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  • Aspiration Pneumonia Wikipedia
    New York: McGraw-Hill Medical. ISBN 978-0-07-147748-2 . ^ a b c d Hay WW, Levin MJ, Deterding RR, Abzug MJ (2016-05-02). ... Principles and practice of hospital medicine (Second ed.). New York. ISBN 978-0-07-184313-3 . OCLC 950203123 . ^ a b c d Kasper DL, Fauci AS, Hauser SL, Longo DL, Jameson JL, Loscalzo J (2015-04-08). Harrison's principles of internal medicine (19th ed.). New York. ISBN 978-0-07-180215-4 . OCLC 893557976 . ^ Mukhopadhyay S, Katzenstein AL (May 2007). ... Fishman's pulmonary diseases and disorders (Fifth ed.). New York. ISBN 978-0-07-180728-9 . OCLC 904408460 . ^ a b c d e f Tada A, Miura H (July 2012). ... Geriatrics (Second ed.). New York. ISBN 978-0-07-179208-0 . OCLC 885407197 . ^ a b c d e Nason KS (August 2015).
    NOS2, EDN1, IL1A, CXCL1, CXCL2, TNF, TIMM8A, PANK2, GRHL3, NADK2, UBB, CHAT, TBC1D24, SAMD9, AFF4, KMT2B, LZTR1, PMM2, TAF1, TAC1, NLRP3, ALB, CAT, MYH2, MECP2, KDR, IL1B, ESD, DPP4, ACE, CRP, PLF
    • Mendelson's Syndrome Wikipedia
      Chemical pneumonitis or aspiration pneumonitis caused by aspiration during anaesthesia Mendelson's syndrome Specialty Respirology Mendelson's syndrome is chemical pneumonitis or aspiration pneumonitis caused by aspiration during anaesthesia , especially during pregnancy . Aspiration contents may include gastric juice, blood, bile, water or an association of them. [1] Contents 1 Presentation 2 Risk factors 3 Eponym 4 References 5 External links Presentation [ edit ] Mendelson's syndrome is characterised by a bronchopulmonary reaction following aspiration of gastric contents during general anaesthesia due to abolition of the laryngeal reflexes. The main clinical features are signs of general hypoxia , two to five hours after anaesthesia. Such features may include cyanosis, dyspnea, fever, pulmonary wheeze, crepitant rales, rhonchi, and tachycardia with a low blood pressure. Decreased arterial oxygen tension is also likely to be evident. Pulmonary edema can cause sudden death or death may occur later from pulmonary complications. [ citation needed ] Risk factors [ edit ] Historically it is said that a patient is at risk if they have: [2] Residual gastric volume of greater than 25ml, with pH of less than 2.5 However these are indirect measurements and are not factors that directly influence aspiration risk. [2] Patients with a high risk should have a rapid sequence induction .
  • Female Foeticide In India Wikipedia
    According to the decennial Indian census, the sex ratio in 0 to 6 age group in India has risen from 102.4 males per 100 females in 1961, [1] to 104.2 in 1980, to 107.5 in 2001, to 108.9 in 2011. [2] The child sex ratio is within the normal natural range in all eastern and southern states of India, [3] but significantly higher in certain western and particularly northwestern states such as Maharashtra , Haryana , Jammu & Kashmir (118, 120 and 116, as of 2011, respectively). [4] The western states of Maharashtra and Rajasthan 2011 census found a child sex ratio of 113, Gujarat at 112 and Uttar Pradesh at 111. [5] The Indian census data indicates that the sex ratio is poor when women have one or two children, but gets better as they have more children, which is result of sex-selective "stopping practices" (stopping having children based on sex of those born). [6] The Indian census data also suggests there is a positive correlation between abnormal sex ratio and better socio-economic status and literacy. ... Consequences of a declining sex ratio in Indian states [ edit ] 2011 Census sex ratio map for the states and Union Territories of India, boys per 100 girls in 0 to 1 age group. [35] This table gives information on the child sex ratio in major states in India throughout the years 1981, 1991, and 2001 [36] The following table presents the child sex ratio data for India's states and union territories, according to 2011 Census of India for population count in the 0-1 age group. [37] The data suggests 18 states/UT had birth sex ratio higher than 107 implying excess males at birth and/or excess female mortalities after birth but before she reaches