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Human African Trypanosomiasis (HAT), also called sleeping sickness, is a vector-borne parasitic disease caused by a protozoa of the Trypanosoma genus transmitted by the bite of a tsetse fly (genus Glossina ), that is found under its chronic form (average duration of 3 years) in western and central Africa (in case of the T. brucei gambiense sub-species), and under its acute form (lasting from few weeks to 6 months) in eastern and southern Africa (in case of the T. brucei rhodesiense sub-species). HAT comprises an initial hemo-lymphatic stage characterized by fever, weakness, musculoskeletal pain, anemia, and lymphadenopathy, along with dermatologic, cardiac and endocrine complications or hepatosplenomegaly, followed by a meningo-encephalitic stage characterized by neurologic involvement (sleep disturbances, psychiatric disorders, seizures) that progresses, in the absence of treatment, towards a fatal meningoencephalitis.
Glycogen storage disease type 0 Glycogen storage disease type 0 has defect in glycogen synthase Specialty Medical genetics Glycogen storage disease type 0 is a disease characterized by a deficiency in the glycogen synthase enzyme (GSY). ... Uncoordinated eye movements, disorientation, seizures, and coma may accompany severe episodes. [2] Glycogen-storage disease type 0 affects only the liver. ... Patients with glycogen-storage disease type 0 have normal-to-increased response to glucagon in the fed state, with hyperglycemia and lactic acidemia. [2] Laboratory Studies [ edit ] Serum glucose levels are measured to document the degree of hypoglycemia. ... The identification of asymptomatic and oligosymptomatic siblings in several glycogen-storage disease type 0 families has suggested that glycogen-storage disease type 0 is underdiagnosed. [2] Mortality/Morbidity [ edit ] The major morbidity is a risk of fasting hypoglycemia, which can vary in severity and frequency. Major long-term concerns include growth delay, osteopenia, and neurologic damage resulting in developmental delay, intellectual deficits, and personality changes. [2] Sex [ edit ] No sexual predilection is observed because the deficiency of glycogen synthetase activity is inherited as an autosomal recessive trait. [2] Age [ edit ] Glycogen-storage disease type 0 is most commonly diagnosed during infancy and early childhood. [2] References [ edit ] ^ Orho M, Bosshard NU, Buist NR, et al.
Neonatal Skin: Structure and Function . CRC Press. pp. 67–. ISBN 978-0-8247-0887-0 . ^ Mariagrazia Stracquadanio; Lilliana Ciotta (20 April 2015). ... A Woman Doctor's Guide to Skin Care: Essential Facts and Information on Keeping Skin Healthy . Hyperion. ISBN 978-0-7868-8100-0 . ^ Sarah Bekaert (2007). ... CUP Archive. pp. 321–. ISBN 978-0-521-22673-8 . ^ Raphael Rubin; David S. ... Lippincott Williams & Wilkins. pp. 1195–. ISBN 978-0-7817-1750-2 . ^ Annual Reports in Medicinal Chemistry . Academic Press. 3 October 1994. pp. 1994–. ISBN 978-0-08-058373-0 . Further reading [ edit ] Shukla, G.
Nevus comedonicus (also known as a " comedo nevus " [1] ) is characterized by closely arranged, grouped, often linear, slightly elevated papules that have at their center keratinous plugs resembling comedones . [2] : 634 [3] : 774 See also [ edit ] Nevus comedonicus syndrome Skin lesion List of cutaneous conditions References [ edit ] ^ Rapini, Ronald P.; Bolognia, Jean L.; Jorizzo, Joseph L. (2007). Dermatology: 2-Volume Set . St. Louis: Mosby. p. 1673. ISBN 1-4160-2999-0 . ^ James, William; Berger, Timothy; Elston, Dirk (2005). Andrews' Diseases of the Skin: Clinical Dermatology . (10th ed.). Saunders. ISBN 0-7216-2921-0 . ^ Freedberg, et al. (2003). Fitzpatrick's Dermatology in General Medicine . (6th ed.). McGraw-Hill. ISBN 0-07-138076-0 . This Epidermal nevi, neoplasms, cysts article is a stub .
