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Arteriovenous Malformation Wikipedia

Surgical Endoscopy . 30 (4): 1667–9. doi : 10.1007/s00464-015-4337-0 . PMID 26156615 . ^ a b Tellapuri, S; Park, HS; Kalva, SP (August 2019).

RASA1, AKT1, PTEN, PIK3CA, TEK, NOTCH1, FANCF, RFWD3, FANCI, ARHGAP31, FANCG, FANCE, FANCB, GUSB, FANCD2, FANCC, FANCA, DOCK6, FANCL, RBPJ, DLL4, MNX1, ERCC4, UBE2T, MAD2L2, ADAMTS3, XRCC2, VHL, VEGFA, RAD51, RAD51C, SLPI, FANCM, GNAQ, BRCA1, EOGT, ENG, CCBE1, FAT4, ACVRL1, PALB2, ARL6IP6, CBS, BRCA2, BRIP1, SLX4, SMAD4, IL6, TNF, KRAS, APOE, EPHB4, SMS, ANGPTL4, IL1B, GDF2, RSS, BRAF, ANGPT2, MAP2K1, MMP9, SOX17, PIK3CB, PIK3CD, PIK3CG, VWF, ANXA5, AGT, VCAM1, MMP3, MMP2, PART1, ICAM1, ELN, F3, EDN1, ACE, CLN3, CD6, SMG8, KDR, ABCA2, PAGE4, SYNGAP1, TGFB2, TGFB3, TGFBR1, THBS1, MIR137, SMUG1, TLR4, PDGFD, NES, COL18A1, CDKN2B-AS1, MIR210, PIK3R4, MIR195, KLF2, TMEM100, ESAM, BMP10, TGFB1, MVP, NOS1AP, MIR18A, PAGE1, BLNK, NOTCH3, STAT3, DLAT, MTOR, FN1, FLT1, FOXC2, FGF2, F2, ETS1, EPHB2, EFNB2, EFNA1, EDNRA, CRYAB, SPP1, COX8A, CDKN2A, CDH5, CD34, CCT, CASP3, VPS51, BDNF, ANPEP, ALX3, ALCAM, GJA5, HIF1A, HRAS, IFNG, SOX2, SNCA, SLC2A1, S100B, RBBP8, PTPN14, MAPK1, PLOD2, PECAM1, PDGFRB, PCYT1A, NOTCH4, ACTB, NOS3, NOS2, NOS1, MYB, MSH3, ABCC1, MGP, MDM4, MDM2, ITGB8, CERNA3
- Arteriovenous Malformation Mayo Clinic
  
  Overview An arteriovenous malformation (AVM) is a tangle of blood vessels that irregularly connects arteries and veins, disrupting blood flow and oxygen circulation. Arteries move oxygen-rich blood from the heart to the brain and other organs. Veins drain the oxygen-depleted blood back to the lungs and heart. When an AVM disrupts this critical process, the surrounding tissues might not get enough oxygen. Also, because the tangled blood vessels in an AVM do not form properly, they can weaken and rupture. If an AVM in the brain ruptures, it can cause bleeding in the brain, stroke or brain damage.
- Pulmonary Arteriovenous Malformation GARD
  
  Pulmonary arteriovenous malformation (PAVM) is a condition that affects blood flow between the heart and the lungs. Affected people have an abnormal connection between the pulmonary vein (carries blood from the lungs to the heart) and pulmonary artery (carries blood from the heart to the lungs). As a result, blood may not be properly oxygenated by the lungs. Some affected people may have no signs or symptoms of the condition. When present, symptoms may include difficulty breathing, nosebleeds, shortness of breath, difficulty exercising, and blue skin. Most people with PAVM have an inherited condition called hereditary hemorrhagic telangiectasia , which is inherited in an autosomal dominant manner.
Mycosis Fungoides Wikipedia

"How I treat mycosis fungoides and Sézary syndrome" . Blood . 114 (20): 4337–53. doi : 10.1182/blood-2009-07-202895 .

