WA 0821 1305 0400 Perusahaan Vendor Hydroseeding Stabilisasi Lereng Lampung Tengah Lampung

Deep Research Advanced

Phallophobia Wikipedia

. ^ "Waduh, Banyak Wanita Gemetaran saat Melihat Mr P Ereksi! - Tribun Lampung" . Tribun Lampung (in Indonesian). 2014-12-25 .
Eurotophobia Wikipedia

. ^ "Kenapa Sih Ada Pria yang Justru Ketakutan saat Melihat Kelamin Wanita?" . Tribun Lampung . 2014-12-25. mengapa ini masih menjadi masalah bagi laki-laki dan perempuan ... seorang dokter baru merasa takut melihat kelamin perempuan ketika mereka bekerja melalui rotasi kebidanan ...
Hiv/aids In Mali Wikipedia

Condom use is also low among other vulnerable populations, such as military personnel, truck drivers, and vendors. A recent survey found that only 12% of women vendors in Bamako (with an estimated HIV seroprevalence of 6.7%) reported using a condom with their last non-regular partner. [1] Some people still do not believe that AIDS is real, citing rumors that it is a myth propagated by people in Western/European countries who don't want Africans to have more children.
Cystic Eyeball Wikipedia

Diagnostic and Interventional Radiology (Ankara, Turkey) . 16 (2): 116–21. doi : 10.4261/1305-3825.DIR.2054-08.1 . PMID 19847771 .

PRSS56
Acral Myxoinflammatory Fibroblastic Sarcoma Wikipedia

Musculoskeletal Imaging 188 (5) 1302-1305 ^ Meis-Kindblom JM, Kindblom LG (1988) Acral myxoinflammatory fibroblastic sarcoma: a low-grade tumor of the hands and feet.

TGFBR3, OGA, VGLL3, BRAF, CD68, FGF8, VIM, TOM1L2
Stewart–treves Syndrome Wikipedia

"[Stewart-Treves syndrome (angiosarcoma on lyphoedema): A rare complication of lymphoedema]". Presse Méd . 39 (12): 1305–8. doi : 10.1016/j.lpm.2010.06.017 .
Desmosis Wikipedia

. : “Pathology of Chronic Constipation in Pediatric and Adult Coloproctology“, Karger 2005 ^ Meier-Ruge WA. (1998). "Desmosis of the colon: a working hypothesis of primary chronic constipation". Eur J Pediatr Surg.8; 299-303 ^ Meier-Ruge WA, Bruder E. (2007). "The morphological characteristics of aplastic and atrophic desmosis of the intestine". Pathologe 28: 149-54 ^ Meier-Ruge WA, Bruder E. (2005). "Atrophic desmosis as secondary connective tissue atrophy in muscularis propria".
Blue Toe Syndrome Wikipedia

PMID 19103358 . ^ Blackshear JL, Oldenburg WA, Cohen MD (Dec 1994). "Making the diagnosis when the patient has 'blue toes ' ". ... PMID 22477301 . ^ Blackshear JL, Oldenburg WA, Cohen MD (Dec 1994). "Making the diagnosis when the patient has 'blue toes ' ". ... PMID 7982584 . ^ Blackshear JL, Oldenburg WA, Cohen MD (Dec 1994). "Making the diagnosis when the patient has 'blue toes ' ". ... PMID 7982584 . ^ Blackshear JL, Oldenburg WA, Cohen MD (Dec 1994). "Making the diagnosis when the patient has 'blue toes ' ".
Cholera Mayo Clinic

Eat food that's completely cooked and hot and avoid street vendor food, if possible. If you do buy a meal from a street vendor, make sure it's cooked in your presence and served hot.

PCYT1B, CTBS, CFTR, SPINK1, EGF, ATP8B1, ANXA5, IL1B, NBAS, WASF2, CYP27A1, ATN1, CYP2B6, GLB1, HSP90AA1, FBXW7, POMC, IL17A, VEGFA, TYRP1, TRPC1, NAGLU, NPY, SOD1, ABO, SLC9A3, SCN7A, ACE2, EVPL, CAV1, VIP, TM7SF2, TNF, TRAF3, TRP-AGG2-5, MIR132, TICAM2, CAVIN1, EZR, WARS1, ZNRD2, PTF1A, BAP1, TNFSF9, NRSN1, SPHK1, WASF1, TPH2, ART5, THBS1, NOD2, MIR146A, MIR155, RANBP2, S100A4, S100A8, CLEC11A, H3P23, SLC5A1, LOC102724197, LOC102723971, TMED7-TICAM2, MFT2, SPR, STAR, SYP, TAPBP, H3P37, TRBV20OR9-2, TF, CAP1, NLRP3, CRLF2, BPIFB1, IL22, TMED7, VPS54, GGTLC1, IL23A, TSLP, PPIL1, MAP1LC3A, KRT20, CTTNBP2, SEPTIN3, CMIP, NSFL1C, PHF12, XYLT2, GOLPH3, TNMD, DUOX2, EFEMP2, DCTN6, PYCARD, WASF3, FASTK, TMED2, SNRNP35, TPPP, AKAP13, FOXP2, CHP1, CNOT1, SLC39A6, SUMF2, LDLRAP1, IFT172, PTH, LAT, ARFIP1, SLC6A16, PTHLH, SERPINA3, PTEN, CYLD, CETP, CHRM1, CHRM3, CKB, COX8A, CRP, CSF2, CCN2, CTSD, CYP11A1, CD81, CYP19A1, CYP26A1, CD55, ACE, DECR1, DLG4, EGFR, ENO1, ERBB2, CDK2, CD44, ESRRA, KLK3, ADCY6, ADPRH, AKT1, AKT2, ALB, ANGPT1, APOB, APOE, APRT, STS, CD40LG, ALDH7A1, BCL2, TSPO, CACNA1E, CAMP, CASR, CAV3, CD9, MS4A1, ESR1, F9, PSMD9, PIK3CA, LEP, LHCGR, MBL2, MBP, MFAP1, MMP9, NT5E, NTF4, ABCB4, PIK3CB, KIT, PIK3CD, PIK3CG, PLEK, POU4F1, PRF1, PRG2, PRSS1, PRSS8, PSEN1, LCN1, ITGB1, PTK2B, HSPA4, FCGR3A, FDX1, FLII, ACKR1, GLP1R, GPR39, HIVEP1, HMGB1, HSD17B1, HSPA8, ING1, HSPD1, ICAM1, IFI27, IFNA1, IFNA13, IKBKB, IL6, CXCL8, IDO1, H3P19
- Cholera GARD
  
