��-��-(��315bet.net��)-��-pp��-��360bet.net��-��-��-��-(��52bet.net��)

Advanced

FindZebra Summary is loading...

Estrogen-Dependent Condition Wikipedia

Springer Science & Business Media. pp. 423–. ISBN 978-1-84882-513-0 . ^ Merril D. ... Textbook of Medicine . Orient Blackswan. pp. 1113–. ISBN 978-81-250-1266-5 . ^ a b c d Edward J. ... Biochemistry and Function of Sterols . CRC Press. pp. 26–27. ISBN 978-0-8493-7674-0 . ^ Michael Crocetti; Michael A. ... Oski's Essential Pediatrics . Lippincott Williams & Wilkins. pp. 564–. ISBN 978-0-7817-3770-8 . ^ W. ... Hormones and Vitamins in Cancer Treatment . CRC Press. pp. 33–. ISBN 978-0-8493-5973-6 . ^ Eugene A.
Gastric Distension Wikipedia

Saunders. 2016. pp. 130–147. ^ Emergency Medicine (2 ed.). ... Elsevier. 2017. pp. 31–42. ^ Abdominal Imaging (2 ed.). Elsevier. 2017. pp. 126–136. ^ Current Surgical Therapy (12 ed.). Elsevier. 2017. pp. 130–135. ^ Essentials of Radiology (3 ed.). Saunders. 2014. pp. 115–156.
Androgen-Dependent Condition Wikipedia

Textbook of Natural Medicine . Elsevier Health Sciences. pp. 1157–. ISBN 1-4377-2333-0 . ^ Steven B. ... Springer International Publishing. pp. 25–. ISBN 978-3-319-16760-2 . ^ Krishna; Usha R. (1 March 2000). Adolescence . Orient Blackswan. pp. 124–. ISBN 978-81-250-1794-3 . ^ Mark G. ... Springer Science & Business Media. pp. 361–. ISBN 978-1-84882-034-0 . ^ P. ... Springer Science & Business Media. pp. 222–. ISBN 978-1-59745-108-6 . ^ Kenneth L.
Breast Atrophy Wikipedia

Springer Science & Business Media. pp. 257–258. ISBN 978-3-540-69560-8 . ^ Melvin A. ... Springer Science & Business Media. pp. 42–. ISBN 978-3-540-89873-3 . ^ Kristen A. ... Breast . Cambridge University Press. pp. 1–. ISBN 978-0-521-88159-3 . ^ Ricardo Azziz (3 July 2007). ... Maternity and Pediatric Nursing . Lippincott Williams & Wilkins. pp. 213 –. ISBN 978-0-7817-8055-1 . ^ J.P. ... Springer Science & Business Media. pp. 99–. ISBN 978-1-4612-5064-7 . ^ a b Julia A.
Interictal Dysphoric Disorder Wikipedia

Neuropsychiatric Disorders . Springer. pp. 107–. ISBN 978-4-431-53871-4 . ^ Steven C. ... Psychiatric Controversies in Epilepsy . Elsevier. pp. 54–. ISBN 978-0-08-055959-9 . ^ Michael R. ... The Neuropsychiatry of Epilepsy . Cambridge University Press. pp. 82–. ISBN 978-1-139-49789-3 . ^ Frank Gilliam; Andres M. ... Depression and Brain Dysfunction . Taylor & Francis. pp. 219–. ISBN 978-1-84214-214-1 . ^ Gregory P. ... Neuropsychology of Epilepsy and Epilepsy Surgery . Oxford University Press. pp. 147–. ISBN 978-0-19-970699-0 .
Autosomal Dominant Popliteal Pterygium Syndrome Orphanet

Epidemiology The prevalence of autosomal-dominant-popliteal pterygium syndrome (AD-PPS) is unknown. However, based on the occurence of cleft lip/palate in the general population, the prevalence at birth of popliteal pterygium syndrome (PPS) has been suggested at 1/300,000 with more than 200 cases of AD-PPS being reported worldwide. ... Growth and intellectual development are normal in patients with AD-PPS although there are some reports of delayed language acquisition and learning disabilities. Etiology AD-PPS is associated with mutations in the IRF6 gene (1q32.2-q32.3), involved in the formation of epithelial tissues, especially the periderm. ... In fact, affected individuals in the same family, having the same mutation in IRF6 , have been diagnosed with AD-PPS and with VWS. The cause of this variable expressivity is not known. ... Management of the other manifestations of AD-PPS is generally supportive and symptomatic.