the age of 1, 13 states/UT had normal child sex ratios in the 0-1 age group, and 4 states/UT had birth sex ratio less than 103 implying excess females at birth and/or excess male mortalities after birth but before he reaches the age of 1. State / UT Boys (0-1 age) 2011 Census [37] Girls (0-1 age) 2011 Census [37] Sex ratio (Boys per 100 girls) India 10,633,298 9,677,936 109.9 Jammu and Kashmir 154,761 120,551 128.4 Haryana 254,326 212,408 119.7 Punjab 226,929 193,021 117.6 Uttarakhand 92,117 80,649 114.2 DELHI 135,801 118,896 114.2 Maharashtra 946,095 829,465 114.1 Lakashadweep 593 522 114.0 Rajasthan 722,108 635,198 113.7 Gujarat 510,124 450,743 113.2 Uttar Pradesh 1,844,947 1,655,612 111.4 Chandigarh 8,283 7,449 111.2 Daman and Diu 1,675 1,508 111.1 Bihar 1,057,050 957,907 110.3 Himchal Pradesh 53,261 48,574 109.6 Madhya Pradesh 733,148 677,139 108.3 Goa 9,868 9,171 107.6 Jharkhand 323,923 301,266 107.5 Manipur 22,852 21,326 107.2 Andhra Pradesh 626,538 588,309 106.5 Tamil Nadu 518,251 486,720 106.5 Odisha 345,960 324,949 106.5 Dadra and Nagar Haveli 3,181 3,013 105.6 Karnataka 478,346 455,299 105.1 West Bengal 658,033 624,760 105.0 Assam 280,888 267,962 104.8 Nagaland 17,103 16,361 104.5 Sikkim 3,905 3,744 104.3 Chhattisgarh 253,745 244,497 103.8 Tripura 28,650 27,625 103.7 Meghalaya 41,353 39,940 103.5 Arunachal Pradesh 11,799 11,430 103.2 Andaman and Nicobar Islands 2,727 2,651 102.9 Kerala 243,852 238,489 102.2 Puducherry 9,089 8,900 102.1 Mizoram 12,017 11,882 Marriage Market and Importation of Brides [ edit ] Classic economic theory views the market for marriage as one in which people bargain for a spouse who maximizes their utility gains from marriage. [38] In India, many of these bargains actually take place within the family and therefore individual utility is replaced by family utility. ... MRf is female child mortality, a is residual female mortality when male mortality is 0, the slope b shows the rate of decline in female mortality for a decline in male mortality, and MRm is male mortality. ... New York: McGraw-Hill. pp. 200. ISBN 0-07-252183-X ^ Johansson, Sten; Nygren, Olga (1991).
  • Hypoxia (Medical) Wikipedia
    New York: Springer. pp. 391–94. doi : 10.1007/978-0-387-75246-4_97 . ISBN 978-0387752457 . ^ Das, K. ... Williams & Wilkins. p. 22. ISBN 978-0-683-08936-3 . ^ Cymerman, A; Rock, PB. ... Philadelphia: Lippincott Williams & Wilkins. ISBN 978-0-683-30604-0 . ^ a b c Nicki R. Colledge; Brian R. ... Edinburgh: Churchill Livingstone/Elsevier. ISBN 978-0-7020-3085-7 . ^ Marvin E. Levin; Lawrence W. ... The Diabetic foot . Mosby Year Book. ISBN 978-0-8016-6878-4 . ^ West, John B. (2008).
    ACADVL, ESD, GAPDH, TFPI, SOD2, GNAS, ACTB, RPS14, MPO, NME1, NOS1, NOS3, NPPA, RPS2, PCK2, PDYN, PLAT, TKT, POMC, CA9, APOA1, ADK, SARDH, CTRB1, AHCY, ALDH1L1, CS, ALDOB, CPA2, EGR1, BDNF, ATP5F1B, ATF4, RET, ZFPM2, SCN8A, SLC35A1, PHOX2B, DNAH11, H3-3A, OAS1, MECP2, HBB, GDNF, GATA6, FBN1, ENG, EDN3, CSF2RB, CSF2RA, ASCL1, MAGEL2
  • Wilderness-Acquired Diarrhea Wikipedia
    Berlin: Springer-Verlag. pp. 380–99. ISBN 0-387-97162-9 . ^ White, George W. (1992). ... New York: Van Nostrand Reinhold. ISBN 0-442-00693-4 . ^ ( Backer 2007 , p. 1374) ^ a b c Boulware DR, Forgey WW, Martin WJ 2nd (2003). ... International Marine/Ragged Mountain Press. p. 46. ISBN 0-07-139303-X . ^ Backer H (February 2002). ... "High Sierra Water: What is in the H 2 0?" . Yosemite Association. ^ Rose JB, Haas CN, Regli S (1991). ... Philadelphia, PA: Mosby Elsevier. pp. 1368–417. ISBN 978-0-323-03228-5 . External links [ edit ] "Sources of Infection & Risk Factors| Giardia | Parasites | CDC" . www.cdc.gov .
  • Epidermolysis Bullosa Wikipedia
    Nature Reviews Disease Primers . 6 (1): 78. doi : 10.1038/s41572-020-0210-0 . ISSN 2056-676X . PMID 32973163 . ... Nature Reviews Disease Primers . 6 (1): 78. doi : 10.1038/s41572-020-0210-0 . ISSN 2056-676X . PMID 32973163 . ... Andrews' Diseases of the Skin: Clinical Dermatology . (10th ed.). Saunders. ISBN 0-7216-2921-0 . ^ a b Freedberg, et al. (2003). Fitzpatrick's Dermatology in General Medicine . (6th ed.). McGraw-Hill. ISBN 0-07-138076-0 . ^ "Junctional epidermolysis bullosa" .