By 10 years of age, his lesion extended inferiorly to the posteromedial right thigh, and consisted of a 10 cm by 2 cm plaque composed of several atrophic cribiform scars and scattered pink comedones, with another smaller plaque below it.
A rare, syndromic nevus characterized by the association of typically unilateral, closely arranged, linear, slightly elevated, multiple, nevus comedonicus lesions located usually on the face, neck, trunk or limbs (with or without a central, dark, firm, hyperkeratotic plug and secondary acneiform lesions) with extracutaneous ocular, skeletal, and/or central nervous system abnormalities, such as ipsilateral cataract, corneal erosion, poly-/syndactyly, absent fifth finger, scoliosis, vertebral defects, corpus callosum agenesis, seizures, interhemispheric cyst, intellectual deficiency, and/or developmental delay.
Nevus comedonicus syndrome Specialty Dermatology Nevus comedonicus syndrome is a skin condition characterized by a nevus comedonicus associated with cataracts , scoliosis , and neurologic abnormalities. [1] : 635 [2] : 776 [3] : 849 See also [ edit ] Epidermal nevus syndrome Cutaneous conditions References [ edit ] ^ James, William; Berger, Timothy; Elston, Dirk (2005). Andrews' Diseases of the Skin: Clinical Dermatology . (10th ed.). Saunders. ISBN 0-7216-2921-0 . ^ Freedberg, et al. (2003). Fitzpatrick's Dermatology in General Medicine . (6th ed.). McGraw-Hill. ISBN 0-07-138076-0 . ^ Rapini, Ronald P.; Bolognia, Jean L.; Jorizzo, Joseph L. (2007). Dermatology: 2-Volume Set . St. Louis: Mosby. ISBN 1-4160-2999-0 .
Lippincott Williams & Wilkins. p. 1152. ISBN 0-7817-7513-2 . ^ Pryse-Phillips, William (2003). ... Neurology in clinical practice . 2 (4th ed.). Taylor & Francis. p. 2074. ISBN 0-7506-7469-5 . ^ Bernstein, Carolyn; Elaine McArdle (2009). ... Lippincott Williams & Wilkins. p. 2695. ISBN 0-7817-5777-0 . ^ Miller, Neil R.; Frank Burton Walsh; Valérie Biousse; William Fletcher Hoyt (2005). ... McGraw-Hill Professional. p. 127. ISBN 0-07-105467-7 . ^ G. D. Schott (2007).
Jaypee Brothers Publishers. 2007. pp. 347–. ISBN 978-81-8061-996-0 . ^ Leonard J. Deftos (1 January 1998). ... Rowman & Littlefield Publishers. pp. 73–. ISBN 978-0-7591-2332-8 . ^ Vasan; R.S. (1 January 1998). ... Biochemistry and Function of Sterols . CRC Press. pp. 26–27. ISBN 978-0-8493-7674-0 . ^ Michael Crocetti; Michael A. ... Lippincott Williams & Wilkins. pp. 564–. ISBN 978-0-7817-3770-8 . ^ W. Steven Pray (2006). ... Elsevier Health Sciences. pp. 1281–. ISBN 978-0-323-08678-3 . ^ Guy I. Benrubi (28 March 2012).
