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Amyloid Cardiomyopathy Wikipedia

Retrieved 2017-12-01 . ^ Liao, Ronglih; Ward, Jennifer E. (2017-06-09). ... PMID 28596171 . ^ Falk, Rodney H.; Dubrey, Simon W. (2010-01-01). "Amyloid Heart Disease". Progress in Cardiovascular Diseases . ... PMID 20109604 . ^ a b Steingart, Richard M.; Chen, Carol; Liu, Jennifer (2016-01-01), Herrmann, Joerg (ed.), "Chapter 7 - Subtypes of Cancer Involving the Heart" , Clinical Cardio-Oncology , Elsevier, pp. 121–131, doi : 10.1016/b978-0-323-44227-5.00007-7 , ISBN 978-0-323-44227-5 , retrieved 2020-11-27 ^ a b c d e f g h i Gillespie, Hamilton S.; Benjamin, Ivor J. (2012-01-01), Hill, Joseph A.; Olson, Eric N. (eds.), "Chapter 43 - Infiltrative and Protein Misfolding Myocardial Diseases" , Muscle , Boston/Waltham: Academic Press, pp. 625–637, doi : 10.1016/b978-0-12-381510-1.00043-0 , ISBN 978-0-12-381510-1 , retrieved 2020-11-27 ^ a b c d e Benson, Merrill D. (2013-01-01), Rimoin, David; Pyeritz, Reed; Korf, Bruce (eds.), "Chapter 79 - Amyloidosis and Other Protein Deposition Diseases" , Emery and Rimoin's Principles and Practice of Medical Genetics , Oxford: Academic Press, pp. 1–18, doi : 10.1016/b978-0-12-383834-6.00083-5 , ISBN 978-0-12-383834-6 , retrieved 2020-11-27 ^ a b c Sher, T.; Gertz, M. A. (2018-01-01), Vasan, Ramachandran S.; Sawyer, Douglas B.

TTR, GSN, DPYD, NPPB, APOA1, PART1, SULT1E1, TNNT1, VEGFA, LSR, RBP4, DCLRE1B, MANEA, C16orf82, C4orf3, MIR155, MIR29A, SNCA, SERPINA1, PPP1R1A, APOE, NEFL, IAPP, HTT, FXN, GPC5, FBN1, FAP, CTSE, CRP, CLU, CACNA1A, MIR339
- Cardiac Amyloidosis Wikipedia
  
  Cardiac amyloidosis has multiple sub-types including light chain , familial , and senile . [3] One of the most studied types is light chain cardiac amyloidosis. [2] Prognosis depends on the extent of the deposits in the body and the type of amyloidosis. [4] New treatment methods are actively being researched in regards to the treatment of heart failure and specific cardiac amyloidosis problems. [5] [6] Contents 1 Types 1.1 Light chain (AL-CM) 1.2 Familial (ATTR m -CM) 1.3 Senile (ATTR wt -CM) 2 Cause 3 Symptoms 4 Diagnosis 4.1 Echocardiography 4.2 ECG/EKG 4.3 Laboratory Tests 4.4 Cardiac Biomarkers 4.5 Biopsies 4.6 Catherization 4.7 Cardiac Magnetic Resonance Imaging 4.8 Scintigraphy/Radionuclide Imaging 4.9 Mass Spectrometry 5 Prognosis 6 Treatments 7 References 8 External links Types [ edit ] The multiple subtypes of cardiac amyloidosis have varying epidemiological, diagnostic, and prognostic characteristics. [4] Light chain (AL-CM) [ edit ] This relatively rare form of cardiac amyloidosis occurs in an estimated 6-10 cases per 1,000,000 people. [4] This sub- type usually affects males over the age of 60 [4] and is rapidly progressive. Pathogenesis of this form is due to the aggregation of immunoglobulin lambda light chains . [3] These chains are created by an abnormal expansion of plasma cells . [3] Over time, these light chains deposit into the interstitial tissue within the myocardium. [4] Diagnostic tests includes serum and urine electrophoresis , [4] laboratory testing for the determination of elevated levels of troponin and BNP , and ECGs showing low QRS voltages. [2] Familial (ATTR m -CM) [ edit ] This type is caused by mutations of proteins involved in amyloid formation, including transthyretin (TTR), fibrinogen , apolipoprotein A1 , or apolipoprotein A2 . ... The vast majority of familial cardiac amyloidosis still present after the age of 60. [4] A common mutation is the TTR gene mutation Val122Ile. [2] It is estimated that 3.5 - 4% of African Americans in The United States have the Val 122lle mutation. [4] This type of amyloidosis can be identified by genetic testing for protein mutation. [4] For the diagnosis of familial cardiac amyloidosis to be made a biopsy with histological evaluation must be obtained. [7] In this histological evaluation special stains are utilized to visualize the amyloid deposits . [7] One such stain is Congo Red , which binds specifically to the amyloid deposit and can be characterized by various lighting methods. [7] Under polarized light, the amyloid deposits while show pathognomonic apple green birefringence, and under plain light the deposits will appear a light salmon pink color. [7] Familial amyloidosis symptoms are centered around neuropathological and cardiac problems. [3] Cardiac manifestations of the TTR mutation present more often in The United States. [4] Senile (ATTR wt -CM) [ edit ] This type is considered the wild-type mutation which leads to the development of TTR deposits . [2] It usually affects males over 70 years with the manifestation of carpal tunnel syndrome . [4] Similar to the other subtypes of cardiac amyloidosis, a biopsy is required for diagnosis. [4] However, formal diagnosis of Senile cardiac amyloidosis is a diagnosis of exclusion. [4] Biopsy with histological evaluation can rule out Light chain and Familial subtypes, leaving the diagnosis of Senile. [4] This type is often misdiagnosed, however, greater use of cardiac magnetic resonance has increased the rate of diagnosis [2] The severity of the disease tends to be less than the Light chain and Familial variants. [4] This is due to the amount of time that it takes to accumulate the amyloid depositions being longer in the Senile variant. [4] Cause [ edit ] The general cause of cardiac amyloidosis is the misfolding of a specific protein precursor depending on the amyloidosis type. ... Echocardiography [ edit ] Echocardiography is a safe and non-invasive method that can be used to assess the structural and functional disease of the heart. [4] Amyloidosis presents with ventricle and valvular thickening, biatrial enlargement, [4] restrictive filling pattern, with normal to mildly reduced systolic function [8] and decreased diastolic filling . [4] An echo can be used to evaluate for prognosis of the disease, measuring the different strains within the hear. [4] Cardiac amyloidosis produces specific alterations to the functionality of the heart. ... PMID 27634125 . ^ a b c d e f g h i j k l m n o p q r s t u v Bhogal S, Ladia V, Sitwala P, Cook E, Bajaj K, Ramu V, et al. (2018-01-01). "Cardiac Amyloidosis: An Updated Review With Emphasis on Diagnosis and Future Directions".
Dissecting Cellulitis Of The Scalp Wikipedia

