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See also [ edit ] Acting out Free love Giacomo Casanova Hypersexuality Narcissistic personality disorder (Millon's variations) Repetition compulsion Sex addict References [ edit ] ^ a b Carol Groneman Nymphomania, a History (New York, 2000). ISBN 0-393-04838-1 ^ C. G. Jung, Psychological Aspects of the Mother Archetype , Collected Works of C. ... Jung , Vol. 9, Part 1. 2nd ed., Princeton University Press, 1968. 451 p. (pp. 75-80). ISBN 978-0-691-01833-1 ^ Jung, Carl. 1959. Symbols of Transformation. New York: Princeton University Press. p85. ^ Millon, Theodore, Personality Disorders in Modern Life, 2004 ^ Sigmund Freud, On Sexuality (PFL7) p. 234-6 ^ Otto Fenichel, The Psychoanalytic Theory of Neurosis (1946) p. 646 ^ Otto Fenichel, The Psychoanalytic Theory of Neurosis (1946) p. 243-4 ^ Otto Fenichel, The Psychoanalytic Theory of Neurosis (1946) p. 502-3 ^ S.
Oxford University Press, USA. pp. 245 ff. ISBN 978-0-19-533955-0 . ^ Alan Rosen (18 October 2010). The Wonder of Their Voices: The 1946 Holocaust Interviews of David Boder . ... Holocaust Memorial Museum. ISBN 978-0-89604-700-6 . ^ S. Kühl (7 August 2013). ... Indiana University Press. pp. 332–. ISBN 0-253-21529-3 . ^ David Nicholls (2000). ... Cambridge University Press. pp. 161 –. ISBN 978-0-521-39802-2 .
Clinical Features Helweg-Larsen and Ludvigsen (1946) reported a kindred of 14 in 5 generations with anhidrosis, 5 of whom had defective hearing with onset between 35 and 45 years of age. ... Inheritance There were several instances of father-son transmission in the family reported by Helweg-Larsen and Ludvigsen (1946), suggesting autosomal dominant inheritance.
Gates (1946) cited a family in which the grandfather showed bilaterally a radial artery that passed over the supinator longus muscle 3 to 4 cm above the wrist and ran over the radial extensors above the styloid process. ... This was clearly the family cited by Gates (1946). Inheritance - Autosomal dominant Vascular - Abnormal radial, ulnar, or interosseous artery ▲ Close
Contents 1 Psychoanalytic views 2 Anti-psychiatry 3 Delusions of reference 4 Examples 5 Literary analogues 6 See also 7 References Psychoanalytic views [ edit ] Main article: Psychoanalytic theory In Sigmund Freud 's view, "Delusions of being watched present this power in a regressive form, thus revealing its genesis...voices, as well as the undefined multitude, are brought into the foreground again by the [ paranoid ] disease, and so the evolution of conscience is reproduced regressively." [7] As early as 1928, Freud's contemporary, Carl Jung , introduced the concept of synchronicity , a theory of "meaningful coincidences". [8] In 1946, Otto Fenichel concluded that "the projection of the superego is most clearly seen in ideas of reference and of being influenced....Delusions of this kind merely bring to the patient from the outside what his self-observing and self-critical conscience actually tells him." [9] Jacques Lacan similarly saw ideas of reference as linked to "the unbalancing of the relation to the capital Other and the radical anomaly that it involves, qualified, improperly, but not without some approximation to the truth, in old clinical medicine, as partial delusion" [10] —the "big other, that is, the other of language, the Names-of-the-Father , signifiers or words", [11] in short, the realm of the superego. ... Springer Science & Business Media. ISBN 978-0-306-45746-3 . ^ Sigmund Freud, On Metapsychology (PFL 11) p. 90 ^ Tarnas, Richard (2006). ... New York: Penguin Group. p. 50 . ISBN 978-0-670-03292-1 . ^ Fenichel, Otto (1946).
The Great Famine: Northern Europe in the Early Fourteenth Century . Princeton University Press. ISBN 0-691-05891-1 . ^ "Murrain (WebBible Encyclopedia) - ChristianAnswers.Net" . christiananswers.net . ... Retrieved 28 July 2008 . ^ Mullett, Charles F. (1946). "The Cattle Distemper in Mid-Eighteenth-Century England" .
Canada: Pearson Canada Inc. pp. 310 . ISBN 978-0-205-64524-4 . ^ Cherney LR (2004). ... Nisbett (b. 1941) Martin Seligman (b. 1942) Ed Diener (b. 1946) Shelley E. Taylor (b. 1946) John Anderson (b. 1947) Ronald C.
