM132B, KCNAB2, RAD21, REV3L, RFC2, DNER, NAV3, FOXN1, TRAF7, DHX30, RREB1, SORCS1, FLCN, POLG, ARNT2, SCN1A-AS1, SLC44A3-AS1, SEMA3E, RPH3AL, PIGL, OTX2, AUTS1, KLLN, PKMP1, LIPG, HDAC4, SLC23A2, SEC23B, SMC3, HCN1, PC, FLG-AS1, LAMA1, PDE4D, SLC13A5, BAZ1B, SDK1, SLC39A11, PKP2, RYR1, RASSF5, SMC1A, PLXND1, SOX3, SOX11, PRR12, FMN2, SIN3A, GTF2IRD1, SH2B1, YEATS2, TBX1, TCF20, SLC22A15, PARS2, SLC35C1, SETD5, PHIP, CDKAL1, NECAP1, DYM, TMCO1, UFD1, TBL2, WWOX, HIRA, FAM135B, TRPS1, NIPBL, YWHAG, NUFIP2, SDHD, ACTL6B, SCN3A, SYNE2, UBA5, AP3B2, ALG13, SZT2, SRD5A3, ALDH5A1, SDHB, SDHC, ALMS1, PCGF2, SIM1, SKI, GALNT14, GNPTAB, MED13L, PCDH15, ZBTB16, ARV1, GSPT2, PRDM16, SLC22A3, SEC24C, AARS1, COX3, TRNQ, COL4A2, COX1, COL6A1, COX2, ARSD, ND1, ND4, FLG, ND5, FGFR1, ND6, FGF14, TRNF, FGF12, TRNH, FER, FBN1, BRCA2, MSNP1, CLTC, MEIS2, CAMK2G, GRIN2D, LHX1, CACNA1B, GP1BB, ALDH1A3, GJB2, GJA8, RERE, GJA5, KCNB1, CHRNA1, GATM, KMT2A, KCNA2, GAMT, TRNL1, GUCY1A2, TRNS1, NDP, ATP6V1A, DNM1, EEF1A2, ARVCF, BPTF, NHS, ERBB4, ERG, NELL1, NONO, NAGLU, TRNS2, NAGA, TRNW, CD38, CYFIP1, HTC2, APP, DNTT, MUL1, CBLL2, IL17A, TNF, ARID1B, MBD1, DYRK1A, NOS1, MRC1, MTOR, PVALB, FRAXA, VDR, GRPR, ADORA2A, NCS1, APOE, NLGN4Y, FMR1-IT1, ZNF804A, SYN1, PCDH19, CARS1, RSS, LOC110806262, SMS, SMARCA1, MTR, DBH, GABRG3, DDC, SLC18A1, GRM5, ARX, HOXB1, DRD4, RBM45, AUTS5, DLG4, NCAM1, RAPGEF4, YWHAE, GFAP, GRIP1, TP53, TRIO, TPH1, VIPR2, PCLO, HTT, CACNA1D, HTR2C, CAPS2, KATNAL2, HLA-G, HLA-B, HLA-DPB1, NRAS, CEP290, DLX5, PHB2, CC2D1A, FAAH, DLD, DLG3, ESR2, DLX6, GRIK4, EPHB2, IL22, ZBTB20, IL1RAPL2, DRD2, DPP4, FAM120C, AMBRA1, MBD5, CNR2, GAD2, GPHN, ABCB6, RIMS3, ARHGAP32, CLOCK, CHAT, NLGN2, GLUL, CDH13, CELF6, TOP3B, WASF1, ATP2B2, DNMT1, MIR137, PIK3CD, SOAT1, PAFAH1B1, RIT2, ANK3, SMARCA4, PTPRZ1, LRFN5, SLC12A2, HEPACAM, SNAP25, PIK3CB, PTLS, ACAN, APRT, ACTB, NDN, CLIP1, AMPD1, TCF4, TAC1, MFAP1, CADM2, MAPK1, MFGE8, NIPA1, MEF2A, OPRM1, MTRR, NCAM2, NEGR1, CCL2, ADCY1, CRP, LSG1, CTSB, MIR137HG, MIR124-1, TESC, CTLA4, CTNNB1, SLC52A1, NOTCH2NLA, CSF2, QRSL1, CS, RPP25, C4orf3, TADA1, CHDS3, PCDHB4, CHPT1, ANKS1B, CELF4, SPHK2, CLK2, SLC2A4RG, RETN, CTNNBL1, HCA1, ANKH, NSMCE3, AGTR2, DNAAF3, MYDGF, CMAS, AGT, AGRP, AGER, EAF2, PAG1, ARSI, CNTF, FCRL6, ZNF407, WDR11, CYP1B1, CTSD, HYDIN, ACACA, DMD, FOXP3, TAS2R1, TAS2R3, LINC01672, IL21R, NT5C, DEL11P13, VSX1, NOTCH2NLC, NOTCH2NLB, ABO, MYLPF, ABCA3, STRN4, THRIL, DNASE1L3, CTNNA3, PYCARD, DYNC1H1, LOC102724334, SETD2, THRA1/BTR, MCTS1, SYNSTH, ADHD6, ACHE, MIR486-1, CUX1, LRRN3, GNB1L, ADARB1, CYP2D6, BDNF-AS, CYP11B1, H4C15, CYP21A2, DAB2, TREM2, DCUN1D1, WAC, H2BS1, IL17D, ADA2, ZNF674, DECR1, SCLY, MIR557, ZGLP1, VCX3A, ACP1, SPG21, PCBP4, FOXK1, SENP7, ATP13A4, C5, SIK1, H2BC12, COMMD1, ZNF385B, EBPL, NTNG2, KIRREL3, TMEM185A, SRRM4, MEGF10, RNF135, PPP1R1B, RPS10P2, UPP2, CTTNBP2, FERMT3, FAM107B, CACNB2, TTC25, PCDH11Y, APBA2, NETO1, DIAPH3, NDEL1, STMN4, BST1, AREG, AS3MT, SYTL4, ASTN1, ASL, OMA1, H4-16, AVPR2, TUBGCP5, ADGRB3, MBD6, AGBL1, PGLYRP2, CEP41, DUSP15, TNFRSF17, RHOT2, XIRP2, OPN1SW, HTR7P1, CNTNAP5, BMPR2, SNX29, FAM43A, CHURC1, NOBOX, UBXN11, ARNTL, DOCK11, SPX, SRCIN1, ABCA13, TSHZ3, CLSTN2, CDH11, AMY1B, AMY1A, RIC8A, PPCDC, IL21, ZNF410, FBXO15, LRRC4C, CDH15, VPS18, PREX1, CPED1, CDK5, ATOH7, CECR, LRFN2, MRTFB, ALDH2, ALB, ATD, IL27, CHRM3, KCTD13, SLC12A5, CDH8, TOR3A, CDH7, CD69, CALB1, CALB2, VPS13B, CAST, LINGO2, MCPH1, CASP3, SH3TC2, SERPINH1, SLC35G1, CARS2, CBS, FA2H, MLPH, CUEDC2, DNAAF4, VKORC1, WNK3, CD5L, ANK2, CD40LG, SIL1, ANK1, ZNF778, AMY1C, DAGLA, WDFY3, DESI1, IL1B, IL18, IL16, IL9, CXCL8, TERF1, TERT, TG, SYP, TGFB1, TH, THBS1, THRA, TLL1, TSPAN4, TAL1, SYN2, TPTE, SRC, KCNJ10, SOD1, SOD2, SOS1, KCNJ1, SP1, SRY, INSR, SST, SSTR5, STAT3, ITIH3, ITGAV, ITGA6, TNC, TRH, SNCG, RASSF7, PXDN, HGF, HFE, FZD3, SEM1, GLRA3, HCCS, LRP8, AKAP1, BAS, DGCR6, H2AZ1, USP9X, H1-4, NRG1, UBE2K, TRPC6, HP, HTR7, HTR4, HTR1A, TXN, HPD, UCP3, UROD, MKRN3, HOXD@, HMGN1, WT1, HLA-DPA1, XPO1, HLA-DOA, SNRNP70, SNCB, DNMT3A, PPP2R5D, NDUFA5, PENK, PIGH, PLP1, PPARD, PPARG, MAPK7, PCDH9, PRODH, MTHFD1, PSD, PTPRF, PURA, RAB3A, NEDD4, NEUROG1, RARA, NPTX2, NTS, NUP98, NPY1R, OCRL, OGG1, OMG, OTX1, NFE2L2, NPY, NOTCH1, PCSK6, NFKBIL1, NFIC, PAX3, MT3, RASGRF1, SMARCA2, LAMC2, LTA, LMX1B, SHMT1, SHMT2, LEPR, SLC1A1, L1CAM, SDC2, SLC6A3, KRT7, KIR3DL1, SLC12A3, KIR2DS1, KCNQ2, MAFD2, CXCL5, OPN1LW, RORC, REN, UPF1, MT1A, RFC1, MOV10, MMP9, RYR2, CCL22, RYR3, S100B, MEST, MBP, MAP2, CCL17, GUK1, H4C9, H4C1, FGFR2, CHP1, FOLR1, DLGAP4, NTNG1, FOLH1, FLT4, FAN1, FOSL2, NT5C2, MAST1, NPY5R, MYT1L, SETD1B, KDM4C, DCTN3, PTENP1, FAP, CHL1, ATP8A1, DEAF1, KAT5, GNB5, GABRG1, GABPA, XRCC6, ZNF277, LYVE1, EHD1, RAB40B, SLC27A4, SLC27A3, SPACA9, POGZ, PHF8, H4C4, GREM1, ELAVL2, PLEKHG3, SETBP1, EIF4EBP2, EBF1, DSCAM, COPG2, ENO2, AKAP8L, DPYSL2, ACAD8, DPP6, AGO2, CNTN6, ELK3, PART1, DIP2A, SYNM, ACSL4, ARHGEF9, FABP7, MCF2L, CUX2, F2RL1, CRTC1, EPS8, EZH2, SLC35A3, MAPK8IP2, PSD4, SH3BP4, SHC2, SEMA3A, AKR1A1, BCAS2, AP1S2, MPDZ, BANF1, GRM7, GRM1, CCN6, NR1I2, GRIN1, KLF7, CACNA1G, GRIK1, GRID2, GRIA2, GPR37, PKD2L1, EIF3A, GRP, AKAP8, H4C5, H4C6, H4C12, H4C11, H4C3, H4C8, H4C2, H4C13, CASK, H4C14, GNPAT, CUL3, GSK3B, CNTNAP1, DENR, GPR35, ARTN, ARHGEF2, GCG, BMS1, PTDSS1, RNF40, GEM, GDNF, SETDB1, RBM8A, XPR1, GATA4, SLC12A6, AKT3, RANBP9, TSPAN32, SPRY3, GH1, GLDC, MACROH2A1, GLP1R, MAGED1, GLRA2, HOMER1, ABCG2, GLRB, GLS, ADIPOQ, GMDS, LHX2, TAOK2, TRIP12, CYTH1, GOT2, STIN2-VNTR
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Body Inflation
Wikipedia
The notion of the fantasy scenarios ending in popping or explosion is often a divisive topic in the community. [5] The first inflatable fetish community organized online in 1994, in the form of an e-mail list; as the popularity of online communication grew, so did the online community. [5] See also [ edit ] Balloon fetish Breast fetishism Fat fetishism Alvinolagnia Inflatable doll Pregnancy fetishism References [ edit ] ^ The story of Mr.
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Receptive Aphasia
Wikipedia
National Institute on Deafness and Other Communication Disorders (NIDCD) . ^ a b "Aphasia: Roles and responsibilities" . ... San Antonio, TX: Pearson. ^ Holland, A.L.; Fromm, D.; Wozniak, L. (2018). Communication Activities in Daily Living (CADL-3) (3rd ed.). ... "Neuroplasticity: Implications for treating cognitive communication disorders". ASHA National Convention . ^ a b Thomson, C.K. (2000). "Neuroplasticity: Evidence from aphasia" . Journal of Communication Disorders . 33 (4): 357–366. doi : 10.1016/S0021-9924(00)00031-9 . ... PMID 15339233 . ^ a b c d e f g Manasco, H. (2021). Introduction to neurogenic communication disorders . Burlington, Massachusetts: Jones & Bartlett Learning. ^ a b LaPointe, L. (2005).
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Bacteremia, Susceptibility To, 1
Omim
The minimal annual incidence of community-acquired bacteremia was estimated at 1,457 cases per 100,000 children among infants under 1 year old, 1,080 among children under 2 years old, and 505 among children under 5 years old. Of all in-hospital deaths, 26% were in children with community-acquired bacteremia. Of 308 deaths in children with bacteremia, 103 (33.4%) occurred on the day of admission and 217 (70.5%) occurred within 2 days. Berkley et al. (2005) concluded that community-acquired bacteremia is a major cause of death among children at a rural sub-Saharan hospital.