Archived from the original on 2020-11-12 . Retrieved 2021-01-06 . ^ Dunwoodie, Hamish (2009-02-11). ... Archived from the original on 2018-12-13 . Retrieved 2021-01-06 . ^ Jones, Leah. "Perifolliculitis capitis abscedens et suffodiens | DermNet NZ" . dermnetnz.org . Archived from the original on 2020-10-31 . Retrieved 2021-01-06 . ^ Freedberg, et al. (2003). Fitzpatrick's Dermatology in General Medicine . (6th ed.). McGraw-Hill. ISBN 0-07-138076-0 . ^ James, William; Berger, Timothy; Elston, Dirk (2005). Andrews' Diseases of the Skin: Clinical Dermatology . (10th ed.). Saunders. ISBN 0-7216-2921-0 . ^ Scheinfeld NS (February 2003).

TNF
- Perifolliculitis Capitis Abscedens Et Suffodiens, Familial Omim
  
  Description Perifolliculitis capitis abscedens et suffodiens is a chronic inflammatory disease of the scalp characterized by the presence of large and small nodules that suppurate and intercommunicate by sinus formation. It may be more frequent in black males than in others (summary by McMullan and Zeligman, 1956). Clinical Features Bjellerup and Wallengren (1990) described typical perifolliculitis capitis abscedens et suffodiens in 2 brothers. One, a 20-year-old man, had a 3-month history of tender, suppurating nodules on the scalp and developed small areas of alopecia. The 33-year-old brother had itching of the scalp and hair loss. In both patients, the only microorganism isolated was S. epidermidis.
- Dissecting Cellulitis Of The Scalp Orphanet
  
  Dissecting cellulitis of the scalp is a rare chronic suppurative dermatosis of the scalp that mainly affects black men and that is characterized by multiple painful inflammatory follicular and perifollicular nodules, pustules, and abscesses that interconnect via sinus tracts and eventually result in scarring alopecia.
- Dissecting Cellulitis Of The Scalp Gard
  
  Dissecting cellulitis of the scalp is a rare and severe form of scalp folliculitis. It is sometimes associated with acne conglobata , hidradenitis suppurativa and spinal arthritis. Signs and symptoms of this condition include lardge nodules and cysts as well as smaller follicular papules. Hair loss occurs over the affected region and permanent scarring and bald patches appear. Treatment options for dissecting cellulitis of the scalp includes steroids and antibiotics but the condition does not typically improve even after treatment.
Tangential Speech Wikipedia

ISBN 9780838543337 . Retrieved 2012-01-17 . ^ Crash Course: Psychiatry by Julius Bourke, Matthew Castle, Alasdair D. Cameron 2008 ISBN 0-7234-3476-X page 255 ^ Introduction to Neurogenic Communication Disorders by Robert H. ... ISBN 9780521810753 . Retrieved 2012-01-12 . ISBN 0-521-81075-2 ^ Tali Ditman & Gina R. ... Martinos Center for Biomedical Imaging . Retrieved 2012-01-17 . ^ Howard H. Goldman 2000 - Review of general psychiatry - 583 pages A Lange medical book McGraw-Hill Professional , Retrieved 2012-01-17 ISBN 0-8385-8434-9 ^ Jeffrey A. ... Perkins, M.D. 2011 Essentials of Schizophrenia - 268 pages American Psychiatric Pub, 2 Jun 2011 Retrieved 2012-01-12 ISBN 1-58562-401-2 ^ S.J.Wood, N.B.Allen & C.Pantelis (Editors) 2009 - The Neuropsychology of Mental Illness Cambridge University Press Archived 2016-03-04 at the Wayback Machine Retrieved 2012-01-17 ISBN 978-0-521-86289-9 ^ Branca Telles Ribeiro 23 Jun 1994 - Coherence in psychotic discourse - 320 pages Oxford studies in sociolinguistics Oxford University Press , Retrieved 2012-01-17 ISBN 0-19-506615-4 ^ Stephens, J.
Lichen Spinulosus Wikipedia