TNFRSF1B, CTLA4, CD28, TNFRSF8, TRBV20OR9-2, TP53, IL2, MIR155, CDKN2A, DPP4, STAT3, IFNG, TRBV16, TRBV7-9, PTEN, TRBC1, TRB, IL4, FOXP3, IL17A, TSPYL2, NAV3, TOX, HLA-DQB1, PIK3CA, AKT1, TNF, IL10, CCR4, IL32, IL6, TARP, ALK, STAT4, FAS, CD40LG, NCAM1, CCR10, CDKN2B, PIK3CB, TRG, PIK3CD, PIK3CG, TRGC1, VEGFA, IL15, IL16, IRF4, IL7, PTGS2, JUNB, KIR3DL2, PTPRC, CADM1, SMUG1, CCL27, NOTCH1, CCL21, VEGFC, MAPK1, CXCL11, LINC02605, CXCR3, HLA-DRB1, MIR223, HLA-B, MIR93, B2M, MALAT1, MIR21, SOCS1, CFLAR, SOCS3, USP14, USP2, MIR34A, RBM45, MIR17, IL31, TRIP13, DEFB103A, PLA2G4D, GRAP2, TCL1B, MIR122, MIR126, DEFB103B, TP63, AHSA1, MTCO2P12, LUNAR1, ERVK-11, ERVK-22, WNT1, YY1, ERVK-12, ERVK-2, DEFB4B, HBD, ZNF217, CDKN2B-AS1, MIR326, CXCR4, AIMP2, NR4A3, TCL1A, TCIRG1, PDPN, IL17F, UVRAG, CD274, ERVW-1, RNF34, RAPH1, BCL11B, NFKBIZ, MIB1, TLR7, IL23A, ATF7IP, INTS2, GDE1, DUSP22, TLR9, DIABLO, SOX18, KRT20, RHOF, PPP1R2C, TRBJ2-1, HM13, TRBJ2-7, GNG8, CKAP4, IL33, ACSBG1, PWWP3A, HAVCR2, KLK5, PART1, RNF19A, DNM3, POLDIP2, INTS1, SGSM3, DLL1, QTRT1, SHARPIN, TRBV5-7, VDR, ACVRL1, UCHL1, GZMB, FGFR1, FKBP4, FKBP5, FLT4, MTOR, FUT4, GATA3, GNAO1, GNAS, GRN, HDAC2, TNFRSF4, HELLS, HHEX, HLA-A, HLA-DPB1, HLA-DQB2, HLA-G, HNRNPK, HRAS, ICAM3, IFNA1, FGF3, FGF2, EPHB2, E2F1, ANPEP, ARG1, CCND1, BCL2, BCL7A, BCR, BRCA2, CASP8, CBL, CD7, MS4A1, CD40, CD44, CDO1, CREB1, CRK, MAPK14, CTNNB1, CTSB, DEFB1, DEFB4A, IFNA13, IL1B, IL5, NOS2, PAK1, PDCD1, PKM, PPP1R1A, PSMB6, RAF1, RPE65, SATB1, PARP1, CCL5, CCL20, CXCL12, SELL, SELPLG, SLPI, SS18, MAP3K7, TGFB1, TLR2, TLR4, NR2C2, NPM1, NOS1, IL9, NKG7, IL13, ITGAM, ITGB2, JAK2, JAK3, JUND, KIT, KRT1, LGALS1, LPP, LTA, MEF2C, MKI67, MLH1, MMP1, MMP2, MMP9, MTAP, COX2, MYBL2, MYCL, SCT
- Mycosis Fungoides OMIM
  
  Description Mycosis fungoides is a malignant T-cell lymphoma of the skin, first reported (and named) by Alibert (1835). Sezary syndrome is a leukemic variant of mycosis fungoides defined by erythroderma with greater than 80% of the skin showing redness, adenopathy and greater than 1,000 circulating Sezary cells/microliter with a CD4+CD26- or CD4+CD7- phenotype. Sezary cells have a type 2 helper T cell cytokine profile. Sezary syndrome has a median overall survival time of only 2.4 years in patients with Sezary cells at a density of greater than 10,000 cells/microliter or 5.4 years in patients with 1,000-10,000 Sezary cells/microliter. Mycosis fungoides and Sezary syndrome are the most common cutaneous T-cell lymphomas. Sezary syndrome can arise de novo or can appear following years of chronic mycosis fungoides.
- Mycosis Fungoides MedlinePlus
  
  Mycosis fungoides is the most common form of a type of blood cancer called cutaneous T-cell lymphoma. Cutaneous T-cell lymphomas occur when certain white blood cells, called T cells , become cancerous; these cancers characteristically affect the skin, causing different types of skin lesions. Although the skin is involved, the skin cells themselves are not cancerous. Mycosis fungoides usually occurs in adults over age 50, although affected children have been identified. Mycosis fungoides may progress slowly through several stages, although not all people with the condition progress through all stages.
- Sézary Syndrome MedlinePlus
  
  Sézary syndrome is an aggressive form of a type of blood cancer called cutaneous T-cell lymphoma. Cutaneous T-cell lymphomas occur when certain white blood cells, called T cells, become cancerous; these cancers characteristically affect the skin, causing different types of skin lesions. In Sézary syndrome, the cancerous T cells, called Sézary cells, are present in the blood, skin, and lymph nodes. A characteristic of Sézary cells is an abnormally shaped nucleus, described as cerebriform. People with Sézary syndrome develop a red, severely itchy rash (erythroderma) that covers large portions of their body.
- Classic Mycosis Fungoides Orphanet
  
  Classical mycosis fungoides is the most common type of mycosis fungoides (MF; see this term), a form of cutaneous T-cell lymphoma, and is characterized by slow progression from patches to more infiltrated plaques and eventually to tumors. Epidemiology The annual incidence of MF and its variants is estimated at between 1/350,000 and 1/110,000, with classical MF accounting for about 80-90% of MF cases. The male to female ratio is 2:1. Classical MF predominantly affects adults and the elderly (median age at diagnosis: 55-60 years). Clinical description The disease first manifests by skin lesions consisting of flat patches, preferentially located asymmetrically on the buttocks and other sun-protected areas (lower trunk and thighs, and the breasts in women). Usually, patches are hypo- or hyperpigmented in dark-skinned individuals.
- Mycosis Fungoides GARD
  
  Mycosis fungoides is a disease in which T-cell lymphocytes (a type of white blood cell) become malignant (cancerous) and affect the skin. This condition is one of the most common types of T-cell lymphoma . Mycosis fungoides is characterized by a scaly, red rash that develops on the skin, particularly on areas that are not usually exposed to the sun. The rash may last for months or years without causing any symptoms. Over time, a thin, reddened, eczema-like rash may develop, followed by thickened, red patches of skin. Finally, tumors form which may develop into ulcers and become infected. Mycosis fungoides is difficult to cure. Treatment is usually palliative, with the intention of relieving symptoms and improving the quality of life.