Pili annulati Specialty Medical genetics Pili annulati (also known as " ringed hair " [1] ) is a genetic trait in which the hair seems ‘banded‘ by alternating segments of light and dark color when seen in reflected light. [1] [2] : 767 [3] : 640 [4] See also [ edit ] Pili pseudoannulati List of cutaneous conditions References [ edit ] ^ a b Rapini, Ronald P.; Bolognia, Jean L.; Jorizzo, Joseph L. (2007). Dermatology: 2-Volume Set . St. Louis: Mosby. ISBN 1-4160-2999-0 . ^ James, William; Berger, Timothy; Elston, Dirk (2005). Andrews' Diseases of the Skin: Clinical Dermatology . (10th ed.). Saunders. ISBN 0-7216-2921-0 . ^ Freedberg, et al. (2003). Fitzpatrick's Dermatology in General Medicine . (6th ed.). McGraw-Hill. ISBN 0-07-138076-0 . ^ http://www.omim.org/entry/180600 External links [ edit ] Hair Transplant Classification D ICD - 10 : Q84.1 ( ILDS Q84.110) ICD - 9-CM : 757.4 MeSH : C537187 v t e Congenital malformations and deformations of skin appendages Nail disease Anonychia Leukonychia Pachyonychia congenita / Onychauxis Koilonychia Hair disease hypotrichosis /abnormalities: keratin disease Monilethrix IBIDS syndrome Sabinas brittle hair syndrome Pili annulati Pili torti Uncombable hair syndrome Björnstad syndrome Giant axonal neuropathy with curly hair hypertrichosis : Zimmermann–Laband syndrome This condition of the skin appendages article is a stub .
Snell and Foley (1932) described 9 affected persons in 4 generations with an instance of male-to-male transmission, consistent with autosomal dominant inheritance. Mapping By linkage analysis of 2 unrelated families with autosomal dominant inheritance of pili annulati, Green et al. (2004) found significant linkage to a 9.2-cM region on chromosome 12q between D12S367 and D12S1723 (maximum combined lod score of 4.78 at D12S1723).
Pili annulati is a hair disorder. In pili annulati, affected hair has a pattern of light and dark banding. People with pili annulati may describe their hair as "striped" or as having silvery beads. Pili annulati typically involves 20-80% of scalp hair, however it can involve facial and body hair as well. Affected hairs may be more prone to breakage. Pili annulati can present in infancy, childhood, or later in life. It can be seen with the naked eye, however it may be more difficult to see in people with dark hair.
Pili annulati is an isolated, benign hair shaft abnormality, usually presenting after the age of 2 and affecting the hair of the scalp or very rarely beard, axillary, or pubic hair, that is characterized by a banded or speckled appearance due to alternating light bands (corresponding to air-filled cavities within the cortex of the affected hair shafts) and dark bands.
Pili torti Menkes disease Specialty Medical genetics Pili torti (also known as "Twisted hairs") is characterized by short and brittle hairs that appear flattened and twisted when viewed through a microscope. [1] : 638 [2] : 764 [3] This phenotype is noted in Menkes disease , and Lichen Planopilaris Pili torti can also occur after use of retinoids, such as isotretinoin See also [ edit ] List of cutaneous conditions Crandall syndrome References [ edit ] ^ Freedberg, et al. (2003). Fitzpatrick's Dermatology in General Medicine . (6th ed.). McGraw-Hill. ISBN 0-07-138076-0 . ^ James, William; Berger, Timothy; Elston, Dirk (2005). Andrews' Diseases of the Skin: Clinical Dermatology . (10th ed.). Saunders. ISBN 0-7216-2921-0 . ^ Rapini, Ronald P.; Bolognia, Jean L.; Jorizzo, Joseph L. (2007). Dermatology: 2-Volume Set . St. Louis: Mosby. ISBN 1-4160-2999-0 .
Pili torti is a hair shaft abnormality characterized by flat hair that is twisted at irregular intervals. Hair is normal at birth but progressively stops growing long and becomes fragile. Pili torti can be isolated or occur in association with syndromes such as Menkes disease or Bazex syndrome (see these terms).
Clinical Features Pili torti was first described and named by Ronchese (1932), who observed 2 affected Italian sisters. The proband had dull blonde scalp hair that was 'lank and frizzy, with a wild, bushy appearance.' ... Gedda and Cavalieri (1963) reported 6 families; in 2 families, the parents were related, and in 2 others, 2 sibs were affected.