Contents 1 See also 2 Notes 3 References 4 External links See also [ edit ] Hook nail List of cutaneous conditions Notes [ edit ] ^ James, William; Berger, Timothy; Elston, Dirk (2005). Andrews' Diseases of the Skin: Clinical Dermatology . (10th ed.). Saunders. ISBN 0-7216-2921-0 . ^ Rapini, Ronald P.; Bolognia, Jean L.; Jorizzo, Joseph L. (2007). ... St. Louis: Mosby. ISBN 1-4160-2999-0 . References [ edit ] Friedman S (1990). ... J Am Acad Dermatol . 22 (2 Pt 1): 261–4. doi : 10.1016/0190-9622(90)70035-G . PMID 2179296 . External links [ edit ] Derm Net NZ Emedicine Thehinhso v t e Diseases of the skin and appendages by morphology Growths Epidermal Wart Callus Seborrheic keratosis Acrochordon Molluscum contagiosum Actinic keratosis Squamous-cell carcinoma Basal-cell carcinoma Merkel-cell carcinoma Nevus sebaceous Trichoepithelioma Pigmented Freckles Lentigo Melasma Nevus Melanoma Dermal and subcutaneous Epidermal inclusion cyst Hemangioma Dermatofibroma (benign fibrous histiocytoma) Keloid Lipoma Neurofibroma Xanthoma Kaposi's sarcoma Infantile digital fibromatosis Granular cell tumor Leiomyoma Lymphangioma circumscriptum Myxoid cyst Rashes With epidermal involvement Eczematous Contact dermatitis Atopic dermatitis Seborrheic dermatitis Stasis dermatitis Lichen simplex chronicus Darier's disease Glucagonoma syndrome Langerhans cell histiocytosis Lichen sclerosus Pemphigus foliaceus Wiskott–Aldrich syndrome Zinc deficiency Scaling Psoriasis Tinea ( Corporis Cruris Pedis Manuum Faciei ) Pityriasis rosea Secondary syphilis Mycosis fungoides Systemic lupus erythematosus Pityriasis rubra pilaris Parapsoriasis Ichthyosis Blistering Herpes simplex Herpes zoster Varicella Bullous impetigo Acute contact dermatitis Pemphigus vulgaris Bullous pemphigoid Dermatitis herpetiformis Porphyria cutanea tarda Epidermolysis bullosa simplex Papular Scabies Insect bite reactions Lichen planus Miliaria Keratosis pilaris Lichen spinulosus Transient acantholytic dermatosis Lichen nitidus Pityriasis lichenoides et varioliformis acuta Pustular Acne vulgaris Acne rosacea Folliculitis Impetigo Candidiasis Gonococcemia Dermatophyte Coccidioidomycosis Subcorneal pustular dermatosis Hypopigmented Tinea versicolor Vitiligo Pityriasis alba Postinflammatory hyperpigmentation Tuberous sclerosis Idiopathic guttate hypomelanosis Leprosy Hypopigmented mycosis fungoides Without epidermal involvement Red Blanchable Erythema Generalized Drug eruptions Viral exanthems Toxic erythema Systemic lupus erythematosus Localized Cellulitis Abscess Boil Erythema nodosum Carcinoid syndrome Fixed drug eruption Specialized Urticaria Erythema ( Multiforme Migrans Gyratum repens Annulare centrifugum Ab igne ) Nonblanchable Purpura Macular Thrombocytopenic purpura Actinic/solar purpura Papular Disseminated intravascular coagulation Vasculitis Indurated Scleroderma / morphea Granuloma annulare Lichen sclerosis et atrophicus Necrobiosis lipoidica Miscellaneous disorders Ulcers Hair Telogen effluvium Androgenic alopecia Alopecia areata Systemic lupus erythematosus Tinea capitis Loose anagen syndrome Lichen planopilaris Folliculitis decalvans Acne keloidalis nuchae Nail Onychomycosis Psoriasis Paronychia Ingrown nail Mucous membrane Aphthous stomatitis Oral candidiasis Lichen planus Leukoplakia Pemphigus vulgaris Mucous membrane pemphigoid Cicatricial pemphigoid Herpesvirus Coxsackievirus Syphilis Systemic histoplasmosis Squamous-cell carcinoma This condition of the skin appendages article is a stub .
Acephalgic Migraine Wikipedia

Lippincott Williams & Wilkins. p. 1152. ISBN 0-7817-7513-2 . ^ Pryse-Phillips, William (2003). ... Oxford University Press US. p. 587. ISBN 0-19-515938-1 . ^ Bradley, Walter George (2004). ... Lippincott Williams & Wilkins. p. 2695. ISBN 0-7817-5777-0 . ^ Miller, Neil R.; Frank Burton Walsh; Valérie Biousse; William Fletcher Hoyt (2005). ... Lippincott Williams & Wilkins. p. 1289. ISBN 0-7817-4811-9 . ^ a b Loder, Elizabeth; Dawn A. ... McGraw-Hill Professional. p. 127. ISBN 0-07-105467-7 . ^ G. D. Schott (2007).
Histiocytosis Wikipedia

Contents 1 Types 2 Diagnosis 2.1 Classification 3 Treatments 4 Society 5 References 6 External links Types [ edit ] Types of LCH have also been known as "eosinophilic granuloma", "Hand-Schuller-Christian disease", "Letterer-Siwe disease", and "histiocytosis X". ... Information concerning histiocytosis and clinicians located in European countries may be found in many languages at the web portal of Euro Histio Net (EHN). This is a project funded by the European Union, coordinated by Jean Donadieu, APHP , Paris, France. ... Andrews' Diseases of the Skin: clinical Dermatology . Saunders Elsevier. ISBN 0-7216-2921-0 . ^ Goldberg, J; Nezelof, C (1986), "Lymphohistiocytosis: a multi-factorial syndrome of macrophagic activation clinico-pathological study of 38 cases", Hematol Oncol , 4 (4): 275–289, PMID 3557322 . ^ Egan, Caoimhe; Jaffe, Elaine S. (2018). ... "What is the Histiocyte Society classification of histiocytosis syndromes?" . Medscape . Retrieved 4 December 2020 . ^ Chang, Karen L.; Snyder, David S. (2007). "17. ... Springer Science & Business Media. p. 383. ISBN 978-0-387-73743-0 . External links [ edit ] Classification D ICD - 10 : C96.1 , D76.0 ICD - 9-CM : 202.3 , 277.89 MeSH : D015614 SNOMED CT : 60657004 External resources MedlinePlus : 000068 eMedicine : ped/1997 v t e Histiocytosis WHO-I/ Langerhans cell histiocytosis / X-type histiocytosis Letterer–Siwe disease Hand–Schüller–Christian disease Eosinophilic granuloma Congenital self-healing reticulohistiocytosis WHO-II/ non-Langerhans cell histiocytosis / Non-X histiocytosis Juvenile xanthogranuloma Hemophagocytic lymphohistiocytosis Erdheim-Chester disease Niemann–Pick disease Sea-blue histiocyte Benign cephalic histiocytosis Generalized eruptive histiocytoma Xanthoma disseminatum Progressive nodular histiocytosis Papular xanthoma Hereditary progressive mucinous histiocytosis Reticulohistiocytosis ( Multicentric reticulohistiocytosis , Reticulohistiocytoma ) Indeterminate cell histiocytosis WHO-III/ malignant histiocytosis Histiocytic sarcoma Langerhans cell sarcoma Interdigitating dendritic cell sarcoma Follicular dendritic cell sarcoma Ungrouped Rosai–Dorfman disease

SLC29A3, IFNGR1, BRAF, ALK, MAP2K7, PTPRC, EPHB2, MAPK1, MAP2K1, MAP2K2, S100A1, ARAF, TLR7, CD274, ZHX2, PPT2, CD163, TP53, TIMP1, S100B, CD68, RET, RAF1, CSF1R, PTGS1, XIAP, POU6F1, KIF5B, IL6, CD1A, HPRT1, SEMA6A
Persistent Left Superior Vena Cava Wikipedia