IRF6, RIPK4, FBXO7, B3GLCT, AVP, ACTB, KITLG, MUL1, POFUT2, PRKN, NHLH1, MEFV, FAS, IL10, CXCL8, IFNG, CHUK, AVPR2, AQP2, CBLL2
- Popliteal Pterygium Syndrome MedlinePlus
  
  Popliteal pterygium syndrome is a condition that affects the development of the face, skin, and genitals. Most people with this disorder are born with a cleft lip , a cleft palate (an opening in the roof of the mouth), or both. Affected individuals may have depressions (pits) near the center of the lower lip, which may appear moist due to the presence of salivary and mucous glands in the pits. Small mounds of tissue on the lower lip may also occur. In some cases, people with popliteal pterygium syndrome have missing teeth. Individuals with popliteal pterygium syndrome may be born with webs of skin on the backs of the legs across the knee joint, which may impair mobility unless surgically removed.
- Popliteal Pterygium Syndrome Wikipedia
  
  Genetics [ edit ] The genetic locus for PPS was localized to chromosome 1 in 1999. ... Most reported cases are sporadic; advanced parental age is found in a number of these cases, suggesting new mutations. [ citation needed ] The term PPS has also been used for two rare autosomal recessively inherited conditions: Lethal PPS and PPS with Ectodermal Dysplasia . Although both conditions feature a cleft lip/palate, syngnathia , and popliteal pterygium , they are clinically distinguishable from the autosomal dominant case. Lethal PPS is differentiated by microcephaly , corneal aplasia , ectropion , bony fusions, hypoplastic nose and absent thumbs , while PPS with Ectodermal Dysplasia is differentiated by woolly hair , brittle nails , ectodermal anomalies, and fissure of the sacral vertebrae . [6] Relationship to Van der Woude syndrome [ edit ] Van der Woude syndrome (VDWS) and popliteal pterygium syndrome (PPS) are allelic variants of the same condition; that is, they are caused by different mutations of the same gene. PPS includes all the features of VDWS, plus popliteal pterygium, syngnathia, distinct toe/nail abnormality, syndactyly , and genito- urinary malformations. [7] Diagnosis [ edit ] This section is empty. ... You can help by adding to it . ( May 2017 ) Epidemiology [ edit ] The diagnosis of PPS has been made in several ethnic groups, including Caucasian , Japanese , and sub-Saharan African .
- Popliteal Pterygium Syndrome OMIM
  
  Syndactyly is a useful diagnostic sign in PPS because it is not seen in most of the syndromes that should be considered in the differential diagnosis. ... In the hands, fusion of fingers 3 and 4 was the most common type of syndactyly. Thus, the syndactyly in PPS most closely resembles type I (185900). ... They suggested that if this sign is present in a patient in combination with cleft palate and/or lip, even without popliteal webbing, the diagnosis of PPS should be made. Lees et al. (1999) reported clinical features in 3 families and 1 isolated individual with PPS. ... Ghassibe et al. (2004) stated that because syndactyly and synechiae are major signs for PPS, these 2 patients were considered to have PPS, whereas the 6 other affected family members were classified as VWS, thus demonstrating that a single mutation could be responsible for both syndromes. ... In a neonate with a clinical diagnosis of PPS, Leslie et al. (2015) identified a homozygous mutation in the IRF6 gene (607199.0019).
- Popliteal Pterygium Syndrome GARD
  
  Popliteal pterygium syndrome is a condition that affects the development of the face, skin, and genitals. Most people with this disorder are born with a cleft lip and/or a cleft palate . Other symptoms may include depressions (pits) near the center of the lower lip, small mounds of tissue on the lower lip, or missing teeth. Other features may include webs of skin on the backs of the legs across the knee joint (popliteal pterygium), webbing or fusion of the fingers or toes (syndactyly), triangular shaped folds of skin over the nails of the large toes, and tissue connecting the upper and lower eyelids or the upper and lower jaw. Affected people may also have abnormal genitals. There is no growth delay and intelligence is usually normal.
Adipomastia Wikipedia

Mechanisms of Clinical Signs - EPub3 . Elsevier Health Sciences. pp. 599–. ISBN 978-0-7295-8561-3 . ^ William T. ... Springer Science & Business Media. pp. 246–. ISBN 978-1-4614-6633-8 . ^ a b c Z. ... Karger Medical and Scientific Publishers. pp. 21–. ISBN 978-3-8055-8220-9 . ^ a b Seth Thaller; Mimis Cohen (28 February 2013). Cosmetic Surgery After Massive Weight Loss . JP Medical Ltd. pp. 133–. ISBN 978-1-907816-28-4 . ^ a b Nihal Thomas (30 April 2016). ... Springer Science & Business Media. pp. 232–. ISBN 978-3-662-04491-9 . ^ Michael Steven Kappy; David B.

