During occasional unique and/or stimulating circumstances, a person with EDS can sometimes remain animated, awake and alert, for brief or extended periods of time. EDS can affect the ability to function in family, social, occupational, or other settings. ... A number of tools for screening for EDS have been developed. One is the Epworth Sleepiness Scale which grades the results of a questionnaire. ... [citation needed] Treatment[edit] Treatment of EDS relies on identifying and treating the underlying disorder which may cure the person from the EDS. Drugs like modafinil,[3] Armodafinil,[4] Xyrem (sodium oxybate) oral solution, have been approved as treatment for EDS symptoms in the U.S.
Millon (Eds.), Oxford textbook of psychopathology (2nd ed.) (pp. 527–548). ... Richard (ed.). Sexual Deviance: Theory, Assessment, and Treatment, 2nd edition. ... Sexual Deviance, Second Edition: Theory, Assessment, and Treatment (2nd ed.). ... Retrieved July 25, 2013. ^ a b c American Psychiatric Association, ed. (2013). ... Hoffman (Eds.), Crime in biological, social, and moral contexts (pp. 100–114).
Borish's Clinical Refraction (2 ed.). p. 112. ^ "accommodative excess". ^ a b c d AK Khurana. ... Theory and Practice of Optics and Refraction (2 ed.). Elsevier. pp. 105–106. ^ a b c William J, Benjamin. ... Clinical Management of Binocular Vision (4 ed.). p. 357. ^ AK Khurana. ... Theory and Practice of Optics and Refraction (2 ed.). p. 77. ^ Wajuihian SO, Hansraj R. ... Clinical Management of Binocular Vision (4 ed.). p. 354.
To prevent spread of the disease, infected animals are separated from non-infected goats, or culled.[5] References[edit] ^ a b c d Maclachlan, NJ; Dubovi, EJ, eds. (2010). ... Fenner's Veterinary Virology (5th ed.). Academic Press. pp. 294–295. ISBN 9780128011706 . ^ Stonos, N; Wootton, SK; Karrow, N (22 August 2014). ... PMID 25153344. ^ a b Smith, MC; Sherman, DM, eds. (2011). "Caprine arthritis encephalitis (CAE) and Maedi Visna (MV)". Goat medicine (2nd ed.). John Wiley & Sons. ISBN 9781119949527 . ^ a b Peacock, Christie (1996). ... Diseases of the goat (4th ed.). John Wiley & Sons. pp. 98–102. ISBN 9781119073529 .
MCQ's in medical surgical nursing : (with explanatory answers) (1st ed.). ... The Brigham intensive review of internal medicine (Second ed.). p. 551. ... Moderate sedation/analgesia : core competencies for practice (2nd ed.). ... William Schwartz; et al., eds. (2012). The 5-minute pediatric consult (6th ed.). ... Urodynamics (3. ed.). London: Springer. p. 120. ISBN 978-1-85233-924-1 .
Episodic dyscontrol syndrome Specialty Psychiatry Episodic dyscontrol syndrome (EDS), otherwise known as intermittent explosive disorder (IED)[1] or sometimes just dyscontrol, is a pattern of abnormal, episodic, and frequently violent and uncontrollable social behavior[2] in the absence of significant provocation;[3] it can result from limbic system diseases, disorders of the temporal lobe,[4] or abuse of alcohol or other psychoactive substances.[5][6] EDS is a clearly identified category in the Diagnostic and Statistical Manual of Mental Disorders (DSM IV).[7] EDS may affect children or adults.[8][9][10] Children are often considered to have epilepsy or a mental health problem. ... (March 2018) Treatment[edit] Treatment for EDS usually involves treating the underlying causative factor(s). This may involve psychotherapy, or medical treatment for diseases.[12] EDS has been successfully controlled in clinical trials using prescribed medications, including carbamazepine,[13][14] ethosuximide,[15] and propranolol.[16] There have been few randomised controlled trials of treatment of EDS/IED. ... Adolescents and young adults may experience educational and social consequences but also mental health problems, including parasuicide, if IED/EDS is undiagnosed in early childhood.[17] Legal implications[edit] A diagnosis of EDS has been used as a defense in court for persons accused of committing violent crimes including murder.[18][19][20] See also[edit] Intermittent explosive disorder References[edit] ^ McTague, A.; Appleton, R. (1 June 2010). ... Review of Clinical Psychiatry and the Law (Hardback) (Version 2 ed.).