CT and MRI scans in a parasagittal section may show a "pipe" sign where the left superior vena cava occurs. [4] Treatment [ edit ] If no other cardiac abnormalities are present, PLSVC will not be treated, as it is usually asymptomatic and unharmful. [4] If it drains into the left atrium , then deoxygenated blood enters the circulation to the body, and cyanosis may occur. [8] References [ edit ] ^ Pahwa R, Kumar A (May 2003). ... Med. J . 96 (5): 528–9. doi : 10.1097/01.smj.0000060885.27846.91 . PMID 12911199 . ^ Gonzalez-Juanatey C, Testa A, Vidan J, et al. ... New York: Macmillan Publishing. ^ a b c d e Crispi, Fatima; Martinez, Josep M. (2018-01-01), Copel, Joshua A.; D'Alton, Mary E.; Feltovich, Helen; Gratacós, Eduard (eds.), "92 - Anomalies of Systemic Venous Return" , Obstetric Imaging: Fetal Diagnosis and Care (Second Edition) , Elsevier, pp. 411–414.e1, doi : 10.1016/b978-0-323-44548-1.00092-9 , ISBN 978-0-323-44548-1 , retrieved 2020-11-16 ^ a b c Fligner, Corinne L.; Clark, John I.; Clark, Judy M.; Larson, Lyle W.; Poole, Jeanne E. (2018-01-01), Poole, Jeanne E.; Larson, Lyle W. (eds.), "2 - Surgical Anatomy for the Implanting Physician" , Surgical Implantation of Cardiac Rhythm Devices , Elsevier, pp. 13–58, doi : 10.1016/b978-0-323-40126-5.00002-1 , ISBN 978-0-323-40126-5 , retrieved 2020-11-16 ^ Berg C, Knüppel M, Geipel A, et al. ... PMID 7368902 . ^ a b Marelli, Ariane J. (2012-01-01), Goldman, Lee; Schafer, Andrew I.

RBM10, HTC2
Dependent Personality Disorder Wikipedia

Springer, Boston, MA. pp. 230–237. doi : 10.1007/978-0-387-28370-8_22 . ISBN 978-0-387-28369-2 . ^ a b Coolidge, F.L.; Thede, L.; Jang, K.L. (2 December 2013). ... Springer, Boston, MA. pp. 230–237. doi : 10.1007/978-0-387-28370-8_22 . ISBN 978-0-387-28369-2 . ^ Nolen-Hoeksema, Susan (2014). ... American Psychiatric Press. pp. 995–8. ISBN 978-0-88048-859-4 . Gjerde, L. C.; Czajkowski, N.; Røysamb, E.; Ørstavik, R. ... Diagnosis and Treatment of the Personality Disorders . Ishiyaku EuroAmerica. ISBN 978-0-912791-89-0 . Ellison, J. M.; Adler, D. ... Treating Co-Occurring Disorders . pp. 63–4. ISBN 978-1-317-82549-4 . Oldham, John M.; Morris, Lois B. (1990).

DRD4
Affluenza Wikipedia

Affluenza: How to Be Successful and Stay Sane . Vermilion . ISBN 978-0-09-190011-3 . ^ James, Oliver (2008). The Selfish Capitalist . Vermilion . ISBN 978-0-09-192381-5 . ^ James, Oliver (2007). ... London: Vermilion. p. 344 . ISBN 978-0-09-190010-6 . 1. The mean prevalence of emotional distress for the six English-speaking nations combined was 21.6%. ... (Archive is the same work, but on a different website) Further reading [ edit ] The Circle of Simplicity , Cecile Andrews, ISBN 0-06-092872-7 The Golden Ghetto: The Psychology of Affluence , Jessie H. O'Neill, ISBN 978-0-9678554-0-0 Voluntary Simplicity , Duane Elgin, ISBN 0-688-12119-5 Voluntary Simplicity , Daniel Doherty & Amitai Etzioni, ISBN 0-7425-2066-8 How Much Is Too Much?
Berlin's Edema Wikipedia

Philadelphia: Lippincott Williams & Wilkins. p. 274. ISBN 978-0-7817-4596-3 . ^ Park, S. J.; Park, K. ... PMID 24384962 . ^ Recchia, Franco M.; Aaberg Jr., Thomas; Sternberg Jr., Paul (2006-01-01). Hinton, David R.; Schachat, Andrew P.; Wilkinson, C. ... Edinburgh: Mosby. pp. 2379–2401. doi : 10.1016/b978-0-323-02598-0.50146-4 . ISBN 9780323025980 . ^ Ah-kee, Elliott; Oladiwura, Dilys; Lim, Lik Thai; Scott, Angus (2014-05-19). ... PMID 24899795 . ^ Chen, Jeannie; Geller, Scott; Flannery, John (2006-01-01). Hinton, David R.; Schachat, Andrew P.; Wilkinson, C. ... Edinburgh: Mosby. pp. 153–170. doi : 10.1016/b978-0-323-02598-0.50014-8 . ISBN 9780323025980 . ^ Ghazanfari-Nasrabad, Mahdi; Amrollahi-Sharifabadi, Mohammad; Kargar-Bideh, Omidreza; Azizi-Sharifabad, Saeed (2016-01-01).
African Trypanosomiasis Wikipedia