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Female Genital Mutilation In New Zealand
Wikipedia
The procedures may have negative health impacts by causing problems such as urinating, infections, severe bleeding and complications during childbirth. [1] Procedures are normally carried out on girls from infancy to 15 years old. [1] FGM is predominantly practiced in countries throughout the Middle East, Asia and in Africa. [1] Contents 1 Background 2 Prevalence 3 International obligations 4 Domestic measures 5 References 6 External links Background [ edit ] FGM is a cultural tradition, based upon the belief that having FGM performed upon young girls is in their best interests. [2] Despite the first known documentation of FGM being recorded in Egypt in 25BC, it is thought that the practice of FGM has been occurring for some centuries prior to this. [3] While FGM practices differ between communities, the theory among them remains relatively similar. That is that women are more attractive prospects for marriage when they have had the FGM procedure and in some regions it is considered a necessary part of a girl’s social development. [2] Where the most severe forms of FGM are practiced (such as where infibulation is performed – this is the narrowing or sealing off of the vaginal opening) [1] women are prevented from engaging in any promiscuity prior to marriage, thus making them more desirable. [2] It is further believed that FGM saves women from temptation and safeguards the morality of women. [2] In some communities where FGM is a tradition, misconceptions regarding the benefits of FGM have been passed through generations. Some beliefs include that FGM: promotes fertility, is an essential measure to ensure good health of the baby and the cleanliness of a woman and that the clitoris is a feature of masculinity and therefore FGM procedures create a smooth skin surface that is more desirable. [2] Prevalence [ edit ] There have been no cases of FGM taken under the New Zealand legislation, although it is known that migrants from countries such as Somalia have undergone FGM procedures before entering New Zealand. [4] The communities that are predominantly affected by FGM are Ethiopian, Sudanese, Eritrean and Somali and many women in these communities have undergone FGM prior to arriving in New Zealand. [5] The most affected group is the Somali community, where 98% of women have already undergone FGM prior to migrating to New Zealand. [5] FGM is also practiced in some Muslim communities in Malaysia, India and Indonesia and some women and girls from these communities, living in New Zealand, may have also undergone FGM. [5] International obligations [ edit ] New Zealand has a good history of ratifying and complying with international law instruments. [6] A number of these instruments refer to or impact upon New Zealand and the domestic laws surrounding FGM. ... However, the New Zealand Health Strategy 2000 referred to programmes relating to FGM, including the need to address refugee communities who practice FGM. [20] In 1997, in response to the increasing number of women and girls settling in New Zealand from countries which practice FGM, a community based programme was established to provide improved health care services for women affected by FGM. [21] This programme is funded by the Ministry of Health and aims to provide education on FGM and support for those affected by FGM. [21] As the result of a study carried out in 2008, the New Zealand FGM Programme developed guidelines for health care professionals. ... The process includes the following steps; a health professional who knows the family is contacted, as well as a community worker (depending on the ethnicity of the child and their family), and engagement of an interpreter is considered. [22] Engagement with the family is conducted in a sensitive manner to allow the family to retain their dignity.
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Mn1 C-Terminal Truncation Syndrome
Gene_reviews
While some affected individuals may experience frustration due to poor verbal communication, no consistent behavior problems have been reported. ... W/emphasis on alternative (nonverbal) communication Speech & language Alternative communication as needed Epilepsy Standardized treatment w/AEDs by experienced neurologist Many AEDs may be effective; none demonstrated effective specifically for this disorder. ... See also Hereditary Hearing Loss and Deafness Overview. Community hearing services through early intervention or school district Cardiovascular Per treating cardiologist Family/Community Ensure appropriate social work involvement to connect families w/local resources, respite, & support. ... When feeding dysfunction is severe, an NG-tube or G-tube may be necessary. Communication issues. Consider evaluation for alternative means of communication (e.g., Augmentative and Alternative Communication [AAC]) for individuals who have expressive language difficulties. ... The evaluation will consider cognitive abilities and sensory impairments to determine the most appropriate form of communication. AAC devices can range from low-tech, such as picture exchange communication, to high-tech, such as voice-generating devices.
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Hiv/aids In Canada
Wikipedia
It is discovered that a single dose of nevirapine is effective in reducing mother-to-child transmission during pregnancy. 2000: The United Nations Secretary General creates the Global Fund to Fight HIV/AIDS, Tuberculosis, and Malaria 2001: The National Aboriginal Council on HIV/AIDS is established to identify and address shared Indigenous community and Public Health Agency of Canada priorities and provide advice informed by community knowledge, lived experience and relevant practice specific to the needs of Indigenous peoples. 2002: HIV becomes the leading cause of death worldwide in people aged 15–59 years. 2003: The WHO announces the “3 by 5” Initiative to bring treatment to 3 million people worldwide by 2005. ... The Canadian Institutes for Health Research (CIHR) announces funding to support the Aboriginal HIV and AIDS Community-Based Research Collaborative Centre and the REACH CBR Collaborative Centre in HIV/AIDS. 2013: The Quebec Ministry of Health publishes the first Canadian interim guidance on PrEP. ... Prevention and Support [ edit ] In 2019, the Government of Canada invested $22.85 million in HIV/AIDS research through the Canadian Institutes for Health Research. [7] This money went towards supporting a thriving community of researchers, people living with HIV and their caregivers, health advocates, and pharmaceutical and biotechnology companies to generate knowledge about prevention, treatment, management, and a cure for HIV/AIDS and other sexually transmitted and blood-borne infections. [8] Many organizations have been instituted by Indigenous peoples to combat the elevated rates of HIV/AIDS in Indigenous communities. ... Using a community-based approach, the unique needs of Indigenous communities in Canada can be addressed in a meaningful and respectful way that upholds Indigenous tradition and culture. ... "Addressing HIV/AIDS among Aboriginal People using a Health Status, Health Determinants and Health Care Framework: A Literature Review and Conceptual Analysis" . Canadian Journal of Aboriginal Community-based HIV/AIDS Research . 3 : 35–46.
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Syringomyelia, Noncommunicating Isolated
Omim
The form of syringomyelia discussed here is 'noncommunicating' with the fourth ventricle, but may communicate with the subarachnoid space. In contrast, 'communicating' syringomyelia, or 'hydromelia,' opens rostrally into the fourth ventricle and almost always occurs in children with hydrocephalus, Chiari malformation type II (CM2; 207950), and spina bifida (see 182940) (Levine, 2004). ... Pathogenesis Gardner (1965) proposed a hydrodynamic mechanism to explain the development of communicating syringomyelia based on abnormal delayed opening of the roof of the fourth ventricle, persistence of the connection between the fourth ventricle and the spinal canal, and high CSF pressure pulsations exerted by the choroid plexus. ... The first type (47 cases) encompassed dilations of the central canal that communicated directly with the fourth ventricle ('hydromelic') and were associated with hydrocephalus and severe birth defects, such as Chiari type II malformation and open spina bifida. ... The third group (35 cases) included extracanalicular parenchymal syrinxes that did not communicate with the central canal and were associated with injurious spinal cord lesions, such as trauma, infarction, or hemorrhage.