XK, NLRP1
Post-Polio Syndrome Wikipedia

Despite this, no absolutely defined causes of PPS are known. The most widely accepted theory of the mechanism behind the disorder is "neural fatigue". ... Objective assessment of muscle strength in PPS patients may not be easy. Changes in muscle strength are determined in specific muscle groups using various muscle scales that quantify] strength, such as the Medical Research Council (MRC) scale. magnetic resonance imaging , neuroimaging , and electrophysiological studies, muscle biopsies , or spinal fluid analysis may also be useful in establishing a PPS diagnosis. [11] Management [ edit ] PPS treatment concerns comfort (relieving pain via analgesics ) and rest (via use of mechanisms to make life easier, such as a powered wheelchair) and is generally of palliative care. ... Medications for fatigue, such as amantadine and pyridostigmine , are ineffective in the management of PPS. [12] Muscle strength and endurance training are more important in managing the symptoms of PPS than the ability to perform enduring aerobic activity. ... PPS patients do not have an elevated risk of ALS. [6] No sufficient longitudinal studies have been conducted on the prognosis of PPS, but speculations have been made by several physicians based on experience. ... United States: Churchill Livingstone. pp. 489–490. ISBN 978-0-443-04345-1 . ^ Lambert, David A; Giannouli, Eleni; Schmidt, Brian J (2005).
- Post-Polio Syndrome Mayo Clinic
  
  Overview Post-polio syndrome is a group of potentially disabling signs and symptoms that appear decades after the initial polio illness. These signs and symptoms usually appear between 30 to 40 years after having polio. Infection from the polio virus once caused paralysis and death. However, the introduction of the inactivated polio vaccine in the 1950s greatly reduced polio's spread. Today, very few people in developed countries are paralyzed from the polio virus, usually related to a reaction from one form of the vaccine. However, there remain many people who had polio at a young age who now later in life might develop the post-polio syndrome.
- Postpoliomyelitis Syndrome Orphanet
  
  Postpoliomyelitis syndrome (PPS) is a neurologic disorder characterized by the development of new neuromuscular symptoms such as progressive muscular weakness or abnormal muscle fatigability occurring in survivors of the acute paralytic form of poliomyelitis (see this term), 15-40 years after recovery from the disease, and that is unexplained by other medical causes.
- Post Polio Syndrome GARD
  
  Post polio syndrome (PPS) is a condition that affects polio survivors years after recovery from the initial polio illness. Symptoms and severity vary among affected people and may include muscle weakness and a gradual decrease in the size of muscles (atrophy); muscle and joint pain; fatigue; difficulty with gait; respiratory problems; and/or swallowing problems. Only a polio survivor can develop PPS. While polio is a contagious disease, PPS is not. The exact cause of PPS years after the first episode of polio is unclear, although several theories have been proposed.
Peters Plus Syndrome GARD

Peters plus syndrome (PPS) affects many different parts of the body. The most common affected parts are the eyes. PPS causes abnormal development of the structures in the front of the eye, known as Peters anomaly . ... The severity of symptoms varies from person to person. Because PPS has only been reported in a small number of people, it is not clear how this condition changes with age. PPS is caused by a variant in the B3GLCT gene and is inherited in an autosomal recessive fashion.

B3GLCT, B3GALT1, POFUT2, ADAMTS9, ADAMTS20
- Peters-Plus Syndrome OMIM
  
  A number sign (#) is used with this entry because of evidence that the Peters-plus syndrome (PTRPLS) is caused by homozygous or compound heterozygous mutation in the beta-1,3-galactosyltransferase-like gene (B3GALTL; 610308) on chromosome 13q12. Description Patients with Peters-plus syndrome exhibit ocular features, systemic malformations, and variable degrees of developmental delay. Ocular abnormalities involve the anterior chamber, and in most patients consist of Peters anomaly, which is characterized by corneal clouding and iridolenticulocorneal adhesions. Growth retardation, short stature, and brachydactyly appear to be present in all patients, and developmental delay is frequent, whereas external ear anomalies, cleft lip and/or palate, and cardiac and genitourinary malformations are less common (Dassie-Ajdid et al., 2009). Clinical Features According to van Schooneveld et al. (1984), Peters (1906) described 3 brothers with the disorder that bears his name (Peters anomaly; see ASGD5, 604229).
- Peters-Plus Syndrome Wikipedia
  