The World Health Organization plans to eradicate sleeping sickness by the year 2030. [37] [38] Trypanosoma brucei gambiense [39] 1990 1991 1992 1993 1994 1995 1996 1997 1998 1999 2000 2001 2002 2003 2004 2005 2006 2007 2008 2009 2010 2011 2012 2013 2014 2015 2016 2017 2018 2019 Angola 1498 2094 2406 1796 1274 2441 6726 8275 6610 5351 4546 4577 3621 3115 2280 1727 1105 648 517 247 211 154 70 69 36 35 19 18 79 30 Benin 0 0 2 1 0 0 0 0 0 20 0 0 0 0 0 0 0 0 0 0 0 0 0 0 0 0 0 0 0 0 Burkina Faso 27 27 20 17 18 13 12 1 15 15 0 0 0 0 0 0 0 0 0 0 0 0 0 0 0 1 0 0 0 0 Cameroon 86 69 21 3 20 21 17 10 54 32 27 14 32 33 17 3 15 7 13 24 16 15 7 6 7 6 6 5 7 17 Central African Republic 308 197 362 262 368 676 492 730 1068 869 988 718 572 539 738 666 460 654 1194 1054 395 132 381 59 194 147 124 76 57 86 Chad 20 221 149 65 214 315 178 122 134 187 153 138 715 222 483 190 276 97 196 510 232 276 197 195 95 67 53 28 12 16 Congo 580 703 727 829 418 475 474 142 201 91 111 894 1005 717 873 398 300 189 182 87 87 61 39 20 21 36 18 15 24 17 Côte d'Ivoire 365 349 456 260 206 326 240 185 121 104 188 92 97 68 74 42 29 13 14 8 8 10 9 7 6 3 0 3 2 1 Democratic Republic of the Congo 7515 5825 7757 11384 19021 18182 19342 25094 26318 18684 16951 17300 13816 11459 10339 10249 8013 8155 7318 7178 5624 5590 5968 5647 3205 2351 1769 1110 660 604 Equatorial Guinea 63 36 45 30 85 37 46 67 62 28 16 17 32 23 22 17 13 15 11 7 8 1 2 3 0 0 3 4 4 3 Gabon 80 45 33 80 61 20 32 11 6 38 45 30 26 26 49 53 31 30 24 14 22 17 9 17 10 9 10 9 16 8 Ghana 3 6 16 0 0 0 1 0 0 0 1 0 0 0 0 0 0 0 0 0 0 0 0 1 0 0 0 0 0 0 Guinea 52 29 24 27 26 33 38 88 99 68 52 72 132 130 95 94 48 69 90 79 68 57 70 78 33 29 107 140 74 69 Mali 0 0 0 27 17 11 0 0 0 0 0 0 0 0 0 0 0 0 0 0 0 0 0 0 0 0 0 0 0 0 Nigeria 24 0 0 0 0 0 0 0 0 27 14 14 26 31 10 21 3 0 0 0 2 3 2 0 0 0 1 0 0 0 South Sudan 67 58 28 62 69 56 157 737 1726 1312 1801 1919 3121 3061 1742 1853 789 469 623 373 199 272 317 117 63 45 17 12 17 11 Togo 2 0 0 0 0 3 0 0 0 0 0 0 0 0 0 0 0 0 0 0 0 0 0 0 0 0 0 0 0 0 Uganda 2066 1328 2042 1764 1469 1062 981 1123 971 1036 948 310 604 517 378 311 290 120 198 99 101 44 20 9 9 4 4 0 1 2 Total 12756 10987 14088 16607 23266 23671 28736 36585 37385 27862 25841 26095 23799 19941 17100 15624 11372 10466 10380 9680 6973 6632 7091 6228 3679 2733 2131 1420 953 864 Trypanosoma brucei rhodesiense [40] 1990 1991 1992 1993 1994 1995 1996 1997 1998 1999 2000 2001 2002 2003 2004 2005 2006 2007 2008 2009 2010 2011 2012 2013 2014 2015 2016 2017 2018 2019 Kenya 91 8 4 2 1 0 2 5 14 22 15 10 11 0 0 0 1 0 0 1 0 0 2 0 0 0 0 0 0 0 Malawi 228 195 143 53 31 15 8 7 10 11 35 38 43 70 48 41 58 50 49 39 29 23 18 35 32 30 37 7 15 91 Mozambique 3 7 24 10 16 No data No data No data No data No data No data No data 1 No data 1 No data No data No data No data No data No data No data No data No data No data No data No data No data No data No data Uganda 1417 832 606 503 342 497 178 217 283 283 300 426 329 338 335 473 261 119 138 129 112 84 71 43 70 28 10 13 4 5 United Republic of Tanzania 187 177 366 262 319 422 400 354 299 288 350 277 228 113 159 186 127 126 59 14 5 1 4 1 1 2 3 3 0 3 Zambia 7 No data 4 1 1 1 3 No data No data 15 9 4 5 15 9 7 6 10 13 4 8 3 6 6 12 8 2 3 5 15 Zimbabwe No data No data No data No data 1 No data No data 9 No data No data No data No data No data No data No data 3 No data No data 0 3 2 4 9 1 3 3 1 1 0 2 Total 1933 1219 1147 831 710 935 591 583 606 619 709 755 617 536 552 707 453 305 259 187 154 111 101 85 115 68 52 27 24 116 History [ edit ] In 1903, David Bruce recognized the tsetse fly as the arthropod vector. ... Lancet . 380 (9859): 2095–128. doi : 10.1016/S0140-6736(12)61728-0 . hdl : 10536/DRO/DU:30050819 . PMID 23245604 . ... "Why trypanosomes cause sleeping sickness". Physiology . 19 (4): 198–206. doi : 10.1152/physiol.00006.2004 . ... The Onderstepoort Journal of Veterinary Research . 71 (4): 307–12. doi : 10.4102/ojvr.v71i4.250 . ... Emerging Infectious Diseases . 10 (4): 567–73. doi : 10.3201/eid1004.020626 .

NOS2, APOL1, TMPRSS11D, APBB3, SRA1, MSR1, IL10, LSAMP, LAMP3, HPR, NXT1, SERPINA1, IL6, ODC1, RMDN1, SMUG1, VSX2, MRPL28, STT3B, SUB1, MCF2L, SEC14L2, DNAJB1P1, TIMM10, CMTR2, RMDN3, SYBU, PAQR7, RMDN2, TUT1, TIMM9, NXF1, CDKN2B, GMPS, SLC9A6, DNAJB1, SARDH, DNAH8, FLNB, GAPDH, UTS2R, HLA-G, HP, PRMT1, IL1A, PEX16, IL1RN, CXCL10, STT3A, SRL, TNF, USO1, DDOST, NR1I2, H3P9
- African Trypanosomiasis Orphanet
  
  Human African Trypanosomiasis (HAT), also called sleeping sickness, is a vector-borne parasitic disease caused by a protozoa of the Trypanosoma genus transmitted by the bite of a tsetse fly (genus Glossina ), that is found under its chronic form (average duration of 3 years) in western and central Africa (in case of the T. brucei gambiense sub-species), and under its acute form (lasting from few weeks to 6 months) in eastern and southern Africa (in case of the T. brucei rhodesiense sub-species). HAT comprises an initial hemo-lymphatic stage characterized by fever, weakness, musculoskeletal pain, anemia, and lymphadenopathy, along with dermatologic, cardiac and endocrine complications or hepatosplenomegaly, followed by a meningo-encephalitic stage characterized by neurologic involvement (sleep disturbances, psychiatric disorders, seizures) that progresses, in the absence of treatment, towards a fatal meningoencephalitis.
Pachyonychia Congenita Gene_reviews