  Cholera is an infection of the small intestines that is caused by the bacterium Vibrio cholera . The condition can range from mild to severe and many affected people may have no obvious signs or symptoms. Approximately 5-10% of infected people will have severe disease with watery diarrhea and vomiting leading to rapid fluid loss, dehydration, and shock. If left untreated, this can cause acute renal failure, severe electrolyte imbalances, coma, or even death. People develop cholera when they eat food or drink water that is contaminated with Vibrio cholera .
- Cholera Orphanet
  
  Cholera is an infectious disease, caused by intestinal infection with Vibrio cholerae , characterized by massive watery diarrhea and severe dehydration that can lead to shock and death if left untreated. Epidemiology Cholera is endemic to over 50 countries (defined as having reported cholera cases for the last 3 years with evidence of local transmission), mainly in Asia and Africa. In addition, outbreaks have occurred throughout Africa, Asia, the Middle East, South and Central America, and the Caribbean. Worldwide, it is estimated that there are 1-4 million cases per year. In Europe, the disease is extremely rare, occurring as isolated, imported cases.
- Cholera Wikipedia
  
  This article is about the bacterial disease. For the dish, see Cholera (food) . Bacterial infection of the small intestine Cholera Other names Asiatic cholera, epidemic cholera [1] A person with severe dehydration due to cholera causing sunken eyes and wrinkled hands and skin. Specialty Infectious disease Symptoms Large amounts of watery diarrhea , vomiting , muscle cramps [2] [3] Complications Dehydration , electrolyte imbalance [2] Usual onset 2 hours to 5 days after exposure [3] Duration A few days [2] Causes Vibrio cholerae spread by fecal-oral route [2] [4] Risk factors Poor sanitation , not enough clean drinking water , poverty [2] Diagnostic method Stool test [2] Prevention Improved sanitation, clean water , hand washing , cholera vaccines [2] [5] Treatment Oral rehydration therapy , zinc supplementation , intravenous fluids , antibiotics [2] [6] Frequency 3–5 million people a year [2] Deaths 28,800 (2015) [7] Cholera is an infection of the small intestine by some strains of the bacterium Vibrio cholerae . [4] [3] Symptoms may range from none, to mild, to severe. [3] The classic symptom is large amounts of watery diarrhea that lasts a few days. [2] Vomiting and muscle cramps may also occur. [3] Diarrhea can be so severe that it leads within hours to severe dehydration and electrolyte imbalance . [2] This may result in sunken eyes , cold skin, decreased skin elasticity, and wrinkling of the hands and feet. [5] Dehydration can cause the skin to turn bluish . [8] Symptoms start two hours to five days after exposure. [3] Cholera is caused by a number of types of Vibrio cholerae , with some types producing more severe disease than others. [2] It is spread mostly by unsafe water and unsafe food that has been contaminated with human feces containing the bacteria. [2] Undercooked seafood is a common source. [9] Humans are the only animal affected. [2] Risk factors for the disease include poor sanitation , not enough clean drinking water , and poverty . [2] There are concerns that rising sea levels will increase rates of disease. [2] Cholera can be diagnosed by a stool test . [2] A rapid dipstick test is available but is not as accurate. [10] Prevention methods against cholera include improved sanitation and access to clean water . [5] Cholera vaccines that are given by mouth provide reasonable protection for about six months. [2] They have the added benefit of protecting against another type of diarrhea caused by E. coli . [2] The primary treatment is oral rehydration therapy —the replacement of fluids with slightly sweet and salty solutions . [2] Rice-based solutions are preferred. [2] Zinc supplementation is useful in children. [6] In severe cases, intravenous fluids , such as Ringer's lactate , may be required, and antibiotics may be beneficial. [2] Testing to see which antibiotic the cholera is susceptible to can help guide the choice. [3] Cholera affects an estimated 3–5 million people worldwide and causes 28,800–130,000 deaths a year. [2] [7] Although it is classified as a pandemic as of 2010 [update] , it is rare in the developed world . [2] Children are mostly affected. [2] [11] Cholera occurs as both outbreaks and chronically in certain areas . [2] Areas with an ongoing risk of disease include Africa and Southeast Asia . [2] The risk of death among those affected is usually less than 5% but may be as high as 50%. [2] No access to treatment results in a higher death rate. [2] Descriptions of cholera are found as early as the 5th century BC in Sanskrit . [5] The study of cholera in England by John Snow between 1849 and 1854 led to significant advances in the field of epidemiology . [5] [12] Seven large outbreaks have occurred over the last 200 years with millions of deaths. [13] Play media Video summary ( script ) Contents 1 Signs and symptoms 2 Cause 2.1 Transmission 2.2 Susceptibility 3 Mechanism 3.1 Genetic structure 3.2 Antibiotic resistance 4 Diagnosis 5 Prevention 5.1 Surveillance 5.2 Vaccination 5.3 Sari filtration 6 Treatment 6.1 Fluids 6.2 Electrolytes 6.3 Antibiotics 6.4 Zinc supplementation 7 Prognosis 8 Epidemiology 9 History 9.1 Research 10 Society and culture 10.1 Health policy 10.2 Notable cases 10.3 In popular culture 11 Country examples 11.1 Zambia 11.2 India 11.3 Democratic Republic of Congo 12 References 13 Further reading 14 External links Signs and symptoms Typical cholera diarrhea that looks like "rice water" The primary symptoms of cholera are profuse diarrhea and vomiting of clear fluid. [14] These symptoms usually start suddenly, half a day to five days after ingestion of the bacteria. [15] The diarrhea is frequently described as "rice water" in nature and may have a fishy odor. [14] An untreated person with cholera may produce 10 to 20 litres (3 to 5 US gal) of diarrhea a day. [14] Severe cholera, without treatment, kills about half of affected individuals. [14] If the severe diarrhea is not treated, it can result in life-threatening dehydration and electrolyte imbalances. [14] Estimates of the ratio of asymptomatic to symptomatic infections have ranged from 3 to 100. [16] Cholera has been nicknamed the "blue death" [17] because a person's skin may turn bluish-gray from extreme loss of fluids. [18] Fever is rare and should raise suspicion for secondary infection. Patients can be lethargic and might have sunken eyes, dry mouth, cold clammy skin, or wrinkled hands and feet. Kussmaul breathing , a deep and labored breathing pattern, can occur because of acidosis from stool bicarbonate losses and lactic acidosis associated with poor perfusion .
Pinealoblastoma Wikipedia

