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  • Abarognosis Wikipedia
    Notes[edit] ^ Dorland 2011, baragnosis ^ Black 1995, pg. 14 ^ abarognosis, Drugs.com, retrieved 2013-03-21 ^ "abarognosis", The American Heritage® Stedman's Medical Dictionary, Houghton Mifflin, retrieved 2013-03-21 ^ Campbell 2012, pg. 554 Sources[edit] Black, Peter McLaren; Rossitch, Eugene (1995), "Neurological Diagnosis", Neurosurgery: An Introductory Text (Google eBook), Oxford University Press, ISBN  9780195044492 , retrieved 2013-02-21 Buck, Carol J. (2013), "Section Index to Diseases and Injuries", 2013 ICD-9-CM for Physicians (Google preview), Vol. 2 (Professional ed.), Elsevier Health Sciences, ISBN  9781455775033 , retrieved 2013-03-21 Campbell, William W. (2012), "36: Sensory Localization", DeJong's The Neurologic Examination (Google eBook) (7th ed.), Lippincott Williams & Wilkins, ISBN  9781469817521 , retrieved 2013-03-21 Dorland (2011), Dorland's Illustrated Medical Dictionary (Google eBook) (32nd ed.), Elsevier Health Sciences, ISBN  9781455709854 , retrieved 2013-03-21 External links[edit] Classification D ICD-9-CM: 781.99
  • Post-Vagotomy Diarrhea Wikipedia
    Current surgical therapy (10th ed.). Philadelphia, PA: Elsevier Saunders. ... Swanström LL, Eubanks WS (eds.). Mastery of endoscopic and laparoscopic surgery (3rd ed.).
  • Progestin-Induced Virilization Wikipedia
    (ed.). Estrogens and Progestogens in Clinical Practice (3rd ed.). London: Churchill Livingstone. pp. 533–53. ... (eds.). Hirsutism and Virilism: Pathogenesis, Diagnosis and Management. ... (eds.). Yen and Jaffe's Reproductive Endocrinology : Physiology, Pathophysiology, and Clinical Management (5th ed.). ... Larry (eds.). Endocrinology (5th ed.). Philadelphia: Elsevier Saunders. pp. 2779–829. ... (eds.). Goodman & Gilman's The Pharmacological Basis of Therapeutics (11th ed.).
  • Tetany Wikipedia
    . ^ Hall, John, ed. (2010). Guyton and Hall textbook of medical physiology (12th ed.). ... ISBN  978-1-4160-4574-8 . ^ Hall, John, ed. (2010). Guyton and Hall textbook of medical physiology (12th ed.). ... Beef Cattle Handbook (PDF). ^ Hall, John, ed. (2010). Guyton and Hall textbook of medical physiology (12th ed.).
    CNNM2, AKAP5, ARVCF, CASR, TRPM6, CYP2R1, CLDN16, TCIRG1, SEC24C, GCM2, UFD1, HIRA, TBCE, TBX1, SLC12A3, SLC12A1, RREB1, PTH, KCNJ1, GP1BB, GNAS, CYP27B1, COMT, JMJD1C
  • Manganese Deficiency (Medicine) Wikipedia
    (eds.). Essential Metals in Medicine:Therapeutic Use and Toxicity of Metal Ions in the Clinic. ... (eds.). Present Knowledge in Nutrition (7th ed.). ILSI Press. pp. 334–343. ... (ed.). Manganese in health and disease. Modern Nutrition. 2. CRC Press. pp. 39–58.
    SLC39A8
  • Essential Fructosuria Wikipedia
    In Saudubray J, van den Berghe G, Walter JH (eds.). Inborn Metabolic Diseases: Diagnosis and Treatment (5th ed.). ... In Berghe G, Fernandes J, Saudubray J, Walter JH (eds.). Inborn Metabolic Diseases. Diagnosis and Treatment (fourth ed.). Springer. p. 137. ISBN  978-3-540-28783-4 . ^ Tran C (April 2017). ... Anesthesia and Uncommon Diseases (third ed.). Philadelphia: Saunders.
    KHK
    • Fructosuria, Essential Omim
      A number sign (#) is used with this entry because of evidence that essential fructosuria is caused by compound heterozygous mutation in the KHK gene (614058) on chromosome 2p23. Description Essential fructosuria is a benign, asymptomatic defect of intermediary metabolism characterized by the intermittent appearance of fructose in the urine (summary by Bonthron et al., 1994). Clinical Features In individuals with essential fructosuria, ingestion of dietary fructose, sucrose, or sorbitol is followed by an abnormally large and persistent rise in blood fructose concentration and by excretion of 10 to 20% of the ingested load in the urine (Gitzelmann et al., 1989). Essential fructosuria was first described independently by Czapek (1876) and Zimmer (1876) in a man who also suffered from diabetes mellitus. Laron (1961) counted 50 published cases, of which 18 were in Jews. The enzyme deficiency was demonstrated in liver by Schapira et al. (1961).