Note: (1) The genes included in the panel and the diagnostic sensitivity of the testing used for each gene varies by laboratory and are likely to change over time. (2) Some multigene panels may include genes not associated with the condition discussed in this GeneReview ; thus, clinicians need to determine which multigene panel is most likely to identify the genetic cause of the condition at the most reasonable cost while limiting identification of variants of uncertain significance and pathogenic variants in genes that do not explain the underlying phenotype. (3) In some laboratories, panel options may include a custom laboratory-designed panel and/or custom phenotype-focused exome analysis that includes genes specified by the clinician. (4) Methods used in a panel may include sequence analysis, deletion/duplication analysis, and/or other non-sequencing-based tests. ... Molecular Genetic Testing Used in Pachyonychia Congenita (PC) View in own window Gene 1 Proportion of PC Attributed to Pathogenic Variants in Gene 2, 3 Proportion of Pathogenic Variants 4 Detectable by Method Sequence analysis 5 Gene-targeted deletion/duplication analysis 6 KRT6A 304/774 (39%) >99% Unknown 7 KRT6B 70/774 (9%) >99% Unknown 7 KRT6C 22/774 (3%) >99% Unknown 7 KRT16 247/774 (32%) >99% Unknown 7 KRT17 8 130/774 (17%) >99% Unknown 7 1. ... The numbers in this table refer only to those individuals enrolled in the International PC Research Registry (IPCRR); not all are published but the data are available on the website. 4. See Molecular Genetics for information on allelic variants detected in this gene. 5. ... International Pachyonychia Congenita Research Registry (IPCRR) Data Summary (as of 10 May 2017) View in own window Gene in Which Pathogenic Variants Were Confirmed KRT6A KRT6B KRT6C KRT16 KRT17 TOTAL Number evaluated for finding 1 304 71 22 247 130 774 Toenails thickened 10 toenails 286 (94%) 26 (37%) 0 (00%) 99 (40%) 100 (77%) 511 (66%) 7-9 toenails 9 (03%) 14 (20%) 0 (00%) 52 (21%) 11 (08%) 86 (11%) 4-6 toenails 2 (01%) 21 (30%) 3 (14%) 63 (26%) 7 (05%) 96 (12%) 1-3 toenails 4 (01%) 9 (13%) 10 (45%) 44 (18%) 8 (06%) 75 (10%) Total w/toenails thickened 301 (99%) 70 (99%) 13 (59%) 238 (96%) 126 (97%) 748 (97%) Age at onset Birth - <1 yr 264 (88%) 10 (14%) 1 (08%) 45 (19%) 92 (73%) 412 (54%) 1-4 yrs 33 (11%) 19 (27%) 6 (46%) 78 (33%) 24 (19%) 160 (21%) 5-14 yrs 4 (01%) 34 (49%) 4 (31%) 74 (31%) 10 (08%) 126 (17%) ≥15 yrs 0 (00%) 7 (10%) 2 (15%) 43 (18%) 1 (01%) 53 (07%) Fingernails thickened 10 fingernails 271 (89%) 4 (06%) 0 (00%) 73 (30%) 62 (48%) 410 (53%) 7-9 fingernails 10 (03%) 4 (06%) 0 (00%) 11 (04%) 13 (10%) 38 (05%) 4-6 fingernails 14 (05%) 17 (24%) 0 (00%) 30 (12%) 28 (22%) 89 (11%) 1-3 fingernails 6 (02%) 7 (10%) 0 (00%) 28 (11%) 9 (07%) 50 (06%) Total w/fingernails thickened 301 (99%) 32 (45%) 0 (00%) 142 (57%) 112 (86%) 587 (76%) Age at onset Birth - <1 yr 267 (89%) 4 (13%) 0 (00%) 33 (23%) 85 (76%) 389 (66%) 1-4 yrs 30 (10%) 8 (25%) 0 (00%) 42 (30%) 19 (17%) 99 (17%) 5-14 yrs 3 (01%) 11 (34%) 0 (00%) 34 (24%) 6 (05%) 54 (09%) ≥15 yrs 1 (00%) 10 (31%) 0 (00%) 34 (24%) 3 (03%) 48 (08%) Plantar keratoderma Always (never completely goes away) 254 (84%) 67 (94%) 19 (86%) 240 (97%) 86 (66%) 666 (86%) Sometimes (feet clear up completely at times) 7 (02%) 1 (01%) 0 (00%) 1 (00%) 14 (11%) 23 (03%) Seldom (feet usually clear of symptoms) 5 (02%) 0 (00%) 0 (00%) 0 (00%) 4 (03%) 9 (01%) Total w/plantar keratoderma 266 (88%) 68 (96%) 19 (86%) 241 (98%) 104 (80%) 698 (90%) Age at onset Birth - <1 yr 39 (15%) 2 (03%) 1 (05%) 23 (10%) 12 (12%) 77 (11%) 1-4 yrs 152 (57%) 23 (34%) 9 (47%) 130 (54%) 35 (34%) 349 (50%) 5-14 yrs 70 (26%) 42 (62%) 9 (47%) 82 (34%) 43 (41%) 246 (35%) ≥15 yrs 5 (02%) 1 (01%) 0 (00%) 8 (03%) 15 (14%) 29 (04%) Plantar pain w/plantar keratoderma 2 Often require medication for pain 65 (24%) 12 (18%) 5 (26%) 74 (31%) 19 (18%) 175 (25%) Very painful, but do not use medication 114 (43%) 32 (47%) 11 (58%) 111 (46%) 34 (33%) 302 (43%) Somewhat painful 77 (29%) 24 (35%) 3 (16%) 50 (21%) 36 (35%) 190 (27%) Total w/plantar keratoderma/pain 256 (96%) 68 (100%) 19 (100%) 235 (98%) 89 (86%) 667 (96%) Palmar keratoderma Always (never completely goes away) 86 (28%) 11 (15%) 2 (09%) 148 (60%) 21 (16%) 268 (35%) Sometimes (hands clear up completely at times) 32 (11%) 7 (10%) 1 (05%) 15 (06%) 22 (17%) 77 (10%) Seldom (hands usually clear of symptoms) 49 (16%) 13 (18%) 3 (14%) 22 (09%) 27 (21%) 114 (15%) Total w/palmar keratoderma 167 (55%) 31 (44%) 6 (27%) 185 (75%) 70 (54%) 459 (59%) Additional findings Oral leukokeratosis 269 (88%) 18 (25%) 4 (18%) 88 (36%) 34 (26%) 413 (53%) Cysts 188 (62%) 49 (69%) 4 (18%) 64 (26%) 121 (93%) 426 (55%) Follicular hyperkeratosis 162 (53%) 30 (42%) 0 (00%) 30 (12%) 86 (66%) 308 (40%) Natal or prenatal teeth 12 (04%) 0 (00%) 0 (00%) 0 (00%) 99 (76%) 111 (14%) 1.