American Journal of Neuroradiology . 16 (1): 157–65. PMID 7900586 . ^ de Jong MC, Kors WA, de Graaf P, Castelijns JA, Moll AC, Kivelä T (December 2015). ... PMID 10561222 . ^ de Jong MC, Kors WA, de Graaf P, Castelijns JA, Kivelä T, Moll AC (September 2014).

DICER1, ASMT, MYC, PDE4DIP, NIBAN1, CRB3, NEUROD4, PCDHGA3, DROSHA, PHOX2B, CRX, TPH1, SMARCB1, RB1, MYCN, CD99, INSM1, DICER1-AS1
- Pineoblastoma Orphanet
  
  Pineoblastoma is a rare, malignant type of supratentorial primitive neuroectodermal tumor (sPNET), found mainly in children (less than 10% of cases are reported in adults), and located in the pineal region of the brain but that can metastasize along the neuroaxis. As it is the most aggressive of the pineal parenchymal tumors, it is usually associated with a poor prognosis.
- Pineoblastoma GARD
  
  Pineoblastoma is a type of cancerous ( malignant ) tumor that grows in a part of the brain known as the pineal gland . It occurs mainly in children. Symptoms of pineoblastoma include a buildup of fluid around the brain (hydrocephalus), headaches, nausea, and difficulty with eye movement. Without treatment, pineoblastomas can cause weakness and difficulty controlling movement. The long term outcome depends on the age at diagnosis, the size of the tumor, and if the tumor has spread outside the brain ( metastasized ). The cause of pineoblastoma is unknown, but specific inherited genetic variants in two genes, RB1 and DICER1 can increase the risk for a pineoblastoma.
Primary Familial Brain Calcification Wikipedia

SourceGeneReviews [Internet]. Seattle (WA): University of Washington, Seattle; 1993–2004 ^ Chiu HF, Lam LC, Shum PP, Li KW (January 1993). ... B Neuropsychiatr. Genet . 153B (7): 1305–10. doi : 10.1002/ajmg.b.31102 .

SLC20A2, PDGFRB, PDGFB, XPR1, MYORG, IBGC1, MIA2, HPT, PTH, RALGAPA1
- Basal Ganglia Calcification, Idiopathic, 4 OMIM
  
  A number sign (#) is used with this entry because idiopathic basal ganglia calcification-4 (IBGC4) is caused by heterozygous mutation in the PDGFRB gene (173410) on chromosome 5q32. Description Idiopathic basal ganglia calcification-4 is an autosomal dominant condition characterized by the accumulation of calcium deposits in various brain regions, most commonly in the basal ganglia. About half of mutation carriers are asymptomatic, but some present later in life with parkinsonism and impaired cognitive function. Migraine or depression may occur in younger individuals (summary by Nicolas et al., 2013). For a detailed phenotypic description and a discussion of genetic heterogeneity of IBGC, see IBGC1 (213600).
- Basal Ganglia Calcification, Idiopathic, 1 OMIM
  