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Specific Developmental Disorder
Wikipedia
Specific developmental disorder Specialty Psychiatry Specific developmental disorders ( SDD ) was a classification of disorders characterized by delayed development in one specific area or areas. [1] [2] [3] [4] Specific developmental disorders were contrasted to pervasive developmental disorders [4] which were characterized by delays in the development of multiple basic functions including socialization and communication . [5] Contents 1 ICD-10 taxonomy 2 DSM-III taxonomy 3 Comparison and conditions 4 See also 5 References 6 External links ICD-10 taxonomy [ edit ] The tenth revision of the International Statistical Classification of Diseases and Related Health Problems (ICD-10) has four categories of developmental disorders: specific developmental disorders of speech and language, specific developmental disorders of scholastic skills, specific developmental disorder of motor function, and mixed specific developmental disorder. [2] [6] DSM-III taxonomy [ edit ] In the third edition of the Diagnostic and Statistical Manual of Mental Disorders (DSM-III), [7] SDD was opposed to the pervasive developmental disorders (PDD). There were two factors that were considered: The specificity of the impairment: in SDD there is one single domain that is affected, whereas in PDD multiple areas of functioning are affected. [8] The nature of the impairment: development in SDD is delayed but not otherwise abnormal, whereas in PDD there are behavioral deviations that are not typical for any developmental stage. [8] In the DSM-IV, specific developmental disorders were no longer grouped together. [9] Instead they were reclassified as communication disorders, learning disorders, and motor skills disorders. [3] Comparison and conditions [ edit ] ICD-10 [10] DSM-IV-TR [11] ICD-11 [12] Specific developmental disorders of speech and language (F80): Specific speech articulation disorder (F80.0) Expressive language disorder (F80.1) Receptive language disorder (F80.2) Acquired aphasia with epilepsy Landau-Kleffner syndrome (F80.3) Other developmental disorders of speech and language (F80.8) Developmental disorder of speech and language, unspecified (F80.9) Communication disorders : Expressive Language Disorder (315.31) Mixed Receptive-Expressive Language Disorder (315.32) Phonological Disorder (315.39) Stuttering (307.0) Communication Disorder Not Otherwise Specified (307.9) Developmental Speech & Language Disorders (6A01): Developmental speech sound disorder (6A01.0) Developmental speech fluency disorder (6A01.1) Developmental language disorder (6A01.2) Developmental language disorder with impairment of receptive and expressive language (6A01.20) Developmental language disorder with impairment of mainly expressive language (6A01.21) Developmental language disorder with impairment of mainly pragmatic language (6A01.22) Developmental language disorder, with other specified language impairment (6A01.23) Other specified developmental speech or language disorders (6A01.Y) Developmental speech or language disorders, unspecified (6A01.Z) Specific developmental disorders of scholastic skills (F81): Specific reading disorder (F81.0) Specific spelling disorder (F81.1) Specific disorder of arithmetical skills (F81.2) Mixed disorder of scholastic skills (F81.3) Other disorders of scholastic skills (F81.8) Developmental disorder of scholastic skills, unspecified (F81.9) Learning disorders : Reading Disorder (315.0) Mathematics Disorder (315.1) Disorder of Written Expression (315.2) Learning Disorder Not Otherwise Specified (315.9) Developmental learning disorder (6A03): Developmental learning disorder with impairment in reading (6A03.0) Developmental learning disorder with impairment in written expression (6A03.1) Developmental learning disorder with impairment in mathematics (6A03.2) Developmental learning disorder with other specified impairment of learning (6A03.3) Developmental learning disorder, unspecified (6A03.Z) Specific developmental disorder of motor function (F82) Motor skills disorders : Developmental coordination disorder (315.4) Developmental motor coordination disorder (6A04) Mixed specific developmental disorder (F83) See also [ edit ] Developmental disability References [ edit ] ^ Diagnostic and Statistical Manual of Mental Disorders, Fifth Edition . ... Taylor: Child and Adolescent Psychiatry , 4th ed. 2005 ^ Robert Jean Campbell, III: Campbell's Psychiatric Dictionary , 2003, page 184 ^ http://apps.who.int/classifications/icd10/browse/2010/en#/F80 Reference for all ICD-10 disorders mentioned in the table. ^ http://behavenet.com/apa-diagnostic-classification-dsm-iv-tr#301 Reference for all DSM-IV-TR disorders mentioned in the table. ^ https://icd.who.int/browse11/l-m/en#/http%3a%2f%2fid.who.int%2ficd%2fentity%2f334423054 Reference for all ICD-11 disorders mentioned in the table External links [ edit ] Classification D ICD - 10 : F80 , F81 , F82 , F83 ICD - 9-CM : 307 , 315 v t e Dyslexia and related specific developmental disorders Conditions Speech, language , and communication Expressive language disorder Infantile speech Landau–Kleffner syndrome Language disorder Lisp Mixed receptive-expressive language disorder Specific language impairment Speech and language impairment Speech disorder Speech error Speech sound disorder Stuttering Tip of the tongue Learning disability Dyslexia Dyscalculia Dysgraphia Disorder of written expression Motor Developmental coordination disorder Developmental verbal dyspraxia Sensory Auditory processing disorder Sensory processing disorder Related topics Dyslexia research Irlen filters Learning Ally Learning problems in childhood cancer Literacy Management of dyslexia Multisensory integration Neuropsychology Reading acquisition Spelling Writing system Lists Dyslexia in fiction Languages by Writing System People with dyslexia
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Deaf-Mute
Wikipedia
The term continues to be used to refer to deaf people who cannot speak an oral language or have some degree of speaking ability, but choose not to speak because of the negative or unwanted attention atypical voices sometimes attract. Such people communicate using sign language . [1] Some consider it to be a derogatory term if used outside its historical context; the preferred term today is simply "deaf". [2] Contents 1 Historical usage of "deaf-mute" and other terms 1.1 United Kingdom 1.2 North America 1.3 Jewish law 2 Deaf-mute people in history 3 Deaf-muteness in art and literature 4 References Historical usage of "deaf-mute" and other terms [ edit ] United Kingdom [ edit ] In 19th century British English "mute" and "dumb" meant "non-speaking", and were not pejorative terms. ... Within the deaf community there are some who prefer the term " Deaf " (upper-case D) to "deaf" (lower-case) as a description of their status and identity. [11] Jewish law [ edit ] Classification as a deaf-mute has a particular importance in Jewish law . Because historically it was thought impossible to teach or communicate with them, deaf-mutes were not moral agents , and therefore were unable to own real estate , act as witnesses , or be punished for any crime. ... Reading Between the Signs: Intercultural Communication for Sign Language Interpreters . ^ Moore, Matthew S. & Levitan, Linda (2003). For Hearing People Only, Answers to Some of the Most Commonly Asked Questions About the Deaf Community, its Culture, and the "Deaf Reality" , Rochester, New York: Deaf Life Press. ^ for the Blind, the Deaf etc., Commission (1889).