    • Essential Fructosuria Orphanet
      Essential fructosuria is a rare autosomal recessive disorder of fructose metabolism (see this term) caused by a deficiency of fructokinaseenzyme activity. It is characterized by elevated fructosemia and presence of fructosuria following ingestion of fructose and related sugars (sucrose, sorbitol). Essential fructosuria is clinically asymptomatic and harmless. Dietary restriction is not indicated.
  • Actinomycosis Wikipedia
    (eds.). Actinomycosis in: Baron's Medical Microbiology (4th ed.). Univ of Texas Medical Branch. ... PMID 25095985. ^ Ryan KJ; Ray CG, eds. (2004). Sherris Medical Microbiology (4th ed.). ... Williams gynecology (2nd ed.). New York: McGraw-Hill Medical. p. 42. ISBN  978-0071716727 . ^ Böhm, Ingrid; Willinek, Winfried; Schild, Hans H. ... Fitzpatrick's Dermatology in General Medicine (7th ed.). McGraw Hill. ^ Sullivan, D. ... Stitt's Diagnosis, Prevention and Treatment of Tropical Diseases (7th ed.). p. 1182. ^ Control of Communicable Diseases in Man (8th ed.).
    CAT
    • Actinomycosis Orphanet
      A rare bacterial infectious disease characterized by a chronic granulomatous infection by Actinomyces species which are commensals in the human gastrointestinal and urogenital tract and oropharynx. Corresponding to the affected site, the disease presents as cervicofacial, respiratory tract, genitourinary tract, digestive tract, central nervous system, or cutaneous actinomycosis and leads to the formation of abscesses and fistulae in the respective region.
    • Actinomycosis Gard
      Actinomycosis is a bacterial infection that occurs most often in the face and neck. Symptoms of actinomycosis include a neck mass, jaw or face pain, and formation of pockets of pus (abscess). When actinomycosis occurs in other parts of the body, symptoms can include cough, chest or stomach pain, fever, and weight loss. It is usually caused by bacteria called Actinomyces israelii. These bacteria are found normally in the mouth, gastrointestinal tract, and female genital tract, and do not cause an infection unless there is a break in the skin or mucosa. The infection is not contagious. Actinomycosis is diagnosed based on the history and clinical exam, and through specific laboratory tests. 
  • Cirsoid Aneurysm Wikipedia
    Histologically, they are composed of both thick- and thin-walled blood vessels.[2] References[edit] ^ McKee's Pathology of the Skin (5 ed.). Elsevier. 2020. pp. 1698–1894. ^ a b Dermatopathology (2 ed.). Saunders. 2016. pp. 513–594.
    MAP2, ACVRL1, ENG, SLPI, RASA1, SMAD4, TNF, VEGFA, KRAS, APOE, IL1B, EPHB4, MMP3, PART1, ANGPTL4, BRAF, PTEN, ANGPT2, IL6, S100B, SPP1, TEK, MAP2K1, MAPK1, PLXNA2, PLOD2, PAGE1, TGFBR1, VCAM1, NOTCH4, SYNGAP1, PAGE4, NOS1AP, MVP, SMUG1, TMEM100, SOX17, PDGFD, COL18A1, ESAM, MIR137, MIR195, PDGFRB, MYB, NOTCH3, NOTCH1, ALCAM, ALX3, ANPEP, ANXA5, BDNF, VPS51, CDKN2A, CRYAB, ELN, EPHB2, ETS1, F3, FOXC2, FLT1, MTOR, GJA5, HRAS, ICAM1, IFNG, MDM4, MGP, MMP2, MMP9, ABCC1, MSH3, AGT, NOS1, NOS2, NOS3, MIR210
  • Accommodative Insufficiency Wikipedia
    Theory and practice of optics and refraction (2nd ed.). Elsevier. pp. 100–107. ISBN  978-81-312-1132-8 . ^ Synopsis of Clinical Ophthalmology (3 ed.). ... The practice of refraction (8th ed.). London: Churchill. ISBN  0-7000-1410-1 . ^ a b c d William J., Benjamin (2006). ... Borish's clinical refraction (2nd ed.). St. Louis Mo.: Butterworth Heinemann/Elsevier. p. 112.