  A number sign (#) is used with this entry because of evidence that idiopathic basal ganglia calcification-1 (IBGC1) is caused by heterozygous mutation in the SLC20A2 gene (158378) on chromosome 8p11. Description Familial idiopathic basal ganglia calcification is an autosomal dominant condition characterized by symmetric calcification in the basal ganglia and other brain regions. Patients with calcifications can either be asymptomatic or show a wide spectrum of neuropsychiatric symptoms, including parkinsonism, dystonia, tremor, ataxia, dementia, psychosis, seizures, and chronic headache. Serum levels of calcium, phosphate, alkaline phosphatase, and parathyroid hormone are normal. The typical age at clinical onset is between 30 and 50 years (summary by Wang et al., 2012).
- Bilateral Striopallidodentate Calcinosis Orphanet
  
  Bilateral striopallidodentate calcinosis (BSPDC, also erroneously called Fahr disease) is characterized by the accumulation of calcium deposits in different brain regions, particularly the basal ganglia and dentate nucleus, and is often associated with neurodegeneration. Epidemiology The prevalence of BSPDC is not known, however it is very rare and fewer than 200 cases have been reported. BSPDC is more common in men (male:female ratio 2:1). Clinical description BSPCD can be asymptomatic. Symptomatic forms usually manifest in the fourth decade of life, whereas calcification may be found in the second decade. Patients present with progressive movement disorders, including parkinsonism, chorea, tremor, dystonia, athetosis and orofacial dyskinesia, ataxia and neuropsychiatric disorders including difficultly with concentration and memory, personality and/or behavior changes, and dementia.
- Basal Ganglia Calcification, Idiopathic, 5 OMIM
  
  A number sign (#) is used with this entry because idiopathic basal ganglia calcification-5 (IBGC5) is caused by heterozygous mutation in the PDGFB gene (190040) on chromosome 22q13. Description Idiopathic basal ganglia calcification-5 (IBGC5) is an autosomal dominant disorder characterized by progressive neurologic symptoms that are associated with brain calcifications mainly affecting the basal ganglia. Calcifications may also occur in the thalamus, cerebellum, or white matter. Affected individuals have motor symptoms, such as dyskinesias or parkinsonism, headache, cognitive impairment, and psychiatric manifestations, including apathy and depression. Some patients are asymptomatic. The age at symptom onset ranges from late childhood to adulthood; the disorder is progressive (summary by Keller et al., 2013).
- Basal Ganglia Calcification, Idiopathic, 2 OMIM
  
  Description Familial idiopathic basal ganglia calcification (IBGC) is characterized by bilateral basal ganglia calcification and has been associated with a variety of neurologic, cognitive, and psychiatric abnormalities. However, some affected individuals may be clinically asymptomatic (summary by Volpato et al., 2009). For a detailed phenotypic description and a discussion of genetic heterogeneity of IBGC, see IBGC1 (213600). Clinical Features Volpato et al. (2008) reported a large multigenerational Italian family from South Tyrol with IBGC. Twenty individuals over the age of 40 had positive CT scans revealing intracranial calcifications, with 14 having bilateral moderate to severe calcification of the basal ganglia, dentate nucleus, and subcortical white matter.
- Basal Ganglia Calcification, Idiopathic, 6 OMIM
  
  A number sign (#) is used with this entry because of evidence that idiopathic basal ganglia calcification-6 (IBGC6) is caused by heterozygous mutation in the XPR1 gene (605237) on chromosome 1q25. Description Idiopathic basal ganglia calcification is an autosomal dominant neurodegenerative disorder characterized by adult onset of progressive neuropsychiatric and movement disorders, although some patients remain asymptomatic. Clinical features can include dystonia, parkinsonism, gait abnormalities, psychosis, dementia, and chorea. Brain imaging shows calcifications of the basal ganglia and other brain regions (summary by Legati et al., 2015). For a detailed phenotypic description and a discussion of genetic heterogeneity of IBGC, see IBGC1 (213600).
- Primary Familial Brain Calcification GARD
  
  Primary familial brain calcification (PFBC) is a neurodegenerative disorder characterized by calcium deposits in the basal ganglia, a part of the brain that helps start and control movement. The first symptoms often include clumsiness, fatigue, unsteady walking ( gait ), slow or slurred speech, difficulty swallowing (dysphagia) and dementia. Migraines and seizures frequently occur. Symptoms typically start in an individual's 30's to 40's but may begin at any age.The neuropsychiatric symptoms and movement disorders worsen over time. Mutations in the SLC20A2 , PDGFRB , and PDGFB genes have been found to cause PFBC. This condition is inherited in an autosomal dominant manner.
2016 Irkutsk Mass Methanol Poisoning Wikipedia