New York, New York: Oxford University Press. pp. 34–36. ISBN 0-19-509514-6 . ^ Gage, Beverly (2009). ... Houghton Mifflin Company. ISBN 978-0-669-39728-4 . OCLC 48675667 . The American Pageant. ^ O. ... C. 411 of February 5, 1946 to investigate the facts relating to and the circumstances surrounding the communication, by public officials and other persons in positions of trust, of secret and confidential information to agents of a foreign power, June 27, 1946. ... Westport, Connecticut: Greenwood Press. pp. xv. ISBN 978-0-313-28506-6 . OCLC 27976811 . ^ Countryman, Edward (2010). ... Boston: Beacon Press. p. 130 . ISBN 978-0-8070-4441-4 . Retrieved May 3, 2011 . long step toward totalitarianism. ^ Liu, Qing (May 2020).
Rickettsialpox Rickettsialpox lesion Specialty Infectious disease Prognosis Resolves in 2-3 weeks without treatment Rickettsialpox is a mite-borne infectious illness caused by bacteria of the genus Rickettsia ( Rickettsia akari ). [1] Physician Robert Huebner and self-trained entomologist Charles Pomerantz played major roles in identifying the cause of the disease after an outbreak in 1946 in a New York City apartment complex, documented in "The Alerting of Mr. ... Physicians who had seen patients starting in early 1946 had assumed that they were dealing with an atypical form of chickenpox , but the realization was made that they were dealing with a localized epidemic of unknown origins starting in the summer of that year. ... Health Service Roots Out Cause of Spotted Ailment That Struck in Queens NO CURE IS FOUND AS YET Victim Made Ill by Bite of Insect--Weinstein Urges War on Rodents Some Removed to Hospitals Blood of Patients Sampled" , The New York Times , October 4, 1946. Accessed July 23, 2009. ^ Parola, Philippe. "Rickettsial Diseases" , p. 63. CRC Press , 2007. ISBN 0-8493-7611-4 . Accessed July 25, 2009. ^ "Smallpox, Rickettsialpox Scare "House" Doctors: Is Threat Real?"
A rare, acquired, self-limiting, infectious disease due to the mite-borne bacteria Rickettsia akari characterized by an asymptomatic, 0.5 to 2 cm in diameter papulovesicle that typically ulcerates and forms an eschar, followed by a generalized papulovesicular rash associating variable constitutional symptoms, such as localized lymphadenopathy, fever, malaise, and headaches. Additonal symptoms may include diaphoresis, myalgia and, less frequently, rhinorrhea, pharyngitis, nausea, vomiting, splenomegaly, conjunctival hyperemia, and abdominal pain. Systemic symtoms resolve within 6-10 days.
Pearson Education . p. 165. ISBN 978-0-13-170350-6 . ^ King, Moses (1919). ... Rowman & Littlefield Pub Incorporated, 1982: 103. ISBN 978-0-8476-6276-0 . ^ Kippen, Cameron (July 2004). ... China Illustrated: Western Views of the Middle Kingdom . the University of Michigan: Tuttle, 2004. p. 138. ISBN 978-0-8048-3519-0 . ^ Hacker, Authur (2012). ... Fenichel, The Psychoanalytic Theory of Neurosis (1946) p. 327 ^ O. Fenichel, The Psychoanalytic Theory of Neurosis (1946) p. 343 ^ Georges Bataille, Visions of Excess (1985) p. 23 ^ AJ Giannini et al., op. cit. ^ Shaw, WJ (1979). ... ISBN 0-415-35182-0 External links [ edit ] Look up podophilia in Wiktionary, the free dictionary.
This article may need to be rewritten to comply with Wikipedia's quality standards , as section. You can help . The talk page may contain suggestions. ( September 2016 ) Band keratopathy Band Keratopathy in a left eye. Specialty Ophthalmology Band keratopathy is a corneal disease derived from the appearance of calcium on the central cornea . This is an example of metastatic calcification, which by definition, occurs in the presence of hypercalcemia . [1] [2] Contents 1 Signs and symptoms 2 Causes 3 Pathology 4 Diagnosis 5 Treatment 6 References 7 External links Signs and symptoms [ edit ] Band keratopathy of a 60-year-old male Signs and symptoms of band keratopathy include eye pain and decreased visual acuity . Causes [ edit ] Band keratopathy has several causes. These causes include uveitis , interstitial keratitis , superficial keratitis , phthisis , sarcoidosis , trauma, intraocular silicone oil, systemic diseases ( high levels of calcium in the blood , vitamin D intoxication , Fanconi's Syndrome , low levels of phosphorus in the blood , gout , milk-alkali syndrome , myotonic dystrophy , and chronic mercury exposure). [ citation needed ] Pathology [ edit ] Band keratopathy is seen when there is calcification of the epithelial basement membrane , Bowman's membrane , and the anterior stroma with destruction of Bowman's membrane.
Consequently, neither maternal antibody titers nor amniotic bilirubin levels are good predictors of the severity of the disease (summary by Westhoff and Reid, 2004). The lack of Kell antigens, K(0), can be caused by several different gene defects. ... The Kell blood group system was discovered in 1946, just a few weeks after the introduction of the antiglobulin test.