  Peters-plus syndrome Other names Krause–van Schooneveld–Kivlin syndrome This condition is inherited in an autosomal recessive manner Peters-plus syndrome or Krause–Kivlin syndrome is a hereditary syndrome defined by Peters' anomaly , dwarfism and intellectual disability . [1] [2] Contents 1 Signs and symptoms 2 Cause 3 History 4 See also 5 References 6 External links Signs and symptoms [ edit ] Features of this syndrome include Peters' anomaly , corneal opacity , central defect of Descemet's membrane , and shallow anterior chamber with synechiae between the iris and cornea. [ citation needed ] Craniofacial abnormalities commonly seen in patients with PPS include hypertelorism , ear malformations, micrognathia , round face and broad neck, and cleft lip and palette . [1] Infants are commonly born small for gestational age and have delayed growth. ... These mutations of the B3GLCT gene lead to the production of an abnormally short, nonfunctional version of the beta 3- glucosyltransferase (B3Glc-T) enzyme, which disrupts glycosylation. [5] The phenotypic effects of the B3GLCT mutations result in a triad of well known phenotypes; Peters anomaly (also classified as anterior segment defects , a defect in the anterior cornea), short stature, brachydactyly , in addition to several other less frequently observed phenotypes. [6] A study of 55 patients with Peters-plus-related phenotypes, but lacking the most common combination (Peters anomaly, short stature, and brachydactyly), revealed none of those cases displayed mutation in the B3GLCT gene. Thus PPS-like signs and symptoms, when they occur independently of each other, provide strong evidence that the B3GLCT gene mutation is in fact responsible for actual cases Peters-plus syndrome. [7] History [ edit ] Krause–van Schooneveld–Kivlin syndrome is listed as a "rare disease" by the Office of Rare Diseases (ORD) of the National Institutes of Health (NIH). [8] It was characterized in 1984 by van Schooneveld. [9] See also [ edit ] Peters anomaly References [ edit ] ^ a b c Smith's Recognizable Patterns of Human Malformation (7 ed.). Elsevier. 2013. pp. 752–795. ^ "Krause–Kivlin syndrome ' " .
- Peters Plus Syndrome GeneReviews
  
  Summary Clinical characteristics. Peters plus syndrome is characterized by anterior chamber eye anomalies, short limbs with broad distal extremities, characteristic facial features, cleft lip/palate, and variable developmental delay/intellectual disability. The most common anterior chamber defect is Peters' anomaly, consisting of central corneal clouding, thinning of the posterior cornea, and iridocorneal adhesions. Cataracts and glaucoma are common. Developmental delay is observed in about 80% of children; intellectual disability can range from mild to severe. Diagnosis/testing. The diagnosis of Peters plus syndrome is a clinical diagnosis that can be confirmed by identification of biallelic B3GLCT pathogenic variants on molecular genetic testing. Management. Treatment of manifestations: Consideration of corneal transplantation (penetrating keratoplasty) for severe bilateral corneal opacification before age three to six months to prevent amblyopia; consideration of simple separation of iridocorneal adhesions in mild cases; management of amblyopia by a pediatric ophthalmologist; surgical/medical intervention for glaucoma as needed; developmental/educational interventions as needed.
- Peters Plus Syndrome Orphanet
  
  Peters plus syndrome is an autosomal recessively inherited syndromic developmental defect of the eye (see this term) characterized by a variable phenotype including Peters anomaly (see this term) and other anterior chamber eye anomalies, short limbs, limb abnormalities (i.e. rhizomelia and brachydactyly), characteristic facial features (upper lip with cupid bow, short palpebral fissures), cleft lip/palate, and mild to severe developmental delay/intellectual disability. Other associated abnormalities reported in some patients include congenital heart defects (i.e. hypoplastic left heart, absence of right pulmonary vein, bicuspid pulmonary valve), genitourinary anomalies (hydronephrosis, renal hypoplasia, renal and ureteral duplication, multicystic dysplastic kidneys, glomerulocystic kidneys) and congenital hypothyroidism.
- Peters Plus Syndrome MedlinePlus
  