Pili torti is a rare hair condition characterized by fragile hair. In pili torti hair has a flattened shaft with clusters of narrow twists at irregular intervals. Some cases may be inherited in autosomal dominant or autosomal recessive patterns, while others are acquired. In the inherited form, symptoms tend to be present from early childhood. It can occur alone or as part of other diseases like ectodermal dysplasias , Menke disease , Bjornstand syndrome , or Bazex syndrome . Acquired cases of pili torti may be associated with anorexia nervosa , malnutrition, oral retinoid treatment, or inflammatory scalp conditions (e.g., cutaneous lupus erythematousus ).
Bifid penis A bifid penis (or double penis ) is a rare congenital defect where two genital tubercles develop. [1] [2] Many male marsupials such as the kangaroo naturally have a bifid penis, with left and right prongs that they insert into multiple vaginal canals simultaneously. [3] See also [ edit ] Diphallia Meatotomy Penile subincision References [ edit ] ^ Lewis, Keeta D. & Bear, Bonnie (2002). ... Elsevier Health Sciences. p. 161. ISBN 978-0-7216-8521-2 . CS1 maint: uses authors parameter ( link ) ^ Jones, Richard E. & López, Kristin H. (2006). Human reproductive biology (3rd ed.). Academic Press. p. 145 . ISBN 978-0-12-088465-0 . CS1 maint: uses authors parameter ( link ) ^ Vaughan, Terry A.; et al. (2010). ... Jones & Bartlett. p. 389. ISBN 978-0-7637-6299-5 . This genetic disorder article is a stub .
A rare, non-syndromic, urogenital tract malformation characterized by complete or partial penile duplication, ranging from only glans duplication to the presence of two penis shafts with either one (i.e. bifid phallus) or two (i.e. true diphallia) corpora cavernosum in each. Additional anomalies, such as urethra duplication, an abnormal voiding pattern, hypo- or epispadias, bifid/ectopic scrotum, bladder exstrophy or duplication, are frequently associated, but it may also present as an isolated anomaly. In severe cases, pubic symphysis diastasis, imperforate or duplicated anus, colon/ rectosigmoidal duplication, inguinal hernia and vertebral anomalies may be observed.
He had a single normal bladder and ureter. (Refer Figure 2.) (D) Partial bifid diphallia without associated anomalies A 15-year-old boy, [14] apart from having two penises, had normal external genitalia. ... International Journal of Genetics and Molecular Biology . 2 (10): 207–216. ^ Sharma KK, Jain R, Jain SK, Purohit A (2000). ... "How to make a primordial germ cell" . Development . 141 (2): 245–252. doi : 10.1242/dev.098269 . ... Pediatric Radiology . 37 (9): 851–862. doi : 10.1007/s00247-007-0495-0 . PMC 1950215 . PMID 17572890 . ^ Elumalai, Ganesh (2017). " " HYPOSPADIAS" ITS EMBRYOLOGICAL BASIS AND CLINICAL IMPORTANCE" . ... National Center for Biotechnology Information , US National Library of Medicine , National Institutes of Health Fleishman, Cooper (May 15, 2012). "Man With 2 Penises, Woman With 2 Vaginas Were the Sexiest Couple of the 19th Century" .