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Pervasive Developmental Disorder Not Otherwise Specified
Wikipedia
Pervasive developmental disorder not otherwise specified (PDD-NOS), (including atypical autism) Specialty Clinical psychology , psychiatry , pediatrics , occupational medicine A pervasive developmental disorder not otherwise specified (Including atypical autism) ( PDD-NOS ) [1] is one of the four autistic disorders (AD) in the DSM-5 [2] and also was one of the five disorders classified as a pervasive developmental disorder (PDD) in the DSM-IV [3] According to the DSM-4 , PDD-NOS is a diagnosis that is used for "severe and pervasive impairment in the development of reciprocal social interaction or verbal and nonverbal communication skills, or when stereotyped behavior, interests, and activities are present, but the criteria are not met for a specific PDD" or for several other disorders. [4] PDD-NOS includes atypical autism , [5] because the criteria for autistic disorder are not met, for instance because of late age of onset, atypical symptomatology, or subthreshold symptomatology, or all of these. [4] Even though PDD-NOS is considered milder than typical autism , this is not always true. While some characteristics may be milder, others may be more severe. [6] Contents 1 Signs and symptoms 2 Diagnosis 2.1 Subgroups 3 Treatment 4 References 5 External links Signs and symptoms [ edit ] It is common for individuals with PDD-NOS to have more intact social skills and a lower level of intellectual deficit than individuals with other PDDs. [3] Characteristics of many individuals with PDD-NOS are: Communication difficulties (e.g., using and understanding language) [7] Difficulty with social behavior Difficulty with changes in routines or environments Uneven skill development (strengths in some areas and delays in others) Unusual play with toys and other objects Repetitive body movements or behavior patterns Preoccupation with fantasy , such as imaginary friends in childhood Diagnosis [ edit ] PDD-NOS is an old diagnostic category. It is not included as an option for an Autism Spectrum Disorder and is not part of the DSM-5 , but is included in the ICD-10 , as either "atypical autism" or "pervasive developmental disorder, unspecified". [8] The diagnosis of a pervasive developmental disorder not otherwise specified is given to individuals with difficulties in the areas of social interaction, communication, and/or stereotypic behavior patterns or interests, but who do not meet the full DSM-4 criteria for autism or another PDD. ... It only means that individuals who receive this diagnosis do not meet the diagnostic criteria of the other PDDs, but that there is still a pervasive developmental disorder that affects the individual in the areas of communication, socialization, and behavior. [3] As for the other pervasive developmental disorders, the diagnosis of PDD-NOS requires the involvement of a team of specialists. The individual needs to undergo a full diagnostic evaluation, including a thorough medical, social, adaptive, motor skills and communication history. [3] Other parts of an assessment can be behavioral rating scales, direct behavioral observations, psychological assessment, educational assessment, communication assessment, and occupational assessment. [9] Description of PDD-NOS merely as a "subthreshold" category without a more specific case definition poses methodological problems for research regarding the relatively heterogeneous group of people who receive this diagnosis.
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Developmental Disorder
Wikipedia
Most learning disabilities are found under the age of 9. [3] Young children with communication disorders may not speak at all, or may have a limited vocabulary for their age. [4] Some children with communication disorders have difficulty understanding simple directions or are unable to name objects. [4] Most children with communication disorders are able to speak by the time they enter school, however, they continue to have problems with communication. [4] School-aged children often have problems understanding and formulating words. [4] Teens may have more difficulty with understanding or expressing abstract ideas. [4] Causes [ edit ] The scientific study of the causes of developmental disorders involves many different theories. ... Structural defects are seen in the cerebellum too, which affect the motor and communication skills. [14] Sometimes the left lobe of the brain is affected and this causes neuropsychological symptoms. ... Speech-language therapy can help those with autism who need to develop or improve communication skills. According to the organization Autism Speaks, “speech-language therapy is designed to coordinate the mechanics of speech with the meaning and social use of speech”. [24] People with low-functioning autism may not be able to communicate with spoken words. Speech-language Pathologists (SLP) may teach someone how to communicate more effectively with others or work on starting to develop speech patterns. [25] The SLP will create a plan that focuses on what the child needs. ... Other disorders [ edit ] Learning disabilities Communication disorders Developmental coordination disorder Genetic disorders , such as Down syndrome or Williams syndrome Tic disorders such as Tourette syndrome .PTEN, MECP2, SETD1A, SHANK3, DOCK8, SLC6A8, TBCD, NANS, CAMKMT, CNTN4, WDFY4, KCNT1, AS3MT, ARFGAP1, CHRNA4, CHD4, KCNQ2, CBL, LRP2, DRD2, SLC4A4, SLC33A1, SLC2A1, STAMBP, PMP22, KIF1A, MAPK3, PNKP, NTRK2, MSL3, PTPN11, FMR1, FGD1, CREBBP, TP63, CHD7, KRAS, FOXF1, PITX2, NIPBL, PAX6, ELN, RAC1, KCNK9, ACTB, KRT5, CDC42, ARSD, FOXL2, RNU4ATAC, FOXP2, DENR, MID1, SNAP25, ASXL1, SOX2, KCNQ3, PQBP1, IKBKG, NR0B1, PAG1, TBX2, IGF2, HTC2, PCBP4, TBX3, JAG1, RPE65, HNF1B, RAI1, NOTCH3, NRAS, DYRK1A, OPN1LW, RAD51, PIK3CA, ZACN, BBS2, PTCH1, PLAGL1, CKAP4, MED13L, CILK1, TPP1, ZEB2, COG6, GLI1, EIF4E, FGFR2, FOXG1, FOXC1, UVRAG, FOLR1, PUM1, MTOR, FRAXE, GJB2, ATRX, CLN3, TFE3, HIRA, GNAS, LINC01081, LINC01082, ZMYND11, GJB6, MTA2, MAP1LC3C, SRCAP, MIR34A, KIAA0319, TGM5, SMC3, GTF2H5, PDLIM1, EIF2S2, EIF2B2, EIF2B4, SYNGAP1, KPNA7, PUF60, LPAR2, BABAM2, DGCR2, KIAA0586, AKT3, DNM1L, GTF2IRD1, GDF3, HNRNPR, DLK1, POLR1C, TBX4, AKR1A1, KDM5B, POTEF, CFDP1, NRXN1, TRIM32, ZBTB18, CXCR6, KMT2B, LONP1, EMG1, CADPS2, MAST1, TBL1XR1, INPP5E, AGPAT4, ACKR3, CPA6, SALL4, ARID1B, ARHGAP31, NUFIP2, ESCO2, PRUNE1, KMT2C, PRDM13, PROK2, COLEC11, EHMT1, KDM3A, MIPOL1, TRIM71, PIFO, CSPP1, WDR81, UBA5, CPXM2, ZRANB3, RNF135, MYO18B, RAB39B, GPT2, PRRT2, GLYATL1, SMG9, VPS13B, SMCHD1, SMARCAL1, SPECC1L, PEG13, KAT6B, DAPK2, DSTYK, PART1, SETBP1, ZBTB20, G6PC3, ATP2C1, CYP26C1, AHDC1, PCLO, TBK1, KMT5B, RABL6, PCDH12, LINC00299, ZBTB7A, RAB23, POLR3K, KDM3B, RABL3, RBM20, TMCO1, ARX, AHI1, BMPER, SMPD4, FREM3, SLC17A5, CDKL5, CDK13, GABRD, GTF2H1, GRIN2B, GPR42, GLI3, GLI2, GJA1, FRAXA, GTF2H3, AFF2, FGFR3, FGF10, FAT1, EXT2, ERCC3, GTF2H2, GTF2H4, EPHB2, ITPR1, KCNQ1, KCNMA1, KCNJ6, KCNH1, KCNC3, ANOS1, IL10, H1-4, IL6, IGF1, HRAS, HIC1, NRG1, HCCS, ERCC2, EIF2B1, OFD1, ATP2A2, MYRF, BRS3, BRAF, BDNF, BCL6, AVP, ATP1A3, CASP9, APP, AHR, ADSL, ADRA2B, ADRA1A, ADAM10, CACNA1E, CDK8, EGFR, DHCR7, EDNRA, DPYD, DOCK3, DNMT3A, DLG2, DIO3, DACH1, CETN3, CTNNB1, CSNK2A1, CRMP1, CPT1A, COL9A3, CLIC2, KIT, SMAD4, MEIS1, SSTR4, TBX5, MAP3K7, TAF1, STX1A, ACVR1, STIM1, SOX11, NR2F1, SOX10, HLTF, SLC6A3, SHH, SET, SCN2A, TCF4, TGIF1, KITLG, FZD5, FZD1, NAA10, SMC1A, USP9X, KMT2D, LHX3, WT1, TWIST1, VEGFA, VCP, UTRN, UROD, UBE3A, TYR, RPS23, ROBO1, RET, NOTCH1, PIGA, CFP, PDE4D, PDC, SIX6, NR4A2, CNOT3, RARA, NF1, NDN, MTTP, MTHFR, MT1B, KMT2A, PIK3CB, PIK3CD, PIK3CG, PKD1, PLXNA1, PMM2, POLD1, PTPA, PPT1, MAPK1, MAP2K2, MAP2K7, MASP1, RELN, PTGS2, PTPRG, RALA, FMR1-IT1
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Red Scare
Wikipedia
Any of several events in which widespread fear of communism or leftism develops Not to be confused with Red Terror . ... OCLC 27976811 . ^ Countryman, Edward (2010). "Communism" . In Kazin, Michael; Edwards, Rebecca; Rothman, Adam (eds.). ... Cuordileone, "The Torment of Secrecy: Reckoning with Communism and Anti-Communism After Venona", Diplomatic History , vol. 35, no. 4 (Sept. 2011), pp. 615–642. ... John Earl Haynes, Red Scare or Red Menace?: American Communism and Anti Communism in the Cold War Era . ... Richard Gid Powers, Not Without Honor: A History of American Anti-Communism . New York: Free Press, 1997.
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Mycobacterium Tuberculosis, Susceptibility To, 1
Omim
An epidemic of TB occurred in a community of aboriginal Canadians during the period of 1987 to 1989. Greenwood et al. (2000) collected genetic and epidemiologic data on an extended family from this community, and assessed evidence for linkage to the NRAMP1 gene (600266).
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Hiv/aids In Kenya
Wikipedia
Social outlook [ edit ] LGBTQ+ [ edit ] As the LGBTQ+ community has always been part of the fight against HIV/AIDS in all countries, the same has happened in Kenya. ... There are many social stigmas involved with people aged 15-19, as they go through harsh environments in schools and in the community. HIV and AIDS in school is viewed as a killer disease that is a sign of sexual immorality. ... It has been reported that 29.3% of sex workers have HIV. The main problem within this community is the fear of coming forward about being raped or abused, because it could lead to prosecution for being a prostitute. ... Self testing kits for low cost have been introduced over recent years, along with community based testing and door-to-door campaigns. ... Free condoms have been distributed throughout different communities, including the sex workers community.
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Logorrhea (Psychology)
Wikipedia
A communication disorder that causes excessive wordiness and repetitiveness This article is about the psychological term. ... In psychology , logorrhea or logorrhoea (from Ancient Greek λόγος logos "word" and ῥέω rheo "to flow"), also known as press speech , is a communication disorder that causes excessive wordiness and repetitiveness, which can cause incoherency. ... Antipsychotics are often used, and lithium is a common supplement given to manic patients. [16] For patients with lesions of the brain, attempting to correct their errors may upset and anger the patients, since the language center of their brain may not be able to process that what they are saying is incorrect and wordy. [ citation needed ] See also [ edit ] Cluttering – Speech and communication disorder Compulsive talking Gibberish – Nonsensical language Glossolalia Graphorrhea – Disorder of incoherent written rambling List of language disorders – Wikipedia list article Schizoaffective disorder – Mental Disorder Schizophrenia – Mental illness characterized by abnormal behavior and misinterpretation of reality Word salad , also known as Schizophasia – Confused unintelligible jumble of words and phrases References [ edit ] ^ "Logorrhea" . ... Language Intervention Strategies in Aphasia and related neurogenic communication disorders . 5 : 3–19. ^ Prigatano, George (2010). ... New York: Oxford University Press. ^ Brookshire, R (2003). "Introduction to neurogenic communication disorders". Introduction to neurogenic communication disorders : 155. ^ Obler, Loraine (1999).