(eds.), Murray and Nadel's Textbook of Respiratory Medicine, 2 (5th ed.), Saunders, ISBN 978-1-4160-4710-0 ^ Shayne C Gad (2005), "Petroleum Hydrocarbons", in Philip Wexler (ed.), Encyclopedia of Toxicology, 3 (2nd ed.), Elsevier, pp. 377–379, ISBN 0-12-745354-7 ^ Rosalyn Carson-DeWitt; Rebecca J. ... Fundukian (ed.), The Gale Encyclopedia of Medicine, 5 (4th ed.), Gale, pp. 3457–3465, ISBN 978-1-4144-8646-8
Edward (eds.). Robinson's Current Therapy in Equine Medicine (7th ed.). ... (ed.). Pathologic basis of veterinary disease (6th ed.). Elsevier. p. 604. ... Equine dermatology (2nd ed.). Maryland Heights, Missouri: Elsevier/Saunders. pp. 431–433. ... Self-Assessment Color Review Equine Internal Medicine (2nd ed.). CRC Press. p. 232.
Millon (Eds.), Oxford Textbook of Psychopathology (2nd ed.) (pp. 527–548). ... Barbaree (Eds.), Handbook of Sexual Assault: Issues, Theories, and Treatment of the Offender (pp. 195–207). ... Sexual Anomalies and Perversions: Physical and Psychological Development, Diagnosis and Treatment (new and revised ed.). ... O'Donohue (Eds.), Sexual deviance: Theory, assessment, and treatment (2nd ed.). ... O'Donohue (Eds.), Sexual Deviance: Theory, Assessment, and Treatment (2nd ed.).
Small Animal Dermatology: A Color Atlas and Therapeutic Guide (4th ed.). ... (eds.). Clinical Medicine of the Dog and Cat (3rd ed.). CRC Press. p. 779. ... Skin Diseases of the Dog and Cat a Colour Handbook (2nd ed.). London: CRC Press. p. 18. ... Muller & Kirk's Small Animal Dermatology (7th ed.). St. Louis, Missouri: Elsevier. pp. 677–678. ... Small Animal Dermatology (6th, rev. ed.). London: CRC Press. p. 90. ISBN 9781840766356 . ^ Susan M.
Contemporary Oral and Maxillofacial Pathology. 2nd ed. St. Louis, MO: Mosby, 2002. 152-53. ^ Cawson, R. ... Cawson's Essentials of Oral Pathology and Oral Medicine. 8th ed. Edinburgh: Churchill Livingstone, 2008. 145-46. ^ a b c d Wood, Norman K., Paul W. ... Differential Diagnosis of Oral and Maxillofacial Lesions. 5th ed. St. ... Dentistry for the Child and Adolescent. 8th ed. St. Louis, MO: Mosby, 2004. 163-64. ^ a b White, Stuart C., and M. ... Oral Radiology: Principles and Interpretation. 6th ed. St. Louis, MO: Mosby/Elsevier, 2009. 385-87.
Eales disease (ED) is characterized by 3 sequential vascular responses that determine the course of the disease: inflammation (peripheral retinal perivasculitis); occlusion (peripheral retinal capillary non-perfusion); and neovascularisation of the retina or disk, which often leads to vitreous hemorrhage. ... In addition, headache, variation in peripheral circulation, dyspepsia, chronic constipation, and epistaxis have also been associated with ED. ... Etiology The etiology of ED remains elusive; Several immunological, molecular biological, and biochemical studies have indicated the roles of human leukocyte antigen, retinal S-antigen autoimmunity, Mycobacterium tuberculosis genome, free radical damage, and hyperhomocysteinemia in the pathogenesis of Eales disease. ... Intravitreal anti-VEGF therapy is currently being tested as a definitive therapy for ED. ... Blindness due to ED is rare. There is no known mortality associated with the disease.
Eales disease is a rare vision disorder that appears as an inflammation and white haze around the outercoat of the veins in the retina. This condition is most common among young males and normally affects both eyes. In most cases, vision becomes suddenly blurred because the vitreous, the clear jelly that fills the eyeball behind the lens of the eye, seeps out. Treatment includes corticosteroids in the inflammation stage and photocoagulation in the proliferative stage of the disease. Visual prognosis is good if treatment begins early in the course of the disease.