KRT17, KRT16, KRT6A, KRT6B, KRT10, KRT6C, KRT80, SLURP1, PNOC, NFE2L2, AQP5, FLG, KRT1, KLKB1, GJB2, GABPA, MTOR, KRT9
- Pachyonychia Congenita 4 Omim
  
  A number sign (#) is used with this entry because pachyonychia congenita-4 (PC4) is caused by heterozygous mutation in the KRT6B gene (148042) on chromosome 12q13.
- Pachyonychia Congenita Orphanet
  
  Etiology Although historically two subtypes have been described, PC-1 and PC2, it is today recommended to classify PC patients into four subgroups based the underlying molecular etiology: PC-K6a, PC-K6b, PC-K16 and PC-K17, as PC is caused by dominant negative mutations in at least 4 genes [ KRT6A , KRT6B (12q13.13), KRT16 , KRT17 (17q21.2)] encoding keratins preferentially expressed in basal and suprabasal layers of palmoplantar skin, epidermal appendages and oral mucosa.
- Pachyonychia Congenita Gard
  
  Pachyonychia congenita (PC) is a rare inherited condition that primarily affects the nails and skin. The fingernails and toenails may be thickened and abnormally shaped . Affected people can also develop painful calluses and blisters on the soles of their feet and less frequently on the palms of their hands ( palmoplantar keratoderma ). Additional features include white patches on the tongue and inside of the mouth (leukokeratosis); bumps around the elbows, knees, and waistline (follicular hyperkeratosis); and cysts of various types including steatocystoma. Features may vary among affected people depending on their specific mutation.
- Pachyonychia Congenita, Autosomal Recessive Omim
  
  Inheritance Chong-Hai and Rajagopalan (1977) suggested autosomal recessive inheritance of pachyonychia congenita in a 4-year-old Malaysian girl with first-cousin parents, although they recognized new dominant mutation as a possibility.
- Pachyonychia Congenita 3 Omim
  
  A number sign (#) is used with this entry because pachyonychia congenita-3 (PC3) is caused by heterozygous mutation in the keratin-6a gene (KRT6A; 148041) on chromosome 12q13. Description Pachyonychia congenita (PC) is an autosomal dominant genodermatosis with the main clinical features of hypertrophic nail dystrophy, painful and highly debilitating plantar keratoderma, oral leukokeratosis, and a variety of epidermal cysts. Although the condition had previously been subdivided clinically into Jadassohn-Lewandowsky PC type 1 and Jackson-Lawler PC type 2, patients with PC were later found to have a mixed constellation of both types, leading to a classification of PC based on genotype (summary by Sybert, 2010; Eliason et al., 2012; McLean et al., 2011). For a discussion of genetic heterogeneity of pachyonychia congenita, see 167200. Historical Classification of Pachyonychia Congenita Gorlin et al. (1976) suggested that 2 distinct syndromes are subsumed under the designation pachyonychia congenita.
- Pachyonychia Congenita 2 Omim
  
  Clinical Features Jackson and Lawler (1951-52) reported 6 affected members of 3 generations of a family with pachyonychia congenita. In 4 generations of a family, Vineyard and Scott (1961) observed steatocystoma associated with pachyonychia congenita.
- Pachyonychia Congenita 1 Omim
  
  Feinstein et al. (1988) classified 168 reported cases into 4 types, of which type IV, present in 7.2% of the cases, had laryngeal lesions, hoarseness, mental retardation, hair anomalies, and alopecia.
Green Nails Wikipedia

It is also able to decompose hydrocarbons and has been used to break down tarballs and oil from oil spills . [3] [4] See also [ edit ] Nail anatomy List of cutaneous conditions References [ edit ] ^ James, William; Berger, Timothy; Elston, Dirk (2005). Andrews' Diseases of the Skin: Clinical Dermatology . (10th ed.). Saunders. ISBN 0-7216-2921-0 . ^ Balcht, Aldona; Smith, Raymond (1994). ... Informa Health Care. pp. 83–84. ISBN 978-0-8247-9210-7 . ^ A. Y. Itah; J. P. ... World Journal of Microbiology and Biotechnology . 21 (6–7): 1317–1322. doi : 10.1007/s11274-004-6694-z . ^ AVI Biopharma (2007-01-18). "Antisense antibacterial method and compound" .
Torsades De Pointes Wikipedia

Phase 3: Calcium channels close (inward Ca ++ stops), but potassium channels are still open (outward K + current); this persists until the cells gain back normal polarization (repolarization achieved). Please note that phase 0 leads to a net gain of Na + , while phases 1-3 lead to a net loss of K + . ... United Kingdom: Elsevier. p. 568. ISBN 978-0-7020-3084-0 . ^ a b Yap, Yee Guan; Camm, A John (2017-01-17). ... PMID 14594906 . ^ Napolitano, C.; Priori, S. G.; Schwartz, P. J. (1994-01-01). "Torsade de pointes. Mechanisms and management". ... Cardiology Research and Practice . 2010 : 1–4. doi : 10.4061/2010/524764 . PMC 3021856 . ... Circulation . 89 (1): 206–15. doi : 10.1161/01.CIR.89.1.206 . PMID 8281648 . ^ a b Thomas, Simon H.