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Attention-Deficit/hyperactivity Disorder
Medlineplus
Affected individuals may also have autism spectrum disorder, which is characterized by impaired communication and social interaction, or Tourette syndrome, which is a disorder characterized by repetitive and involuntary movements or noises called tics. ... Other genes associated with ADHD provide instructions for proteins that are important for communication between neurons. Some of these proteins help produce or control the chemicals that relay communication signals between neurons (neurotransmitters ). Other proteins have roles at the connections between neurons (synapses ) where this communication takes place. The specific ways that changes in associated genes are involved in the development of ADHD are unknown.DRD5, COMT, DRD4, STS, GRM5, FGD1, AS3MT, MED13, ADGRL3, DRD2, CIC, SLC6A3, TPH2, CHRNA4, GRM7, GIT1, CNR1, TACR1, GRM8, CALY, PTPRD, CHRNB2, GRM1, ZNF292, DHDDS, CHRNA7, GRIN2A, ASTN2, MECP2, UPF3B, PTCHD1, SYP, CACNA1C, FOXP2, ST3GAL3, GJB2, PTEN, ITIH3, FAS, AUTS2, THRB, NTRK2, CPLX2, SLC6A8, DMD, IFNG, PIK3CA, PRKG1, FMR1, SLITRK1, PTPRG, GABRB3, PAH, IQSEC2, SMC3, TRNS2, TRNW, NBN, SEC24C, GTF2IRD1, ARHGEF6, SYNGAP1, NCL, SYNJ1, BAZ1B, BRSK2, PPM1D, HERC2, SEMA3E, IKBKG, CACNA1H, JRK, FEZ1, NOP56, FRMPD4, MID2, ND6, TRNF, MKRN3-AS1, GNB5, RAI1, GPC6, TRNH, GNE, TRNL1, TRNQ, USP9X, TRNS1, MED12, KIF14, ATP2C2, CPLX1, NUAK1, DEAF1, MAGED2, TLK2, HDAC4, SMC1A, PAK3, CHAF1B, AP3B2, SLC6A9, SLC6A4, OCRL, SLC6A2, SLC2A1, SLC1A2, SIM1, SDHA, SCN8A, SCN3A, OPHN1, RXRG, RREB1, RPS20, RPS6KA3, RFC2, RAD21, PTPRF, PRNP, MAPK1, PRKCG, PPP3CA, POLG, PMS2, PMS1, SMPD1, SNAP25, SNRPN, TSC2, ADAM12, TRIM26, PCGF2, MKRN3, ZNF41, ZNF711, YWHAG, CLIP2, PCNT, UBE3A, HIRA, TSC1, SOX5, TRIO, NDN, TSPAN7, IL1RAPL1, TGFBR2, NDP, TCF20, TCF4, TBX1, STXBP1, SPG7, UFD1, VPS13A, ITGA11, AK8, FBXL16, PWAR1, PIWIL4, MUCL3, IRAK1BP1, OSR1, MTFMT, RAB39B, NUS1, CSMD2, C12orf57, ALKBH8, MLIP, SLC9A7, ASCC2, FERMT3, TMEM47, CSRNP3, SEMA6D, PANK2, UBA5, ALG13, SPAG16, ARV1, CSMD1, SEMA4A, CXorf56, VPS13B, SPRED1, BCORL1, ARX, LINC01572, OBI1-AS1, P2RX5-TAX1BP3, BORCS7-ASMT, KDM4A-AS1, TAF9BP2, SNORD116-1, PWRN1, LINC00461, MIR137HG, USP27X, SFTA2, MIR99AHG, CEP85L, KIF7, HCN1, TUBB2B, ZNF81, SNORD115-1, ZNF615, LAMA1, SLC13A5, ASPM, JMJD1C, CEP112, CFAP221, ARID2, PIEZO2, SORCS2, PUF60, SH3KBP1, MLH3, CYFIP2, TBL2, SETBP1, TENM4, NECAP1, PARS2, SH2B1, SIN3A, NIPBL, FTSJ1, NPAP1, CDK20, ZFPM2, ADNP, MED13L, SYNE1, SZT2, SATB2, ND4, SORCS3, NT5C2, SHANK2, FAN1, TRAK1, NLGN1, CNKSR2, SRPX2, ZNRD1, TBC1D24, TAX1BP3, SLC4A10, C12orf4, HDAC8, MCTP2, CHD7, DHTKD1, PI4K2B, ACOXL, MAP11, SETD5, PHIP, NSUN2, DYM, CNNM2, RBFOX1, MAGEL2, WWOX, MPP6, ACTL6B, WAC, RSRC1, CRBN, ZDHHC9, MLXIPL, WDPCP, NTM, SCAPER, ND5, AARS1, ND1, IPW, BDNF, BMPR1A, BRCA2, CACNA1A, CACNA1B, IGF1, CACNB2, DNM1, HTR2A, HTR1B, HSPG2, HOXA2, HLA-DPB2, HLA-DMB, HIVEP1, BCR, ITGAE, AGTR2, ATP6V1A, LIMK1, LIG4, LHCGR, ANK3, KRAS, KIF11, KIF5B, ARF1, ARSD, KCNB1, KCNA2, ARVCF, ITPR3, STT3A, RERE, CHD2, CLCN4, HDC, HCFC1, CSNK2A1, FLII, FLI1, FGF12, ACSL4, EMP2, ELN, EEF1A2, DBH, DPP6, DDX3X, TIMM8A, DHCR7, DYNC1I2, DLG3, GABRA1, GABRB2, GABRG2, GRIA4, GTF2I, MSH6, CLTC, GRIN2D, COL2A1, COL11A2, GP1BB, GATA4, GNAS, GNAQ, GLUD1, CREBBP, CRKL, GDI1, ABCD1, DYNC1H1, MOBP, MEF2C, ADCY2, KMT2A, MAOB, ADRA2A, MAOA, MSH2, MANBA, EPCAM, COX1, MAN2A2, MLH1, COX2, COX3, MITF, DRD1, CDH13, CLOCK, ELK3, CYP2D6, DNTT, MTHFR, NTF3, STX1A, WASF2, ADRA1A, EPHB1, SLC9A9, NGF, CHPT1, HTR1A, LOC110806262, RSS, NR3C1, SSTR4, LPAR2, ADRA2B, HTR2C, SMS, ADRA2C, MC4R, GTS, BAIAP2, GRIN2B, CXCR6, REM1, DDC, DRD3, LOC107987479, EDNRA, NOS1, GPR42, NPY, BRS3, TPH1, NET1, ACKR3, HCRT, VEGFA, DISC1, SHANK3, CES1, ABCB10P1, WASF1, DIRAS2, NR4A2, IMPACT, STUB1, ADIPOQ, CHRNA3, ZNF804A, AMPH, SLC1A3, VAMP2, ANKK1, SHBG, MIRLET7D, ADORA2A, CDK5, FLRT3