  Peters plus syndrome is an inherited condition that is characterized by eye abnormalities, short stature, an opening in the lip (cleft lip ) with or without an opening in the roof of the mouth (cleft palate ), distinctive facial features, and intellectual disability. The eye problems in Peters plus syndrome occur in an area at the front part of the eye known as the anterior segment. The anterior segment consists of structures including the lens, the colored part of the eye (iris), and the clear covering of the eye (cornea). An eye problem called Peters anomaly is the most common anterior segment abnormality seen in Peters plus syndrome. Peters anomaly involves abnormal development of the anterior segment, which results in a cornea that is cloudy (opaque) and causes blurred vision.
Buruli Ulcer Wikipedia

. ^ a b c d e f g h i j Guarner 2018 , pp. 3–4. ^ Röltgen & Pluschke 2020 , p. 9. ^ a b Kpeli & Yeboah-Manu 2019 , pp. 227–228. ^ Bravo 2019 , p. 118. ^ a b c Zingue et al. 2018 , pp. 10–13. ^ Demangel, Stinear & Cole 2009 , p. 52. ^ Tortoli 2014 , p. 739, Figure 7. ^ a b c d e f g h i j Guarner 2018 , pp. 1–2. ^ Yotsu et al. 2018 , pp. 247–248. ^ a b c d Yotsu et al. 2018 , p. 251. ^ Röltgen & Pluschke 2020 , pp. 7–8. ^ a b c Yotsu et al. 2018 , p. 250. ^ a b Jacobsen & Padgett 2010 , pp. e678–e679. ^ a b c Manry 2020 , p. 3. ^ Vincent et al. 2018 , pp. 1–17. ^ Röltgen et al. 2019 , pp. 190–191. ^ a b c d Röltgen et al. 2019 , pp. 185–186. ^ Röltgen et al. 2019 , pp. 186–187. ^ a b c Guarner 2018 , pp. 4–6. ^ Röltgen et al. 2019 , pp. 189–190. ^ "Buruli ulcer (Mycobacterium ulcerans infection) – Treatment" . ... Retrieved 19 June 2020 . ^ a b c d e f Yotsu et al. 2018 , pp. 251–252. ^ Yotsu, Richardson & Ishii 2018 , p. 3. ^ a b c d e Guarner 2018 , pp. 6–7. ^ Kpeli & Yeboah-Manu 2019 , pp. 235–236. ^ a b World Health Organization 2012 , pp. 6–9. ^ Zimmerman, Finn & Curtis 2018 , pp. 682–684. ^ Simpson et al. 2019 , pp. e912–e913. ^ a b Simpson et al. 2019 , pp. e917–e918. ^ a b Yotsu et al. 2015 , pp. 1033–1034. ^ Tabah et al. 2019 , pp. 51–54. ^ Johnson 2019 , pp. 62–63. ^ Suzuki et al. 2019 , pp. 87–88. ^ Yotsu et al. 2018 , p. 249. ^ Couppié et al. 2019 , pp. 77–78. ^ Simpson et al. 2019 , pp. e918. ^ a b Bolz & Ruf 2019 , p. 159. ^ Bolz & Ruf 2019 , pp. 160, 168–173. ^ a b Tabah et al. 2019 , p. 44. ^ Nichter 2019 , p. 258. ^ Nichter 2019 , pp. 256–258. ^ Meyers 2007 , p. 1. ^ a b c d Röltgen & Pluschke 2019 , pp. 1–2. ^ O'Brien et al. 2014 , p. 267. ^ Johnson 2019 , p. 64. ^ Igo & Murthy 1988 , p. 391. ^ Zingue et al. 2018 , pp. 4–8. ^ van der Werf et al. 2005 , p. 2. ^ "Mengo Hospital medical notes – 1897" . ... Retrieved 14 June 2020 . ^ Hotez et al. 2020 , pp. 1–3. ^ a b c Yotsu, Richardson & Ishii 2018 , pp. 6–7. ^ Tabah et al. 2019 , p. 50. ^ a b Bolz & Ruf 2019 , pp. 160–161. ^ Bolz & Ruf 2019 , pp. 163–165. ^ Bolz & Ruf 2019 , p. 165. ^ a b Bolz & Ruf 2019 , pp. 166–167. ^ a b Bolz & Ruf 2019 , p. 163. ^ a b Bolz & Ruf 2019 , pp. 162–163. ... Buruli ulcer: Mycobacterium ulcerans disease . Cham, Switzerland: Springer. pp. 77–85. doi : 10.1007/978-3-030-11114-4_4 . ... Buruli ulcer: Mycobacterium ulcerans disease . Cham, Switzerland: Springer. pp. 1–41. doi : 10.1007/978-3-030-11114-4_1 .