The two alcohols are similar in many respects and cannot readily be distinguished, and their contents differed from the labels on the bottles, which indicated that they contained ethanol [1] [11] [12] —specifically, "93 percent of ethyl alcohol, hawthorn extract, lemon oil , diethyl phthalate and glycerol ". [13] An investigation later revealed that the methanol was usually used in the local production of antifreeze . [7] According to early reports on 19 December, a total of 57 people were hospitalized, with 49 dying. [1] [11] The victims were described as being poor residents of the Novo-Lenino neighborhood in Irkutsk, all between the ages of 35 and 50. [12] [14] Subsequent reports increased the number affected: first to 55 deaths (with a total of 94 affected), [15] then 62 (with 107 affected), [16] [17] 77 (number of affected not given), [18] and 78. [7] The final death toll was 74, lowered from earlier reports after it was discovered that some of the deaths were the result of drinking too much of the non-fraudulent ethanol-based bath lotion. [7] A total of 123 people were hospitalized. [7] About a third of them were found in their homes, having died before being able to call for an ambulance. [19] Of the remainder, a problem in attempting to treat them was that fomepizole , a methanol antidote , is not certified for use in Russia and is therefore not available in the country's hospitals. [2] Overall, the victims included a doctor, teachers, nurses, and drivers; The New York Times described the majority as holding "steady if low-paying jobs". [2] [19] Aftermath [ edit ] In the immediate aftermath of the poisoning, a state of emergency was declared. [7] Twenty-three people involved in the production of the lotion were arrested by Russian authorities, many of which were local vendors who sold the product, and one senior regional government official for the greater Siberian region being charged with negligence. [18] [20] About 500 litres (130 US gal) of remaining counterfeit lotion were seized from the underground facility where it had been produced, [1] and a few days later 13,500 litres (3,600 US gal) of methanol-containing liquid was seized from a warehouse in Irkutsk. [21] A further five people were arrested in January 2017, charged with selling and publicizing surrogate alcohol. [22] After the incident, a spokesperson for Russian president Vladimir Putin called it a "terrible tragedy", [1] blaming it on a failing of "supervisory bodies", [2] and added: "What happened in Irkutsk was a tremendous tragedy. ... The latter's legalization had been mooted prior to the poisoning. [7] Furthermore, the minimum legal price of vodka was lowered in both January and May 2017. [7] Individuals interviewed by a New York Times reporter in February 2017 were skeptical that any measures would be successful in significantly impacting illegal alcohol sales, given that it was such a high percentage of the total market for alcohol. [2] Indeed, vendors in Irkutsk reported that sales of surrogate alcohols did not decline after the poisoning. [7] Still, Rospotrebnadzor announced at the end of January that the country had seen its first decline in monthly alcohol poisoning deaths in five years. [29] See also [ edit ] List of methanol poisoning incidents Prohibition in the Russian Empire and the Soviet Union References [ edit ] ^ a b c d e f g h Isachenkov, Vladimir (19 December 2016).
Retinoblastoma Wikipedia

"Retinoblastoma". GeneReviews . Seattle, WA: University of Washington. PMID 20301625 . ^ Parsam Ali MJ, Parsam VL, Honavar SG, et al. (2010).