KCNH2, SCN5A, CYP3A4, KCNQ1, KCNE1, ANK2, KCNE2, NAA10, KCNJ5, SCN4B, SCN10A, SNTA1, ALG10B, CAV3, CALM2, CALM1, AKAP9, TRDN, C18orf21, NOS1AP, IL6, KCNK3, KCNA5, ATP12A, ATP4A, CYP19A1, KCNA4, ACE, SLC24A3, SLCO3A1, RAPGEF4, SLCO1B1, ATP5F1A, CRP, CYP2D6, NR1I2, TNF, SRL, SARDH, TLX2, HTR4, RYR2, RNPEP, ABCB1, ORM1, MSD, LBR, IL1A, ADRB1, IL1B, ADRA2C
Abomasitis Wikipedia

The Canadian Veterinary Journal . 27 (4): 194. ISSN 0008-5286 . PMC 1680202 . PMID 17422653 . ^ a b c d Navarre, Christine B.; Baird, A. N.; Pugh, D. G. (2012-01-01), Pugh, D. G.; Baird, A. N. (eds.), "Chapter 5 - Diseases of the Gastrointestinal System" , Sheep and Goat Medicine (Second Edition) , W.B. ... PMID 16701586 . ^ Constable, Peter D.; Hinchcliff, Kenneth W.; Done, Stanley H.; Grünberg, Walter, eds. (2017-01-01), "7 - Diseases of the Alimentary Tract: Nonruminant" , Veterinary Medicine (Eleventh Edition) , W.B. Saunders, pp. 175–435, ISBN 978-0-7020-5246-0 , retrieved 2019-12-13 ^ a b c Constant, Caroline; Francoz, David; Babkine, Marie; Desrochers, André (2016). ... PMID 27144594 . ^ a b c Fubini, Susan L.; Yeager, Amy E.; Divers, Thomas J. (2018-01-01), Peek, Simon F.; Divers, Thomas J.
Nuclear Anxiety Wikipedia

See also [ edit ] Eco-anxiety Ecological grief Psychic numbing References [ edit ] ^ Feldman, James W. (2017-05-01). Nuclear Reactions: Documenting American Encounters with Nuclear Energy . University of Washington Press. ISBN 978-0-295-99963-0 . ^ Smith, Tom W. (24/1988). "A Report: Nuclear Anxiety". Public Opinion Quarterly . 52 (4): 557. doi : 10.1086/269131 . Check date values in: |date= ( help ) This article related to nuclear weaponry is a stub .
Xerotic Eczema Wikipedia

Shaving can cause it to become inflamed . [ citation needed ] Contents 1 Diagnosis 2 Treatment 3 See also 4 References Diagnosis [ edit ] This section is empty. ... If the re-hydration process does not alleviate the symptoms, moisturizers such as Lac-Hydrin 5% or 12% moisturizer containing urea can be used; furthermore, if the skin becomes inflamed or cracked, mid- to high-potency corticosteroids can be used. [4] A study published in 2005 found positive results from soaking the affected area in water for twenty minutes and then applying mid- to high-strength corticosteroid ointment. [5] See also [ edit ] Skin lesion References [ edit ] ^ James, William; Berger, Timothy; Elston, Dirk (2005). Andrews' Diseases of the Skin: Clinical Dermatology . (10th ed.). Saunders. ISBN 0-7216-2921-0 . ^ Rapini, Ronald P.; Bolognia, Jean L.; Jorizzo, Joseph L. (2007). ... ISBN 978-1-4160-2999-1 . ^ Mash, Marlene J.; Fedor, Maria; Bonnington, Lucy (2007-01-01). "Skin Problems" . Primary Care Geriatrics : 612–623. doi : 10.1016/B978-032303930-7.50061-4 . ISBN 9780323039307 . ^ Mash, Marlene J.; Fedor, Maria; Bonnington, Lucy (2007-01-01). "Skin Problems" . Primary Care Geriatrics : 612–623. doi : 10.1016/B978-032303930-7.50061-4 .
Glycogen Storage Disease Type 0 Wikipedia

Glycogen storage disease type 0 Glycogen storage disease type 0 has defect in glycogen synthase Specialty Medical genetics Glycogen storage disease type 0 is a disease characterized by a deficiency in the glycogen synthase enzyme (GSY). ... Contents 1 Signs and symptoms 2 Causes 3 Pathophysiology 4 Diagnostic 4.1 Laboratory Studies 4.2 Imaging Studies 4.3 Other Tests 4.4 Procedures 4.5 Histologic Findings 4.6 Differential Diagnoses 4.7 Types 5 Treatment 6 Epidemiology 6.1 Mortality/Morbidity 6.2 Sex 6.3 Age 7 References 8 External links Signs and symptoms [ edit ] The most common clinical history in patients with glycogen-storage disease type 0 (GSD-0) is that of an infant or child with symptomatic hypoglycemia or seizures that occur before breakfast or after an inadvertent fast. ... Serum electrolytes calculate the anion gap to determine presence of metabolic acidosis ; typically, patients with glycogen-storage disease type 0 (GSD-0) have an anion gap in the reference range and no acidosis. ... In patients with glycogen-storage disease type 0, hyperlipidemia is absent or mild and proportional to the degree of fasting. ... Types [ edit ] There are two types of this\\e condition glycogen storage disease type 0 to be considered, they are: [3] [4] Glycogen storage disease due to liver glycogen synthase deficiency Glycogenosis due to muscle and heart glycogen synthase deficiency Treatment [ edit ] The goal for treatment of Glycogen-storage disease type 0 is to avoid hypoglycemia.

GYS2, CPLANE1
Scanning Speech Wikipedia

Crash Course Neurology . Elsevier Limited. ISBN 978-0-7234-3469-6 . ^ a b c Terence R. Anthoney (1994). ... Boca Raton: CRC Press. pp. 482–483. ISBN 978-0-8493-8631-2 . ^ Stachowiak, Julie. "Scanning Speech" . ms.about.com . Retrieved 2012-01-04 . ^ "Charcot's triad I" . whonamedit.com . Retrieved 2012-01-04 . ^ Thomas, Huw. "Cerbellar Signs including Cerebellar Ataxia" . Retrieved 2012-01-04 . External links [ edit ] Classification D ICD - 10 : R47.1 MeSH : D004401

NPC1, PDE8B
Abortion In Belgium Wikipedia

Archived from the original on 2016-07-01 . Retrieved 2016-06-08 . ^ "World Abortion Policies 2013" . ... Continuum International Publishing Group. p. 389. ISBN 978-0-8264-1227-0 . Retrieved 7 January 2012 . ^ Barker, Eileen (15 April 2010). ... Political Leaders of Contemporary Western Europe . Greenwood Press. p. 30 . ISBN 978-0-313-28623-0 . ^ Roberts, Geoffrey K.; Hogwood, Patricia (2003). ... Manchester University Press. p. 84 . ISBN 978-0-7190-5421-1 . ^ Witte, Els; Craeybeckx, Jan; Meynen, Alain (2010). ... Facts on File. 2006. p. 92. ISBN 978-0-8160-6078-8 . ^ Sandro Magister, "Obama's Pick for Vice President Is Catholic.