  • Megalocornea Wikipedia
    Taylor and Hoyt's pediatric ophthalmology and strabismus (5th ed.). Edinburgh: Elsevier. ... Oxford handbook of ophthalmology (4th ed.). New York: Oxford university press. ... Kanski's clinical ophthalmology : a systematic approach (9th ed.). Edinburgh: Elsevier.
    CHRDL1, LTBP2, TINF2, NSD2, NELFA, BAZ1B, LARGE1, GTF2IRD1, POMT1, PRDM5, KIFBP, TBL2, COL1A1, GMPPB, POMT2, POMGNT1, PIEZO2, FKRP, GNPTAB, SLC2A10, POMK, ZNF469, CLIP2, CDH11, FGFR2, GTF2I, COL5A1, COL5A2, COL11A1, ELN, FBN1, FKTN, FGD1, RFC2, TEK, LETM1, LIMK1, NFIX, PIK3R1, PITX2, PPP2CA, SMAD1, COL8A2, COL8A1, ZAP70, RPS6KB2
    • Isolated Congenital Megalocornea Orphanet
      Isolated congenital megalocornea is a genetic, non-syndromic developmental defect of the anterior eye segment characterized by bilateral enlargement of the corneal diameter (>12.5 mm) and a deep anterior eye chamber, without an elevation in intraocular pressure. It can manifest with mild to moderate myopia as well as photophobia and iridodonesis (due to iris hypoplasia). Associated complications include lens dislocation, retinal detachment, presenile cataract development, and secondary glaucoma.
    • Isolated Congenital Megalocornea Gard
      Isolated congenital megalocornea affects the cornea, the part of the eye that lets in light. The symptoms of isolated congenital megalocornea include mild nearsightedness, light sensitivity, and/or unusual vibration of the iris. At birth, the corneas are thin and enlarged. Cataracts may develop by 30-50 years of age. Other complications may include other cornea abnormalities, lens dislocation, and break down of the iris. Isolated congenital megalocornea occurs when the CHRDL1 gene is not working correctly.
    • Megalocornea Omim
      Autosomal inheritance is much rarer than X-linked (309300). Megalocornea is often found in the Marfan syndrome (154700). HEENT - Large cornea Inheritance - Autosomal recessive much rarer than X-linked ▲ Close
    • Megalocornea Omim
      A number sign (#) is used with this entry because of evidence that megalocornea (MCG1) is caused by mutation in the CHRDL1 gene (300350) on chromosome Xq23. Description Megalocornea is an inherited eye disorder in which the corneal diameter is bilaterally enlarged (greater than 13 mm) without an increase in intraocular pressure. It may also be referred to as 'anterior megalophthalmos,' since the entire anterior segment is larger than normal. Features of megalocornea in addition to a deep anterior chamber include astigmatic refractive errors, atrophy of the iris stroma, miosis secondary to decreased function of the dilator muscle, iridodonesis, and tremulousness, subluxation, or dislocation of the lens. Whereas most affected individuals exhibit normal ocular function, complications include cataract development and glaucoma following lenticular dislocation or subluxation.
  • Marche À Petit Pas Wikipedia
    In: Simon RP, Aminoff MJ, Greenberg DA. eds. Clinical Neurology, 10e New York, NY: McGraw-Hill. ^ a b Gait Disorders. In: Srinavasan J; Chaves CJ; Scott BJ; Small JE, eds. Netter's Neurology, 3rd ed.
    L1CAM, SOD1, MSL3, TTC19
  • Endometrosis Wikipedia
    In Samper, JC (ed.). Equine breeding management and artificial insemination (2nd ed.). ... Scott (eds.). Equine clinical medicine, surgery, and reproduction. London: Manson Publishing/The Veterinary Press. p. 318.
  • Fibrinogenolysis Wikipedia
    Fibrinogenolysis Specialty Hematology Primary fibrinogenolysis is a medical condition that appears with abnormal production of fibrinogen/fibrin degradation products (FDP), degradation of coagulation factors V, VIII, IX, XI and/or degradation of the fibrin present in any pre-existing localized thrombi and hemostatic clots.[1][2] References[edit] ^ Marder VJ, Williams WJ, Beutler E, Erslev AJ, Lichtman MA, eds. (1990). ... Hematology. ^ Potron G, Caen JP, Tobelem G, Soria C, eds. (1988). "Fisiopatologia delle iperfibrinolisi".
    PLAU
  • Bark-Binding Wikipedia
    Further reading[edit] Trees portal  This article incorporates text from a publication now in the public domain:  Chambers, Ephraim, ed. (1728). ... Cyclopædia, or an Universal Dictionary of Arts and Sciences (1st ed.).