SLC11A1, IFNG, ARAF, CD40LG, HPD, PRKN, TNF, CCN6, BCL2L11, AGO2, SEC61A1, VSX1, PERCC1
Cyclotropia Wikipedia

Indian Journal of Ophthalmology . 56 (1). pp. 39–43. doi : 10.4103/0301-4738.37594 . ... J Pediatr Ophthalmol Strabismus . 21 (4). pp. 126–132. PMID 6470908 . ^ a b c S. ... Postgraduate Ophthalmology . JP Medical Ltd. pp. 1965 ff. ISBN 978-93-5025-270-3 . ... Documenta Ophthalmologica . 58 (1). pp. 131–139. doi : 10.1007/bf00140911 . ^ a b Phyllis E. ... J Pediatr Ophthalmol Strabismus . 21 (4). pp. 126–132. PMID 6470908 . As cited by: S.
Menopause, Natural, Age At, Quantitative Trait Locus 1 OMIM

Gynecologic information was obtained, and if women reported surgical menopause, validation of type and indication of surgery was accomplished by checking medical records. The ESR1 (133430) genotypes PP, Pp, and pp representing the anonymous intronic PvuII RFLP (133430.0004) were assessed by PCR using the PvuII endonuclease. Compared with women carrying the pp genotype, homozygous PP women had a 1.1 year (P less than 0.02) earlier onset of menopause. ... The risk of surgical menopause was 2.4 (95% confidence interval, 1.5 to 3.8) times higher for women carrying the PP genotype compared to those in the pp group, with the most prominent effect in women who underwent hysterectomy due to fibroids or menorrhagia.
Irf6-Related Disorders GARD

Van der Woude syndrome (VWS) is at the mild end of the spectrum and popliteal pterygium syndrome (PPS) is at the severe end of the spectrum. ... People with VWS can have lip pits alone, cleft lip or cleft palate alone, or a combination of these anomalies. People with PPS have a thick web of skin (pterygium) on the backs of both legs (popliteal), extending from the hip (ischial tuberosity) to the heel (calcaneus) or in other locations, as well as other problems. A cone-shaped fold of skin on the nail of the big toe is a very distinctive finding in PPS. Supportive/symptomatic treatment may include surgery, pediatric dentistry, orthodontia, speech therapy, feeding and hearing evaluation, physical therapy, and orthopedic care.
Micrographia (Handwriting) Wikipedia

ISBN 978-0-12-374105-9 . ^ O'Sullivan , pp. 1339 harvnb error: no target: CITEREFO'Sullivan ( help ) ^ Kinematic analysis of handwriting movements in patients with obsessive-compulsive disorder ^ a b O'Sullivan , pp. 858 harvnb error: no target: CITEREFO'Sullivan ( help ) ^ O'Sullivan , pp. 857 harvnb error: no target: CITEREFO'Sullivan ( help ) ^ Ziliotto A, Cersosimo MG, Micheli FE (2015). ... Diagnostic Principles and Applications: Avoiding Medical Errors, Passing Board Exams, and Providing Informed Patient Care . Springer. pp. 57–. ISBN 978-1-4614-1111-6 . ^ Paul Tuite; Hubert Fernandez; Cathi Thomas; Laura Ruekert (23 March 2009). ... Philadelphia, PA: F.A. Davis Company. pp. 857–1339.
Spot Blotch (Barley) Wikipedia

American Phytopathological Society. pp. 120 pp. ISBN 0-89054-180-9 . ^ Martens, J.W.; W.L. ... Canadian Phytopathological Society. pp. 160 pp. ISBN 0-9691627-0-7 .
Paroniria Wikipedia

Please introduce links to this page from related articles ; try the Find link tool for suggestions. ( August 2016 ) Paroniria Other names Nightmares, night terrors, excess dreams Specialty Psychiatry Paroniria is a medical condition involving an excess of morbid dreams and nightmares . [1] [2] [3] Also, Paroniria may be cause by the effects of the antihistamines . [4] Paroniria is suspected to have many causes, including fear, stress, depression, trauma, and sleep deprivation, among others. [5] References [ edit ] ^ Quarterly Journal of Psychological Medicine and Medical Jurisprudence . A. Simpson & Company. 1867. pp. 280–. ^ John Mason GOOD (1820). A physiological system of Nosology; with a corrected and simplified nomenclature . Cox and Son. pp. 293 –. ^ The Medical Press & Circular . 1867. pp. 455–. ^ Mathias B, Schutte M, Hopf G. (31 December 1987).