While diarrhea is common in people with SARS, the fecal–oral route does not appear to be a common mode of transmission. [9] The basic reproduction number of SARS-CoV, R 0 , ranges from 2 to 4 depending on different analyses. ... As a result of quarantine procedures, some of the post-SARS patients have been documented as suffering from post-traumatic stress disorder (PTSD) and major depressive disorder . [31] [32] Epidemiology [ edit ] Main article: 2002–2004 SARS outbreak SARS was a relatively rare disease; at the end of the epidemic in June 2003, the incidence was 8,422 cases with a case fatality rate (CFR) of 11%. [4] The case fatality rate (CFR) ranges from 0% to 50% depending on the age group of the patient. [9] Patients under 24 were least likely to die (less than 1%); those 65 and older were most likely to die (over 55%). [33] As with MERS and COVID-19 , SARS resulted in significantly more deaths of males than females. 2003 Probable cases of SARS – worldwide Probable cases of SARS by country or region, 1 November 2002 – 31 July 2003 [34] Country or region Cases Deaths Fatality (%) China [a] 5,327 349 6.6 Hong Kong 1,755 299 17.0 Taiwan [b] 346 73 [35] [36] 21.1 Canada 251 43 17.1 Singapore 238 33 13.9 Vietnam 63 5 7.9 United States 27 00 Philippines 14 2 14.3 Thailand 9 2 22.2 Germany 9 00 Mongolia 9 00 France 7 1 14.3 Australia 6 00 Malaysia 5 2 40.0 Sweden 5 00 United Kingdom 4 00 Italy 4 00 Brazil 3 00 India 3 00 South Korea 3 00 Indonesia 200 South Africa 1 1 100.0 Colombia 1 00 Kuwait 1 00 Ireland 1 00 Macao 1 00 New Zealand 1 00 Romania 1 00 Russia 1 00 Spain 1 00 Switzerland 1 00 Total excluding China [a] 2,769 454 16.4 Total (29 territories) 8,096 774 9.6 ^ a b Figures for China exclude Hong Kong and Macau, which are reported separately by the WHO . ^ After 11 July 2003, 325 Taiwanese cases were 'discarded'. ... Journal of Virological Methods . 128 (1–2): 21–8. doi : 10.1016/j.jviromet.2005.03.021 . ... Emerging Infectious Diseases . 10 (2): 173–5. doi : 10.3201/eid1002.031038 . ... American Journal of Biomedical Science & Research . 6 (2): 001017. doi : 10.34297/AJBSR.2019.06.001017 . ^ Senior K (November 2003).
Overview Severe acute respiratory syndrome (SARS) is a contagious and sometimes fatal respiratory illness. severe acute respiratory syndrome (SARS) first appeared in China in November 2002. Within a few months, SARS spread worldwide, carried by unsuspecting travelers. SARS showed how quickly infection can spread in a highly mobile and interconnected world. On the other hand, a collaborative international effort allowed health experts to quickly contain the spread of the disease. There has been no known transmission of SARS anywhere in the world since 2004.
A rare pulmonary disease induced by SARS-CoV coronavirus infection, with a reported incubation period varying from 2 to 7 days. Patients present flu-like symptoms, including fever, malaise, myalgia, headache, diarrhoea, and rigors.
Erosive pustular dermatitis of the scalp Other names Erosive pustular dermatosis of the scalp [1] Specialty Dermatology Erosive pustular dermatitis of the scalp presents with pustules , erosions, and crusts on the scalp of primarily older Caucasian females, and on biopsy, has a lymphoplasmacytic infiltrate with or without foreign body giant cells and pilosebaceous atrophy. [2] : 650 [3] : 761 See also [ edit ] Skin lesion Cicatricial alopecia List of cutaneous conditions References [ edit ] ^ Rapini, Ronald P.; Bolognia, Jean L.; Jorizzo, Joseph L. (2007). Dermatology: 2-Volume Set . St. Louis: Mosby. ISBN 978-1-4160-2999-1 . ^ Freedberg, et al. (2003). Fitzpatrick's Dermatology in General Medicine . (6th ed.). McGraw-Hill. ISBN 0-07-138076-0 . ^ James, William; Berger, Timothy; Elston, Dirk (2005). Andrews' Diseases of the Skin: Clinical Dermatology . (10th ed.). Saunders. ISBN 0-7216-2921-0 . This condition of the skin appendages article is a stub .