RB1, MDM4, BCOR, TP53, BRCA2, CHEK2, PIK3CD, CDK4, CDK2, PCNA, PIK3CA, PIK3CB, H3P10, PAX6, TMED7-TICAM2, PSMD9, PTEN, TMED7, CASP3, RBL1, PIK3CG, CDK6, FANCM, CDKN1A, EGFR, ESD, CTNNB1, ESR1, CRX, IFI27, TICAM2, EPCAM, MDM2, TCHP, MYC, CDKN2B, CDKN2A, MYCN, CDKN1B, RBL2, E2F1, BCL2, ZNRD2, VEGFA, NOLC1, DCTN6, PRDM2, AKT1, CCND1, RAB3GAP1, H3P23, MAPK1, E2F3, PRB2, ERBB2, RBBP7, CIB1, FOXM1, TGFB1, RBP3, MTOR, CCNE1, MIR34A, KRAS, HMGB1, ATM, SLC12A9, HIF1A, DDX1, CDK1, GRAP2, KIF14, BDNF, MTDH, RNF19A, AIMP2, HDAC1, MAP2K7, CRK, ABCB1, POLDIP2, AHSA1, PLK1, BRAF, UHRF1, EZH2, MAPK14, TERT, SERPINF1, RBBP4, GRB10, FGF2, MIR17HG, FOXO1, MIR204, RNF40, RB1CC1, NXT1, CXCR4, MIR140, MIR106B, RGCC, HPGDS, S100A4, SAI1, MAPK8, SKP2, STAT3, SYK, TFF1, SUB1, RBBP9, DEK, MMP9, MRPL28, HMGA2, RBM45, IL6, POLD1, ABCG2, CDKN3, CD44, AR, CDKN1C, APC, CHEK1, H3P9, CDH11, CRH, FGFR3, COMMD3-BMI1, MIR183, MIR506, MAPK3, SIRT1, MIR21, SP1, ARR3, MIR215, SMAD2, SMARCA4, SOX2, MIR18A, MIR17, IGF1, H2AX, KLF6, MIR137, INTS6, RAF1, CAV1, RBBP6, BMI1, MIR613, HMGA1, HRAS, TNF, TFRC, CDKN2C, VDR, ACTB, DHFR, MGMT, PROM1, LMNA, E2F4, MTHFR, TSC2, XIST, EPHB2, SNHG16, ZNF266, OAT, OTX2, HOTAIR, ELOF1, MSH2, RUNX2, PTGS2, PTPN14, ODC1, AFAP1-AS1, ATRAID, MKI67, CDH13, CEACAM5, MCL1, BRS3, BRCA1, ACKR3, RCVRN, PSG2, NRAS, CCND2, MEG3, PIK3R1, LAMTOR1, AD12, MIB1, SEMA6A, PPARG, NME1, EAF2, CDC25C, NFKB1, MYBL2, CCNB1, MXI1, MMUT, MTR, CDK5, PDK1, MED4, PPM1D, TYMS, NEK6, UBE2I, APRT, VHL, CXCR6, WT1, LINC02210-CRHR1, MAFK, CLLS2, ADRA2B, PIK3R3, SGSM3, KHSRP, PSMG1, ZNF197, BECN1, ADRA1A, ADCYAP1R1, ADCYAP1, HDAC9, LPAR2, EEF1E1, RECQL4, TNFRSF1B, IL24, PSIP1, RASSF1, RFC1, RHO, SIGLEC7, ROCK1, PRDM1, SH2B1, BGN, SMAD4, BAX, SLC19A1, SMARCA1, SMARCB1, SSTR4, DICER1, SUV39H1, SYP, TAZ, TCF3, ATR, ATF3, H3P12, ASMT, TFF3, CCL2, MCM2, UCA1, IDH1, CEACAM7, IGFBP3, MIR34B, CUX1, IL2RB, CEACAM3, ILK, MIR98, MIR99A, ELN, FOLH1B, KIT, ELF1, EIF4E, EFNA2, IFNB1, MIR22, E2F5, MALAT1, MIR145, GPR42, H1-0, HCLS1, MAD2L1, MIR182, MIR186, FOS, HSPA4, FOLH1, HSP90AA1, FGFR1, CRHR1, FASN, FAP, EDNRA, NEAT1, MIR361, MIR449A, E2F2, CFL1, STMN1, ACVR1C, LGALS3, DNMT1, NEK7, TPPP2, DUSP2, MIR504, MIR485, ELL2, NT5C2, KLRK1, MIR200C, MIR203A, ZHX2, AKAP12, GDF3, KDM4A, MIR503, MIR212, WIF1, CEP57, HDAC4, MIR373, MIR340, FSTL1, MELK, NUP205, USP22, FBXW11, BRD4, MIR382, ATG5, MIR181A2, MIR181C, SH3BP4, MIR376A1, NUP62, CCNDBP1, BAG3, EI24, MIR184, BCAR1, CRB1, ZFPM2, H3P17, MIR188, MIR191, KLRC4-KLRK1, SRGAP2, MIR198, MIR19B1, ZEB2, CDC37, MIR223, TOPBP1, MIR330, MIR93, KHDRBS1, MVP, NUP153, ARID3B, MIR202, NKILA, RN7SL263P, THOC1, MTCO2P12, LOC110806263, UBE2C, ABCB6, MIR338, H3P13, DNM1L, PRMT5, APC2, AKAP8, MIR491, GPC6, ABCC4, CTCF, PANDAR, EBP, MIR34C, MIR221, MPZL2, MIR498, MIR25, COPS5, TMED10, CBX1, MED4-AS1, LYVE1, MIR29A, MIR320A, C1QL1, ZBTB5, SRCAP, MIR497, MIR495, MIR492, PTGES3, POU5F1P3, MIR433, BANCR, CADM1, CYTOR, AIPL1, CEMIP, KIF13A, BCORL1, CADM3, A2ML1, MIR758, EP400, PPM1K, MRTFA, MIR638, FEZF1-AS1, SCYL1, DANCR, CBLL2, TP73-AS1, KIF4A, RASSF6, DDX53, RALGAPB, MIR675, PNPLA2, TIGAR, MIR422A, PCBP4, TBCEL, HOXA11-AS, CADM2, PAG1, H19, PRDM16, SOX17, SMURF2, WNK1, MED23, SCGB3A1, POTEF, MIR655, KRT8P3, WNT3A, DIXDC1, EAF1, AZIN2, LRG1, PCAT4, PRAP1, CDCA7, RGPD2, ARHGAP24, CDT1, DRAM2, COL18A1, ULBP2, E2F8, LIN28A, ZNF329, SLCO6A1, MUL1, CDC73, PIWIL4, SLC25A23, CDKN2B-AS1, MIR665, PGP, LUCAT1, CBR3-AS1, TBPL2, ANAPC2, FAM238C, CD274, THORLNC, CCAT1, LINC01194, ATAD2, GTF2H5, MIRLET7B, MIR106A, MIR598, MIR125A, PDCD4, BBC3, MIR130B, MIR132, TCL6, CYFIP2, LATS2, MIR139, POU5F1P4, SIN3A, MICA, WWTR1, TMX2-CTNND1, KCNIP3, MIR3613, STX17, SOST, TMED10P1, MIR874, MIR365B, ZCCHC2, PGPEP1, TRPM7, OTUD4, RTEL1, LIN9, ARID4B, PIAS4, IL23A, CINP, TRIM59, GADL1, RBMY2DP, ENDO1, NOL7, MIR3163, GSTK1, MZB1, DCTN4, LINC00328, RBMY1D, PHF20L1, IL31, MIR3619, MIR448, AANAT, LIPG, HMGB2, GTF2H1, HDGF, HELLS, HIC1, HLA-A, HLF, HLA-G, HNF4A, GLI3, HOXB5, AGFG2, HSF1, HSPB1, HSPB2, HTR2A, ICAM1, GPI, GLI1, MMP2, FOXO3, F9, PTK2B, FANCB, FGF1, FGF9, FGF13, FGFR2, FOLR1, GLB1, FUT7, GAS6, GATA1, GDF10, GFAP, GHRHR, GJA3, ID2, IDH2, IFNG, LOX, LCP1, LDHA, LEP, LIG4, LMNB1, LMO2, LMO7, LY9, IGF1R, SMAD3, MCM6, MCM7, MEFV, MEN1, MFAP1, MLH1, LASP1, L1CAM, KRT19, KRT8, IGF2, IGF2R, IGFBP2, IL1A, IL1B, IL2, IL3, IL17A, INSM1, IRS1, ANOS1, CD82, KDR, KNG1, KPNA2, F3, EWSR1, EVPL, CCK, FOXL2, CAPN5, CA9, CASP5, CASP8, RUNX3, CBR3, CCND3, CDX1, CCNG1, TNFRSF8, CD40, CDC25A, CDC25B, CDH17, CDK9, BNIP3, BMP4, BCL6, BCL3, ABL1, ACY1, ADRB3, GRK3, JAG1, AGT, ALB, ALK, APEX1, APOC2, APOD, FAS, ASS1, ATRX, BAG1, CDKN2D, CEBPD, MECOM, ELAVL2, DCT, DDIT3, DDX3X, DDX5, DNMT3A, DNMT3B, DUSP1, ELF4, CETN2, ELK3, ENDOG, ENO1, ENO2, EPHB1, ERCC2, ESR2, DCC, DAXX, DAP, DAB1, CGA, CHRM3, CHRM5, CKS1B, CLU, PLK3, COX8A, CLDN7, CREBBP, CRHR2, CRYAB, CSE1L, CSF2, CTNND1, CXADR, MMP1, MMP3, ADIPOQ, TP73, TGFB2, TGFBI, TGFBR2, THBS1, THY1, TIAM1, TIMP1, TPM3, TFF2, TPO, TPT1, TRAF3, TYR, TYRO3, UBC, UBE2B, TG, TFDP1, MMP15, SST, SLPI, SNCG, SOAT1, SOD1, SOX4, ABCA4, SREBF1, STAT1, TFAP2B, STC1, TACR1, TBX1, TBX5, ZEB1, TCF19, TFAP2A, UCN, KDM6A, TRPV1, HSPB3, SUCLA2, CDK5R1, SQSTM1, SLC5A6, CCNA1, PHOX2B, MBD2, TRPA1, VRK1, PRC1, CLDN8, CLDN1, ARHGEF1, EXO1, PIWIL1, TRIP11, NAPG, FADD, TNFSF10, ADAM19, WEE1, WNT1, WNT10B, XPO1, XRCC4, KMT2D, XRS, SLC7A5, COIL, MKKS, CUL2, CASK, STC2, TP63, MBTPS1, SLC6A2, SLC5A5, SRSF3, PDE3B, NOTCH1, NPY, NOVA2, NPM1, NRF1, NTRK1, PRKN, PDGFA, PLAU, SLC26A4, PECAM1, PFDN4, PGF, PI3, PIGF, PLAG1, NOS2, NKTR, NGF, NFE2L1, MPG, MSN, MT1JP, MTAP, COX2, MTTP, MYB, MYCL, MYOD1, NCAM1, NEDD9, NEK2, NEUROG1, NF1, NF2, PLAGL1, PLXNA2, SATB1, BRD2, OPN1LW, REL, REG1A, UPF1, RET, TRIM27, RNASE3, ABCE1, POMC, ROS1, RPE, RPL34, RPS6KB1, RPS27A, RRAS, SAG, RBP2, RBP1, RBMY1A1, RBBP8, PON1, POU5F1, PPARD, PPP1CA, PTPA, PRB1, PRKCB, RELN, PSMD10, PTPN12, PVT1, RAD51, RARA, RASGRF1, KDM5A, SRC
- Retinoblastoma GeneReviews
  