REM1, CALR, CYP2D6, UNG
- Nightmare Disorder Mayo Clinic
  
  Overview A nightmare is a disturbing dream associated with negative feelings, such as anxiety or fear that awakens you. Nightmares are common in children but can happen at any age. Occasional nightmares usually are nothing to worry about. Nightmares may begin in children between 3 and 6 years old and tend to decrease after the age of 10. During the teen and young adult years, girls appear to have nightmares more often than boys do. Some people have them as adults or throughout their lives. Although nightmares are common, nightmare disorder is relatively rare.
- Nightmare Disorder Wikipedia
  
  Nightmare disorder The Nightmare , by Johann Heinrich Füssli Specialty Psychiatry Frequency c. 4% [1] Nightmare disorder , also known as dream anxiety disorder , is a sleep disorder characterized by frequent nightmares . The nightmares, which often portray the individual in a situation that jeopardizes their life or personal safety, usually occur during the REM stages of sleep. Though most people have experienced at least one nightmare during their life, subjects with nightmare disorder experience them with a greater frequency. The disorder's DSM-IV number is 307.47. Nightmare disorders are included in the parasomnias, which cover all the unusual behaviours during sleep. [2] Nightmare disorders can be confused with sleep terror disorders. [3] The difference is that after a sleep terror episode , the patient wakes up with more dramatic symptom than for a nightmare disorder, such as screaming and crying. [3] Furthermore, they don't remember the reason of the fear, while a patient with a nightmare disorder remembers every detail of the dream. [3] Finally, the sleep terrors usually occur during NREM Sleep. [4] [5] Nightmares also have to be distinguished from bad dreams, which are less emotionally intense. [6] Furthermore, nightmares contain more stories of aggression than bad dreams and more unhappy endings. [6] Finally, people experiencing nightmares feel more fear than for bad dreams. [6] The treatment depends whether or not there is a comorbid PTSD diagnosis. [1] About 4% of American adults are affected. [1] Contents 1 Signs and symptoms 1.1 Consequences 1.2 Content of idiopathic nightmares 2 Criteria 3 Causes 4 Assessment 5 Comorbidity 6 Treatment 6.1 Pharmacological treatments 7 Epidemiology 8 Research 9 References 10 External links Signs and symptoms [ edit ] A boy having a nightmare During the nightmare, the sleeper may scream and yell out things. The nightmare sufferer is often awakened by these threatening, frightening dreams and can often vividly remember their experience.
Muselmann Wikipedia

The Oxford Handbook of Oral History . Oxford University Press, USA. pp. 245 ff. ISBN 978-0-19-533955-0 . ^ Alan Rosen (18 October 2010). ... The Holocaust and History: The Known, the Unknown, the Disputed, and the Reexamined . Indiana University Press. pp. 332–. ISBN 0-253-21529-3 . ^ David Nicholls (2000). Adolf Hitler: A Biographical Companion . ABC-CLIO. pp. 34–. ISBN 978-0-87436-965-6 . ^ Henry Friedlander (9 November 2000). ... : Explaining the Holocaust . W. W. Norton. pp. 86–. ISBN 978-0-393-25437-2 . ^ Michael Burleigh; Wolfgang Wippermann (7 November 1991). The Racial State: Germany 1933-1945 . Cambridge University Press. pp. 161 –. ISBN 978-0-521-39802-2 .
Eye Injuries During General Anaesthesia Wikipedia