Erosive pustular dermatosis of the scalp is a rare chronic inflammation of the scalp usually occurring in elderly women (>70 years old) and characterized by the development of painful pustules, shallow erosions, and crusting on atrophic skin that eventually result in cicatricial alopecia.
Nevus lipomatosus (cutaneous) superficialis ( NLS or NLCS , also known as "Nevus lipomatosis of Hoffman and Zurhelle" [1] ) is characterized by soft, yellowish papules or cerebriform plaques , usually of the buttock or thigh, less often of the ear or scalp, with a wrinkled rather than warty surface. [1] [2] : 625 It is usually congenital in origin or appears within the first three decades. [3] A pedunculated lipofibroma is a solitary variant of nevus lipomatosus superficialis . [3] It usually appears in adult life, and usually on the axilla, knee, ear, arm, scalp and the lower trunk. [3] Pedunculated lipofibroma, gross pathology Micrograph of pedunculated lipofibroma, low magnification, with dermal expansion of fatty tissue. ... The main differential diagnoses are acrochordon , seborrheic keratosis , intradermal melanocytic nevi , neurofibromas , verrucae and fibroepithelioma of Pinkus. [3] See also [ edit ] Skin lesion List of cutaneous conditions References [ edit ] ^ a b Rapini, Ronald P.; Bolognia, Jean L.; Jorizzo, Joseph L. (2007). Dermatology: 2-Volume Set . St. Louis: Mosby. p. 1840. ... Andrews' Diseases of the Skin: Clinical Dermatology . (10th ed.). Saunders. ISBN 0-7216-2921-0 . ^ a b c d Das, Anupam; Chandra, Somodyuti; Mohanty, Swosti; Gharami, RameshC; Podder, Indrashis (2015). ... Indian Journal of Paediatric Dermatology . 0 (0): 0. doi : 10.4103/2319-7250.165641 .
John Wiley & Sons. p. 11. ISBN 978-0-470-06638-6 . ^ Ann-Louise Shapiro (1996). ... Stanford University Press. p. 100. ISBN 0-8047-2693-0 . ^ Daniel Hack Tuke (1892). ... Stanford University Press. pp. 114 ff . ISBN 0-8047-3860-2 . ^ a b Femi Oyebode (2008). ... Cornell University Press. p. 48. ISBN 0-8014-4410-1 . ^ Anthony J. Close (1990). ... McGraw-Hill Professional. p. 226. ISBN 0-8442-8393-2 . ^ Susan Bordo (1996).
Pili pseudoannulati Other names Pseudo pili annulati [1] : 640 Specialty Dermatology Pili pseudoannulati is an anomaly of human hair that mimics pili annulati ; however, the two differ in that the light bands of pili annulati are caused by internal effects, whereas the bright segments of pili pseudoannulati are caused by reflection and refraction of light by flattened, twisted surfaces of hair. [2] : 767 References [ edit ] ^ Freedberg, et al. (2003). Fitzpatrick's Dermatology in General Medicine . (6th ed.). McGraw-Hill. ISBN 0-07-138076-0 . ^ James, William; Berger, Timothy; Elston, Dirk (2005). Andrews' Diseases of the Skin: Clinical Dermatology . (10th ed.). Saunders. ISBN 0-7216-2921-0 . This condition of the skin appendages article is a stub .