  Summary Clinical characteristics. Retinoblastoma is a malignant tumor of the developing retina that occurs in children, usually before age five years. Retinoblastoma develops from cells that have cancer-predisposing variants in both copies of RB1 . Retinoblastoma may be unifocal or multifocal. About 60% of affected individuals have unilateral retinoblastoma with a mean age of diagnosis of 24 months; about 40% have bilateral retinoblastoma with a mean age of diagnosis of 15 months. Heritable retinoblastoma is an autosomal dominant susceptibility for retinoblastoma. Individuals with heritable retinoblastoma are also at increased risk of developing non-ocular tumors.
- Retinoblastoma Mayo Clinic
  
  Overview Retinoblastoma is an eye cancer that begins in the retina — the sensitive lining on the inside of your eye. Retinoblastoma most commonly affects young children, but can rarely occur in adults. Your retina is made up of nerve tissue that senses light as it comes through the front of your eye. The retina sends signals through your optic nerve to your brain, where these signals are interpreted as images. A rare form of eye cancer, retinoblastoma is the most common form of cancer affecting the eye in children.
- Retinoblastoma GARD
  
  Retinoblastoma (RB) is a rare type of eye cancer in the retina that typically develops before the age of 5. It usually affects only one eye, but 1/3 of children with RB develop cancer in both eyes. The first sign is typically a visible whiteness in the pupil called "cat's eye reflex" or leukocoria , which is particularly noticeable in photographs taken with a flash. Other signs and symptoms include strabismus; persistent eye pain, redness or irritation; and blindness or poor vision in the affected eye(s). Retinoblastoma is caused by mutations in the RB1 gene. In about 60% of people with retinoblastoma, mutations are not inherited and occur only in retinal cells.
- Retinoblastoma MedlinePlus
  
  Retinoblastoma is a rare type of eye cancer that usually develops in early childhood, typically before the age of 5. This form of cancer develops in the retina, which is the specialized light-sensitive tissue at the back of the eye that detects light and color. In children with retinoblastoma, the disease often affects only one eye. However, one out of three children with retinoblastoma develops cancer in both eyes. The most common first sign of retinoblastoma is a visible whiteness in the pupil called "cat's eye reflex" or leukocoria .
- Retinoblastoma OMIM
  