Vascular endothelial growth factor (VEGF) is found in an increased amount in patients with ED and explains the increase in vascularization within the eye. ... OCT is a noninvasive way to view the retina and identify macular edema, neovascularization, and hemorrhage.[4] The diagnosis of Eales disease can be broken down into several stages due to the regular progression of the disease.[6] Stage Description I Periphlebitis of small (Ia) and large (Ib) caliber vessels with superficial retinal hemorrhages IIa Capillary non-perfusion IIb Revascularization elsewhere/of the disc IIIa Fibrovascular proliferation IIIb Vitreous hemorrhage IVa Traction/combined rhegmatogenous retinal detachment IVb Rubeosis iridis, neovascular glaucoma, complicated cataract, and optic atrophy Differential diagnosis[edit] There are many other ailments that show similar symptoms to ED. ... PMID 23514227. ^ Comprehensive Ophthalmology, (4th Ed) 2007 by A K Khurana, Professor, Regional Institute of Ophthalmology, Postgraduate Institute of Medical Sciences, Rohtak- 124001, India ^ Eales disease at eMedicine ^ "Eales Disease - an overview | ScienceDirect Topics". www.sciencedirect.com.
Nomenclature The kyphoscoliotic type of Ehlers-Danlos syndrome (EDS VI; 225400) was at one time separated into EDS VIA (with lysyl hydroxylase deficiency) and EDS VIB (with normal lysyl hydroxylase activity). The designation EDS VIB was then thought to include the brittle cornea syndrome. Another entity formerly called EDS VIB is now known as the musculocontractural type of EDS (601776), caused by mutation in the CHST14 gene (608429). ... Cadle et al. (1985) studied 3 sisters with EDS VI phenotype but normal lysyl hydroxylase and the additional feature of macrocephaly. ... A distinct pattern of collagen crosslinks, indicative of decreased lysyl hydroxylation, could be identified in EDS VIA patients, but there was no clear correlation between collagen crosslink pattern and changes in the individual lysyl hydroxylase mRNAs of EDS VIB patients.
A rare, hereditary connective tissue disease characterized by severe ocular manifestations due to extreme corneal thinning and fragility with rupture in the absence of significant trauma, often leading to irreversible blindness. Extraocular manifestations comprise deafness, developmental hip dysplasia, and joint hypermobility.
Ehlers-Danlos syndrome is a group of disorders that affect connective tissues supporting the skin, bones, blood vessels, and many other organs and tissues. Defects in connective tissues cause the signs and symptoms of these conditions, which range from mildly loose joints to life-threatening complications. The various forms of Ehlers-Danlos syndrome have been classified in several different ways. Originally, 11 forms of Ehlers-Danlos syndrome were named using Roman numerals to indicate the types (type I, type II, and so on). In 1997, researchers proposed a simpler classification (the Villefranche nomenclature) that reduced the number of types to six and gave them descriptive names based on their major features.
Brittle cornea syndrome (BCS) is a genetic disease involving the connective tissue in the eyes, ears, joints, and skin. The symptoms of BCS typically involve thinning of the protective outer layer of the eye (cornea), which may lead to tearing or rupture after minor damage to the cornea. Other eye symptoms may include nearsightedness (myopia), a blueish tint in the white part of the eyes (blue sclera), and retinal detachment. Other symptoms may include hearing loss, abnormal positioning of the hip bones (hip dysplasia), and soft skin with abnormal scarring. There are 2 types of BCS. BCS type 1 is caused by changes (mutations) in the ZNF469 gene and BCS type 2 is caused by changes in the PRDM5 gene.
A number sign (#) is used with this entry because of evidence that brittle cornea syndrome-2 (BCS2) is caused by homozygous mutation in the PRDM5 gene (614161) on chromosome 4q27. Description Brittle cornea syndrome (BCS) is characterized by blue sclerae, corneal rupture after minor trauma, keratoconus or keratoglobus, hyperelasticity of the skin, and hypermobility of the joints (Al-Hussain et al., 2004). It is classified as a form of Ehlers-Danlos syndrome (Malfait et al., 2017). For a discussion of genetic heterogeneity of brittle cornea syndrome, see BCS1 (229200). Clinical Features Cameron (1993) described 11 patients with blue sclerae, limbus-to-limbus corneal thinning, hypermobile joints, and consanguineous parents.
.; Frazer, Grant (eds.). Equine medicine, surgery and reproduction (2nd ed.). ... (ed.). Equine Ophthalmology (3rd ed.). John Wiley & Sons. p. 175. ISBN 9781119047858 .
. ^ Gynecologic pathology : a volume in the series Foundations in diagnostic pathology (Second ed.). p. 502. ISBN 978-0-323-35909-2 . ^ Gynecologic pathology : a volume in the series Foundations in diagnostic pathology (Second ed.). p. 502. ISBN 978-0-323-35909-2 . ^ Gynecologic pathology : a volume in the series Foundations in diagnostic pathology (Second ed.). p. 500. ISBN 978-0-323-35909-2 . ^ Gynecologic pathology : a volume in the series Foundations in diagnostic pathology (Second ed.). p. 501.