Polarizing light indicated variability in the thickness of the hair shaft, and illuminating light did not show bands, 2 additional features that distinguished it from pili annulati. Further examination of the hair shaft showed that the pseudopili annulati hairs had periodic widening and narrowing, that the fiber cross-section was roughly elliptical, and that a periodic twisting of the hair (30 to 40 degrees in 2 alternating directions) was superimposed on the ellipticity. ... INHERITANCE - Isolated cases SKIN, NAILS, & HAIR Hair - Scalp hair appears banded, with alternating light and dark segments (0.5 to 1.5 mm) - Hair may appear alternating shiny and dull - Hairs have elliptical diameter with flattened surface - Hair shaft shows partial twisting on its axis (periodicity of 1 to 2 mm) - Reflected light shows bright and dark bands - Transmitted light shows no banding or inhomogeneities MISCELLANEOUS - Onset in infancy or early childhood - No increased fragility of hair - Considered a normal variant - Distinct from pili annulati ( 180600 ) ▲ Close
Pigmented hairy epidermal nevus syndrome is a cutaneous condition characterized by a Becker nevus , ipsilateral hypoplasia of the breast , and skeletal defects such as scoliosis . [1] : 635 [2] : 776 See also [ edit ] Epidermal nevus syndrome Skin lesion List of cutaneous conditions References [ edit ] ^ James, William; Berger, Timothy; Elston, Dirk (2005). Andrews' Diseases of the Skin: Clinical Dermatology . (10th ed.). Saunders. ISBN 0-7216-2921-0 . ^ Freedberg, et al. (2003). Fitzpatrick's Dermatology in General Medicine . (6th ed.). McGraw-Hill. ISBN 0-07-138076-0 . This Epidermal nevi, neoplasms, cysts article is a stub .
Cutaneous diphtheria infection Specialty Infectious disease Cutaneous diphtheria is an infection of the skin by Corynebacterium diphtheriae . [1] : 265 It is also known as "desert sore". [2] See also [ edit ] Diphtheria Skin lesion References [ edit ] ^ James, William D.; Berger, Timothy G.; et al. (2006). Andrews' Diseases of the Skin: clinical Dermatology . Saunders Elsevier. ISBN 0-7216-2921-0 . ^ Geoffrey V. Gill; Nick Beeching (1 March 2004). ... Wiley-Blackwell. pp. 33–. ISBN 978-0-632-06496-0 . Retrieved 14 May 2010 .
Pterygium unguis (also known as "Dorsal pterygium" [1] : 660 ) forms as a result of scarring between the proximal nailfold and matrix, with the classic example being lichen planus , though it has been reported to occur as a result of sarcoidosis and Hansen's disease . [2] See also [ edit ] Pterygium inversum unguis Nail anatomy List of cutaneous conditions References [ edit ] ^ Freedberg, et al. (2003). Fitzpatrick's Dermatology in General Medicine . (6th ed.). McGraw-Hill. ISBN 0-07-138076-0 . ^ James, William; Berger, Timothy; Elston, Dirk (2005). Andrews' Diseases of the Skin: Clinical Dermatology . (10th ed.). Saunders. ISBN 0-7216-2921-0 . This condition of the skin appendages article is a stub .
Pterygium inversum unguis Other names Pterygium inversus unguis, [1] and Ventral pterygium [2] : 660 Specialty Dermatology Pterygium inversum unguis is characterized by the adherence of the distal portion of the nailbed to the ventral surface of the nail plate. [3] : 788 The condition may be present at birth or acquired, and may cause pain with manipulation of small objects, typing, and close manicuring of the nail. [3] : 788 secondary due to connective tissue disorders See also [ edit ] Pterygium unguis Nail Anatomy List of cutaneous conditions References [ edit ] ^ Rapini, Ronald P.; Bolognia, Jean L.; Jorizzo, Joseph L. (2007). Dermatology: 2-Volume Set . St. Louis: Mosby. ISBN 978-1-4160-2999-1 . ^ Freedberg, et al. (2003). Fitzpatrick's Dermatology in General Medicine . (6th ed.). McGraw-Hill. ISBN 0-07-138076-0 . ^ a b James, William; Berger, Timothy; Elston, Dirk (2005). Andrews' Diseases of the Skin: Clinical Dermatology . (10th ed.). Saunders. ISBN 0-7216-2921-0 . This condition of the skin appendages article is a stub .