Historically, this was a common phenomenon in Hiroshima and Nagasaki during World War II with the atomic bomb attacks from the United States . [1] The phenomenon was famously described by John Hersey in his 1946 article, and later book, Hiroshima .
They suggested that the families of Schoene et al. (1970), Ogryzlo (1946), and Parks and Staples (1945) had the same condition. ... Lafreniere et al. (2004) reported 5 families with HSAN2, including the large family from Newfoundland originally reported by Ogryzlo (1946). Beginning in early childhood, affected individuals experienced numbness in the hands and feet, aggravated by cold, together with reduced sensation to pain. ... Mapping By linkage analysis of 8 affected members from the consanguineous multigenerational Newfoundland pedigree reported by Ogryzlo (1946), and an additional family with 2 affected members, Lafreniere et al. (2004) mapped the HSAN2 disease locus to 12p13.33 (maximum lod score of 8.4). Molecular Genetics Among 5 families with HSAN2, including those from Newfoundland reported by Ogryzlo (1946) and patients from rural Quebec and Nova Scotia, Lafreniere et al. (2004) identified 3 different truncating mutations in the HSN2 isoform of the WNK1 gene (605232.0003-605232.0005) In 4 affected members of a large consanguineous Lebanese family with HSAN2, Riviere et al. (2004) identified a homozygous 1-bp deletion in the HSN2 isoform of the WNK1 gene (605232.0006).
A number sign (#) is used with this entry because hereditary sensory neuropathy type IIC (HSN2C) is caused by homozygous or compound heterozygous mutation in the KIF1A gene (601255) on chromosome 2q37. Mutation in the KIF1A gene can also cause hereditary spastic paraplegia-30 (SPG30; 610357). Description HSN2C is an autosomal recessive disorder characterized by onset in the first decade of progressive distal sensory loss leading to ulceration and amputation of the fingers and toes. Affected individuals also develop distal muscle weakness, primarily affecting the lower limbs (summary by Riviere et al., 2011). For a discussion of genetic heterogeneity of HSN, see HSAN1 (162400).
A number sign (#) is used with this entry because hereditary sensory and autonomic neuropathy type IIB (HSAN2B) is caused by homozygous mutation in the FAM134B gene (613114) on chromosome 5p15. Description Hereditary sensory and autonomic neuropathy type IIB is an autosomal recessive neurologic disorder characterized by early childhood onset of distal sensory impairment usually resulting in ulceration and associated with variable autonomic features, such as hyperhidrosis and urinary incontinence. Some patients may show impaired gait (summary by Ilgaz Aydinlar et al., 2014). HSAN2A (201300) is caused by mutation in the HSN2 isoform of the WNK1 gene (WNK1/HSN2; see 605232). For a discussion of genetic heterogeneity of HSAN, see HSAN1 (162400).
A rare hereditary sensory and autonomic neuropathy characterized by profound and universal sensory loss involving large and small fiber nerves. Epidemiology To date, less than 100 cases have been reported. There is no sex preference or particular ethnic preponderance. Clinical description Disease onset is typically in infancy and is non-progressive. Initial symptoms (from birth to 3 years) include lack of crying with trauma, self-mutilation (tongue, lips), swallowing and feeding problems. Gastroesophageal reflux is common. Sensory dysfunction is manifested by reduced or absent pain and temperature perception, and depressed or absent deep tendon reflexes.
A number sign (#) is used with this entry because autosomal recessive congenital indifference to pain and autosomal recessive hereditary sensory neuropathy type IID (HSN2D) are both caused by homozygous or compound heterozygous mutations in the SCN9A gene (603415) on chromosome 2q24. Description Congenital indifference to pain is a rare autosomal recessive disorder characterized by the complete absence of pain perception typically associated with noxious stimuli. Affected individuals are aware of a stimulus, but have lost the ability to perceive pain. Most patients are hyposmic or anosmic. Other sensory modalities are unaffected, and there is an absence of overt autonomic symptoms. Sural nerve biopsy and nerve conduction velocity studies are normal (summary by Cox et al., 2006; and Goldberg et al., 2012).
Toxicology . 183 (1–3): 39–56. doi : 10.1016/S0300-483X(02)00439-0 . PMID 12504341 . ^ Aronchick, J. ... PMID 20386622 . ^ Hardy, HL; Tabershaw, IR (1946). "Delayed chemical pneumonitis in workers exposed to beryllium compounds". ... Lamps and Lighting (3rd ed.). Edward Arnold. p. 127. ISBN 978-0-7131-3487-2 . ^ a b Lang, Leslie (June–July 1994). ... National Academies Press. p. 11. ISBN 978-0-309-11167-6 . ^ David Geraint James, Alimuddin Zumla, The granulomatous disorders , Cambridge University Press, 1999, ISBN 0-521-59221-6 , pages 336–337 ^ Brown University Medical School. ... PMID 23794247 . ^ "Photograph of Chicago Pile One Scientists 1946" . Office of Public Affairs, Argonne National Laboratory. 19 June 2006 .