, DCDC2, NDRG2, CFP, HLA-DRB1, PART1, PER2, ATXN1, CRP, TSPAN31, NCAM1, DTNBP1, LOC102723407, GUCY2C, CNTNAP2, SCLY, RBM12, ADHD5, OPN1SW, BAG3, LOC102724971, OXTR, CHRNA5, ERICD, SPN, COA7, F2R, APOE, CYBC1, NANS, DCLK1, GAD1, SOD1, DNAAF4, TERF1, PRS, GNPAT, GRIA1, INSRR, BTBD9, C4B, SLC9A6, INS, IL1B, IL1RN, SLC2A3, NPSR1, IL5, IL6, TRH, TPO, TERC, KRT7, IL10, DNMT3B, BDNF-AS, SYN3, EBPL, RFC1, CAT, SYT1, BCHE, INTS8, MTR, TAL1, PIK3CG, POMC, POLR3A, ZFPM2-AS1, CSF2, PSC, APP, ACACA, CSE1L, FBXO33, CRY1, SLC30A10, CREM, NPNT, MIR5692B, PIDD1, TTC12, CYP3A4, PRTG, NCAN, AGA, IPO11, NAT8L, COL6A4P1, ALDH2, FEZF1, ALB, AKT1, GAL, NR0B1, ASCC1, DLG2, SPOCK3, STIN2-VNTR, DLX4, LOC390714, AMH, BRINP1, CCHCR1, CTNND1, TRIT1, ELFN1, CYP2B6, P4HTM, CYP2D7, CNTN5, PLA1A, AMPD1, GPRC5B, CPT2, THRA1/BTR, DAPK3, LOC105379528, ADRB2, CNTFR, ADM, BPIFA2, CBS, PPP1R1B, KYAT1, CCT, LMAN2L, KCNIP4, CD38, CD40, AGT, NRSN1, CD79A, CDH11, MIR34B, MIR320A, RNF122, MICALL2, NTPCR, STXBP5-AS1, AVP, ZGPAT, MIR148B, RBM45, SYT2, BHMT, DCD, THEM4, ADHD2, ADHD4, XKR4, MIR34C, MIR378A, NDUFAF2, SLC39A13, CASP3, CLEC19A, ZBBX, ATM, EAF2, KLK3, DGKH, TMX2-CTNND1, MIR107, CNTN1, OXER1, ARNTL, PRXL2C, APRT, FEZF1-AS1, LXN, C9orf72, DNAJC12, DYX8, KYAT3, ASH1L, CORD1, KIDINS220, NUFIP2, LUCAT1, MIR138-1, MBOAT7, FTO, ADRA1B, ASMT, NSD1, CHRNB3, SLC39A8, CHRNB4, CLTA, MIR3171, MIR142, LINC01672, HAMP, DPP10, LRRC7, SNORC, NUDT3, NT5C, VDR, SLC18A2, SLC16A1, KCNC1, KCNJ5, KCNJ6, KCNJ11, SLC6A1, SLC5A4, SKI, LAIR1, SDHD, LAMB2, CCL11, SCT, LAMC2, LGALS3, SCD, ATXN7, S100B, FADS1, LMNA, REG1A, RARA, SMN1, SMN2, SNCA, THAS, IL1A, TXN, TWIST1, PHLDA2, IL2, TNR, TSPAN8, THRSP, THRA, THBS3, TH, ITPA, IL16, ABCA4, ITGA1, SULT2A1, STXBP3, CDKL5, STATH, STAT6, SST, SRY, LMX1B, RAB3A, PEX2, NGFR, PDE4A, PDE2A, MFAP1, PC, PRKN, MFGE8, MIP, OPRM1, NTRK3, NTRK1, NFKB1, PFN1, NF1, NR3C2, MNT, MOG, MYT1, MYO5B, MPP2, MSMB, MTHFD1, MTNR1A, PDE4D, SERPINA1, PTPRN2, PROC, LRP6, LRP5, CYP4F3, PTPRC, PTH, PTGDS, PSPH, PSG5, PSD, RELN, PSPN, PIK3CB, MXD1, PRKCD, PRKCB, PPIA, PON1, MAP1B, MARK1, PMCH, SERPINF2, PIK3CD, IGFALS, VIPR2, KCNIP1, IGF2, RRS1, CYFIP1, FN1, VWA8, GAST, TRIM32, GALR1, GCH1, GFI1, PARK7, HRH3, SEC23IP, NISCH, CORO1A, TRIM31, RAB40B, PDE10A, GPER1, WASF3, NRG3, GRIA2, SH2B2, GRIK5, FKBP5, FHIT, FGF2, EGF, PADI1, ZCCHC4, IGHV3-69-1, IGHV3OR16-7, HTRA2, ZNF544, CNTN6, DOCK3, AGO1, EDA, EIF4EBP1, SYNM, TPSG1, PRPF6, EPHA5, FRRS1L, CADM1, SMUG1, OTP, KCNH3, ERBB2, FAAH, CIB1, CACNG2, RIDA, HPRT1, HK1, KALRN, SELENBP1, CHRNA6, ARHGEF7, HP, NRP1, NRP2, LIN7A, B4GALT2, USO1, PKMYT1, BHLHE40, HRC, HTR1D, HTR1E, ZMYM2, TRIM25, HTR3A, HTR4, HTR5A, HTR6, ARTN, PDLIM1, GPHN, HFE, PSMD14, TSHZ1, RASGRP1, BCL2L10, SLC12A6, GSK3B, BMS1, GYPA, GUCY2D, CARTPT, BCAR1, LGI1, ADAMTS2, HGF, ITM2B, FADS2, NRXN1, TGM5, PDLIM7, GPR50, DLGAP1, MTA2, H3P40
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Culture Shock
Wikipedia
Still, the most important change in the period is communication: People adjusting to a new culture often feel lonely and homesick because they are not yet used to the new environment and meet people with whom they are not familiar every day. ... Weaver wrote that culture shock has "three basic causal explanations": loss of familiar cues, the breakdown of interpersonal communications, and an identity crisis. [10] Peter S. ... Cultural conflict Cultural cringe Cultural intelligence Cultural schema theory Expatriate Fresh off the boat Future Shock Intercultural communication Jetlag Neophobia Outsourced (film) Student exchange program References [ edit ] ^ Macionis, John, and Linda Gerber. ... Westport, Conn: Greenwood Press, 1995. ^ Barna, LaRay M. "HOW CULTURE SHOCK AFFECTS COMMUNICATION." Communication 5.1 (n.d.): 1-18. ... In: G.R. Weaver, Editor, Culture, communication, and conflict: Readings in intercultural relations, Ginn Press, Needham Heights, MA (1994), pp. 169–189. ^ Adler, P.S. 1975.