Dermatitis repens Other names Acrodermatitis perstans Specialty Dermatology Dermatitis repens (also known as Acrodermatitis continua , [1] : 1026 Acrodermatitis perstans , Pustular acrodermatitis , Acrodermatitis continua of Hallopeau , Acrodermatitis continua suppurativa Hallopeau , Hallopeau's acrodermatitis , [1] Hallopeau's acrodermatitis continua , and Dermatitis repens Crocker ) is a rare, sterile, pustular eruption of the fingers and toes that slowly extends proximally. [1] : 1026 [2] : 631 [3] : 195 See also [ edit ] List of cutaneous conditions François Henri Hallopeau Psoriasis Skin lesion References [ edit ] ^ a b c Rapini, Ronald P.; Bolognia, Jean L.; Jorizzo, Joseph L. (2007). Dermatology: 2-Volume Set . St. Louis: Mosby. ISBN 978-1-4160-2999-1 . ^ Freedberg, et al. (2003). Fitzpatrick's Dermatology in General Medicine . (6th ed.). McGraw-Hill. ISBN 0-07-138076-0 . ^ James, William; Berger, Timothy; Elston, Dirk (2005). Andrews' Diseases of the Skin: Clinical Dermatology . (10th ed.). Saunders. ISBN 0-7216-2921-0 . This cutaneous condition article is a stub .
A rare, genetic, chronic, recurrent, slowly progressive, epidermal disease characterized by small, sterile, pustular eruptions, involving the nails and surrounding skin of the fingers and/or toes, which coalesce and burst, leaving erythematous, atrophic skin where new pustules develop. Onychodystrophy is frequently associated and anonychia and osteolysis are reported in severe cases. Local expansion (to involve the hands, forearms and/or feet) and involvement of mucosal surfaces (e.g. conjunctiva, tongue, urethra) may be observed.
Acrokeratoelastoidosis of Costa Other names Keratoelastoidosis marginalis [1] Autosomal dominant is the inheritance manner for this condition Specialty Dermatology Acrokeratoelastoidosis of Costa is a familial condition characterized by multiple keratotic papules on the dorsum of the hands and feet, palms, soles, in which electron microscopy shows rarified, abnormal elastic tissue. [2] : 993 [3] : 214 It was characterized in 1953. [4] Treatments such as liquid nitrogen , salicylic acid , tretinoin , and prednisone have been tried, though with limited success. [5] See also [ edit ] Skin lesion List of cutaneous conditions References [ edit ] ^ Rapini, Ronald P.; Bolognia, Jean L.; Jorizzo, Joseph L. (2007). Dermatology: 2-Volume Set . St. Louis: Mosby. ISBN 978-1-4160-2999-1 . ^ Freedberg, et al. (2003). Fitzpatrick's Dermatology in General Medicine . (6th ed.). McGraw-Hill. ISBN 0-07-138076-0 . ^ James, William; Berger, Timothy; Elston, Dirk (2005). Andrews' Diseases of the Skin: Clinical Dermatology . (10th ed.). Saunders. ISBN 0-7216-2921-0 . ^ Costa OG (1953). "Akrokerato-elastoidosis; a hitherto undescribed skin disease". ... "Acrokeratoelastoidosis" . Eur J Dermatol . 16 (2): 201–2. PMID 16613753 . External links [ edit ] Classification D OMIM : 101850 MeSH : C535653 DiseasesDB : 29804 External resources eMedicine : article/1113926 This cutaneous condition article is a stub .
A rare dermatosis characterized by small, firm papules or plaques (resembling warts) on the sides of the hands and feet. These stationary and asymptomatic lesions appear generally at puberty, or sometimes later Epidemiology The prevalence is unknown. Differential diagnosis The histology combines hyperkeratosis and acanthosis. Genetic counseling Both autosomal dominant and sporadic forms have been observed. Management and treatment Treatment is not indicated in most patients.