. ^ Kocaturk, O, Kocaturk, T, Kaan, N & Dayanir V 2012 'The Comparison of Four Different Methods of Perioperative Eye Protection under General Anesthesia in Prone Position', Journal of Clinical and Analytical Medicine , vol. 3, no. 2, pp. 163-5 "Read Article" ^ a b c d S Contractor & JG Hardman 2006, 'Injury During Anaesthesia', Continuing Education in Anaesthesia, Critical Care & Pain , vol. 6, no. 2, pp. 67-70, "Read Article" ^ a b VK Grover, KV Kumar, S Sharma & SP Grewal 1998, 'Comparison of Methods of Eye Protection under General Anaesthesia', Canadian Journal of Anesthesia , vol. 45, no. 6, pp. 575-7, "Read Article" ^ J Anson, 'Perioperative Corneal Abrasions: Etiology, Prevention, and Management', Pennsylvania Society of Anesthesiologists , "Read Article" . ^ a b c PN Nair & E White 2014, 'Care of the Eye During Anaesthesia and Intensive Care', Anaesthesia and Intensive Care Medicine , vol. 15, no. 1, pp. 40-43, "Read Article" . ^ a b E White 2004, 'Care of the eye during anaesthesia', Anaesthesia and Intensive Care Medicine , vol. 5, pp. 302-3 "Read Article" . ^ K Zheng, CG Guta, V Kulkarni & J Brock-Utne 2009, 'Prevention of Corneal Abrasions in Patients with Autoimmune Dry Eyes', Anaesthesia and Analgesia , vol. 108, no. 1, pp. 385-6, "Read Article" ^ a b A Grixti, M Sadri & MT Watts 2013, 'Corneal Protection during General Anesthesia for Nonocular Surgery', The Ocular Surface , vol. 11, no. 2, pp. 109-18, "Read Article" ^ NJ Friedman & PK Kaiser 2007, Essentials of Ophthalmology , Elsevier Health Sciences, Philadelphia PA USA. ^ N Tarmey & LA White 2009, 'Section 5: Damage to the eye during General Anaesthesia', Risk Associated with your Anaesthetic , Royal College of Anaesthetists website, "Read Article" . ^ AN Gent 1996, 'Adhesion and strength of viscoelastic solids. Is there a relationship between adhesion and bulk properties?', Langmuir , vol. 12, no. 19, pp. 4492-96, "Read Article" . ^ Clinical and Laboratory Standards Institute 2010, 'Performance Standards for Antimicrobial Susceptibility testing: Twentieth Informational Supplement', CLSI Document M100-S20 "Read Article" . ^ C Konya, H Sanada, J Sugama, M Okuwa, Y Kamatani, G Nakagami & K Sakaki 2010, 'Skin Injuries Caused by Medical Adhesive Tape in Older People and Associated Factors', Journal of Clinical Nursing , vol. 19, no. 9-10, pp. 1236-1242 "Read Article" . ^ a b c M Weed & N Syed 2012, 'Perioperative Corneal Abrasions: Systems-based review and analysis', EyeRounds.org, "Read Article" . ^ a b S Prakash 2013, 'Perioperative Eye Protection under General Anaesthesia', Journal of Anaesthesiology Clinical Pharmacology , vol. 29, no. 1, pp. 138-139 "Read Article" .
Aspermia Wikipedia

Springer Science & Business Media. pp. 54–. ISBN 978-3-662-04491-9 . ^ Wayne J.G. ... Springer Science & Business Media. pp. 34–. ISBN 978-1-62703-179-0 . ^ Carrie Bagatell; William J. ... Springer Science & Business Media. pp. 314–. ISBN 978-1-59259-388-0 . ^ Susan Blackburn (14 April 2014). Maternal, Fetal, & Neonatal Physiology . Elsevier Health Sciences. pp. 39–. ISBN 978-0-323-29296-2 . ^ John J. ... Springer Science & Business Media. pp. 3–. ISBN 978-1-59259-098-8 . External links [ edit ] Classification D ICD - 10 : N46 ICD - 9-CM : 606.0 MeSH : D053714 SNOMED CT : 448921000 v t e Male diseases of the pelvis and genitals Internal Testicular Orchitis Hydrocele testis Testicular cancer Testicular torsion Male infertility Aspermia Asthenozoospermia Azoospermia Hyperspermia Hypospermia Oligospermia Necrospermia Teratospermia Epididymis Epididymitis Spermatocele Hematocele Prostate Prostatitis Acute prostatitis Chronic bacterial prostatitis Chronic prostatitis/chronic pelvic pain syndrome Asymptomatic inflammatory prostatitis Benign prostatic hyperplasia Prostate cancer Seminal vesicle Seminal vesiculitis External Penis Balanoposthitis / Balanitis Balanitis plasmacellularis Pseudoepitheliomatous keratotic and micaceous balanitis Phimosis Paraphimosis Priapism Sexual dysfunction Erectile dysfunction Peyronie's disease Penile cancer Penile fracture Balanitis xerotica obliterans Other Hematospermia Retrograde ejaculation Postorgasmic illness syndrome This medical symptom article is a stub .
Sinbyeong Wikipedia

The shinbyeong is dissociated from reality and enters a higher form of consciousness . [6] See also [ edit ] Mu (shaman) Muism References [ edit ] ^ Kim 1998 , pp. 41–42 ^ Kim 1998 , pp. 42–43 ^ Kim 1998 , pp. 43–44 ^ Kim 1998 , pp. 48–49 ^ American Psychiatric Association (2000). ... Washington, D.C. DSM–IV. ^ Kim 1998 , pp. 52–53 Bibliography [ edit ] Kim, Tae-kon (1998).