  • Ehlers-Danlos Syndrome, Cardiac Valvular Type Omim
    The first patient was a 45-year-old man who had received the diagnosis of EDS type II (see 130010) because of recurrent shoulder dislocations since childhood, small joint hypermobility, pectus excavatum, muscle and tendon tears, bilateral inguinal hernias, small scars under the chin and on the forehead, atrophic scars over the knees and shins, easy bruisability, and generally hyperextensible and thin skin. ... The second patient, who had been described by Hata et al. (1988) and Kojima et al. (1988), had also been diagnosed with EDS type II. ... Molecular Genetics In 2 of the patients with EDS studied by Schwarze et al. (2004), COL1A2 mRNA instability resulted from compound heterozygosity for splice site mutations in the COL1A2 gene (120160.0045-120160.0046 and 120160.0047-120160.0048, respectively). ... Malfait et al. (2006) recorded the clinical features of a 6-year-old boy in whom complete lack of alpha-2 chains of type I collagen (120160.0052) was associated with a phenotype reminiscent of mild hypermobility EDS (225320). ... The phenotypic consequences are those of a form of EDS with hypermobility in childhood, complicated by cardiac valve disease in adulthood.
    COL1A2
    • Cardiac-Valvular Ehlers-Danlos Syndrome Orphanet
      A rare form of Ehlers-Danlos syndrome (EDS) characterized by soft skin, skin hyperextensibility, easy bruisability, atrophic scar formation, joint hypermobility and severe, progressive cardiac valvular defects comprising mitral and/or aortic valve insufficiency.
  • Ovine Pulmonary Adenocarcinoma Wikipedia
    . ^ a b c d Meuten, DJ, ed. (2016). "Chapter 12: Tumors of the respiratory tract". Tumors in domestic animals (5th ed.). John Wiley & Sons. pp. 467–498. ... In Fan, Hung (ed.). Jaagsiekte Sheep Retrovirus and Lung Cancer. Berlin, Heidelberg: Springer Berlin Heidelberg. pp. 55–79. doi:10.1007/978-3-642-55638-8_2. ... In Fan, Hung (ed.). Jaagsiekte sheep retrovirus and lung cancer. Volume 275 of Current Topics in Microbiology and Immunology. ... In Fan, Hung (ed.). Jaagsiekte Sheep Retrovirus and Lung Cancer. Berlin, Heidelberg: Springer Berlin Heidelberg. pp. 55–79. doi:10.1007/978-3-642-55638-8_3.
  • Pili Pseudoannulati Wikipedia
    Fitzpatrick's Dermatology in General Medicine. (6th ed.). McGraw-Hill. ... Andrews' Diseases of the Skin: Clinical Dermatology. (10th ed.). Saunders.
    • Pseudopili Annulati Omim
      Description Pseudopili annulati is an unusual variant of normal hair characterized by a banded appearance of the hair under reflective light, as observed in pili annulati (180600), but resulting from a distinct underlying physical defect. There is no increased hair fragility (Price et al., 1970). Clinical Features Price et al. (1970) reported a 9-year-old girl with blond hair whose scalp hair showed light and dark banding since infancy. Light microscopy showed that the banding appeared only if the light struck the hair at right angles to the long axis of the hair, which was distinct from that observed with pili annulati. Polarizing light indicated variability in the thickness of the hair shaft, and illuminating light did not show bands, 2 additional features that distinguished it from pili annulati. Further examination of the hair shaft showed that the pseudopili annulati hairs had periodic widening and narrowing, that the fiber cross-section was roughly elliptical, and that a periodic twisting of the hair (30 to 40 degrees in 2 alternating directions) was superimposed on the ellipticity.
  • Pterygium Unguis Wikipedia
    Fitzpatrick's Dermatology in General Medicine. (6th ed.). McGraw-Hill. ... Andrews' Diseases of the Skin: Clinical Dermatology. (10th ed.). Saunders.
    COL17A1, DKC1, NOP10, NHP2
  • Pigmented Hairy Epidermal Nevus Syndrome Wikipedia
    Andrews' Diseases of the Skin: Clinical Dermatology. (10th ed.). Saunders. ... Fitzpatrick's Dermatology in General Medicine. (6th ed.). McGraw-Hill.
  • Equine Recurrent Uveitis Wikipedia
    In Gilger, BC (ed.). Equine Ophthalmology (3rd ed.). John Wiley & Sons. pp. 380–415. ISBN  9781119047742 . ^ Equine Recurrent Uveitis ^ a b Maggs, D; Miller, P; Ofri, R, eds. (2013). ... Slatter's fundamentals of veterinary ophthalmology (5th ed.). St. Louis, Mo.: Elsevier. pp. 235–240.
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