  A number sign (#) is used with this entry because hereditary retinoblastoma is caused by a heterozygous germline mutation on one allele and a somatic mutation on the other allele of the RB1 gene (614041) on chromosome 13q14. See also the chromosome 13q14 deletion syndrome (613884) in which retinoblastoma is a feature. Description Retinoblastoma (RB) is an embryonic malignant neoplasm of retinal origin. It almost always presents in early childhood and is often bilateral. Spontaneous regression ('cure') occurs in some cases. The retinoblastoma gene (RB1) was the first tumor suppressor gene cloned.
- Retinoblastoma Orphanet
  
  A rare eye tumor disease representing the most common intraocular malignancy in children. It is a life threatening neoplasia but is potentially curable and it can be hereditary or non hereditary, unilateral or bilateral. Epidemiology Retonoblastoma (RB) has an incidence of approximately 1/15-20,000 in Europe. Clinical description RB manifests most often in young children (90% of cases <3 years old). Early clinical signs are leukocoria and strabismus. RB is most often painless and children rarely complain of visual impairment despite its rapid progression towards loss of vision in the affected eye.
Toxic Oil Syndrome Wikipedia

It was sold as " olive oil " by street vendors at weekly street markets, and was used on salads and for cooking.
- Toxic Oil Syndrome Orphanet
  
  Toxic oil syndrome is a rare intoxication, due to consumption of a rapeseed oil denatured with aniline 2%, characterized by generalized vascular lesions affecting all organs and vessels (including veins and arteries) and presenting with severe incapacitating myalgias, marked peripheral eosinophilia and pulmonary infiltrates. Epidemiology Spain is the only country to have reported cases of this disease in the spring of 1981 and patients resided in fourteen Central and North West provinces. Almost 20,000 people have been recorded, with women under the age of 40 years being more frequently and severely affected than men. Clinical description While TOS can affect all organs and vessels (such as the lungs, peripheral nerves, muscles, skin, digestive tract, liver and pancreas) the main outcome is fibrosis of the lumen of the vessels, skin, peripheral nerves and intestines. The disease course can be characterized by three clinical phases: i) the acute phase (lasting approximately 2 months) with a presentation of severe lung edema, eosinophilia, rash and myalgia, ii) the intermediate phase (2-3 months) with dysphagia, cramps, severe myalgia, skin edema, pulmonary hypertension, paresthesia, major vessel thromboembolism and severe weight loss, iii) the chronic phase, where the skin edema evolves to scleroderma, and the neuromuscular manifestations into paresis and paralysis due to polyneuropathy.
Silent Sinus Syndrome Wikipedia

AJR Am J Roentgenol . 178 (2): 503–6. doi : 10.2214/ajr.178.2.1780503 . PMID 11804926 . Full text Numa WA, Desai U, Gold DR, Heher KL, Annino DJ (2005).
- Silent Sinus Syndrome Orphanet
  
  Silent sinus syndrome is characterised by adult-onset progressive enophthalmos due to collapse of some or all of the maxillary sinus walls. Epidemiology Its prevalence is unknown but around 100 cases have been reported in the literature so far. Clinical description The progressive enophthalmos may occasionally be associated with cheek pain, diplopia and blurred vision. Patients sometimes report a history of remote episodes of sinusitis. The syndrome may be idiopathic or occur following a bony orbital decompression resulting from Graves' ophthalmopathy or orbital floor fracture. Etiology The underlying mechanism involves obstruction of the maxillary antrum aeration followed by generation of negative antral pressure.
Blastocystis Hominis Mayo Clinic

More specifically, avoid: Food from street vendors Unpasteurized milk and dairy products, including ice cream Raw or undercooked meat, fish, shellfish or eggs Food at room temperature, such as sauces and buffet offerings Fresh greens; foods that can't be peeled, such as berries; fruits or vegetables that you did not peel yourself Frozen pops and flavored ice Dishes or condiments made with uncooked fruits or vegetables Safe drinking-water tips If you're visiting a country with poor sanitation or possible unsafe drinking water, use the following tips: Avoid unsterilized water — from tap, well or stream.
Vibration White Finger Wikipedia

Tools are given an Exposure Action Value (EAV, the time which a tool can be used before action needs to be taken to reduce vibration exposure) and an Exposure Limit Value (ELV, the time after which a tool may not be used). [ citation needed ] In the United States, the National Institute for Occupational Safety and Health published a similar database where values for sound power and vibrations for commonly found tools from large commercial vendors in the United States were surveyed.
Norovirus Infection Mayo Clinic

If you're traveling to areas with a high risk of norovirus infection, consider eating only cooked foods, drinking only hot or carbonated beverages, and avoiding food sold by street vendors. To help prevent norovirus infection spread, during illness and for 2 to 3 days after your symptoms end: Avoid contact with others as much as possible.

FUT2, ABO, NLRP3, GSDMD, NBEAL1, NOD2, FLVCR2, ATG16L1, TLR7, G3BP1, USP14, TLR4, SNCA, CCL5, RAG2, IL18, IL2RG, CD300LF
Trilateral Retinoblastoma Wikipedia

PMID 10561222 . ^ De Jong MC, Kors WA, De Graaf P, Castelijns JA, Kivelä T, Moll AC (September 2014).

RB1, MYCN, STK11
Traumatic Cardiac Arrest Wikipedia

References [ edit ] ^ Hunt PA, Greaves I, Owens WA (January 2006). "Emergency thoracotomy in thoracic trauma-a review".