    ITGB4, KRT14, KRT5, EXPH5, COL7A1, COL17A1, PLEC, LAMC2, LAMB3, DST, ITGA6, ITGA3, ACTB, LAMA3, CCL21, SDC2, TMSB4X, CXCL12, TGM3, TP53, BEST1, PLOD3, TGM5, CD160, B3GAT1, SLCO1B3, KLHL24, FERMT1, RPS27A, LAD1, MMP1, FLII, CEACAM5, DMD, DSC2, DSC3, DSP, EBM, GPT, LDLR, HMGB1, HSPG2, IL1A, IL1B, IL6, KRT18, ERBIN
    • Epidermolysis Bullosa Mayo Clinic
      Overview Epidermolysis bullosa (ep-ih-dur-MOL-uh-sis buhl-LOE-sah) is a rare condition that causes fragile, blistering skin. The blisters may appear in response to minor injury, even from heat, rubbing or scratching. In severe cases, the blisters may occur inside the body, such as the lining of the mouth or stomach. Epidermolysis bullosa is inherited, and it usually shows up in infants or young children. Some people don't develop symptoms until they're teens or young adults.
    • Epidermolysis Bullosa GARD
      Epidermolysis bullosa (EB) is a group of genetic skin diseases that cause the skin to blister and erode very easily. In people with EB, blisters form in response to minor injuries or friction, such as rubbing or scratching.[2310] There are four main types of EB, which are classified based on the depth, or level, of blister formation: Epidermolysis bullosa simplex Dystrophic epidermolysis bullosa Junctional epidermolysis bullosa Kindler Syndrome EB may then be further classified based on severity and specific symptoms, such as distribution (localized or generalized) and whether parts of the body other than the skin are affected. Specific sub-types may then be determined based on identifying the exact protein that is defective in a person with EB. This may be done by tests performed on a skin biopsy, or when possible, genetic testing. Identifying the exact sub-type can be hard because there are many sub-types of EB.
  • Factitious Disorder Imposed On Self Wikipedia
    Hoboken, New Jersey: John Wiley & Sons, Ltd. p. 680. ISBN 978-0-470-01854-5 . ^ Huffman, Jeffrey C.; Stern, Theodore A. (2003). ... Mississauga: John Wiley & Sons Canada. p. 412. ISBN 978-0-470-84072-6 . ^ Giannini, A. James; Black, Henry Richard; Goettsche, Roger L. (1978). ... New Hyde Park, NY: Medical Examination Publishing. pp. 194–5. ISBN 978-0-87488-596-5 . ^ McCoy, Monica L.; Keen, Stefanie M. (2013). ... Mississauga: John Wiley & Sons Canada, Ltd. p. 412. ISBN 978-0-470-84072-6 . Prasad, A.; Oswald, A. ... How numbers get used and abused in the courtroom . Basic Books. ISBN 978-0-465-03292-1 . Staff, Mayo Clinic (13 May 2011).
  • Iron Deficiency Wikipedia
    Why We Get Sick: The New Science of Darwinian Medicine . New York. page 30 ISBN 0-679-74674-9 . ^ T. William Hutchens, Bo Lönnerdal; Lactoferrin: Interactions and Biological Functions (1997). page 379 on Google Books ^ Nesse, R. ... Why We Get Sick: The New Science of Darwinian Medicine . New York. page 29 ISBN 0-679-74674-9 . ^ Weinberg, E. D. (1984). ... Oxford University Press . pp. 626–628. ISBN 0-19-852558-3 . ^ Rockey D, Cello J (1993). ... Guidelines for Patient Blood Management and Blood . ISBN 978-1-56395-333-0 . Archived from the original on 15 October 2014 . ... Belmont, California: Wadsworth, Cengage Learning . ISBN 978-0-495-11657-8 . Umbreit, Jay (2005).
    EPO, TMPRSS6, SLC11A2, HFE, TFRC, HAMP, CRP, FGF23, HBA2, CHMP2B, DMRT1, HIF1A, IL1B, SLC40A1, EPAS1, HEPH, PMCH, BDNF, HBA1, CP, AIF1, RET, IREB2, IL6, SLC39A1, ATF4, PARP3, RN7SL263P, HMOX1, TNF, ALB, IDUA, FECH, LCN2, ACAD8, FXN, ERFE, BDH2, CYBRD1, RLS1, TFR2, TDGF1P3, ACO1, RPP14, ZFP36, SUB1, PPARGC1A, AHSA1, AIMP2, KHSRP, STK16, AKR1A1, CDKL1, BCAP31, CDK2AP2, GRAP2, DNALI1, HDAC3, EIF2S2, SLC39A7, NOL3, GOSR1, XPR1, MLLT10, PSIP1, GSTO1, ABCG2, ABO, SETD2, SIRT2, SLC36A1, ZGPAT, COX19, SLC46A1, CYGB, OLIG1, HJV, IL27, XYLT2, COPD, SLC39A5, CYCSP51, NUP43, H3P8, H3P24, HHIP, NLN, JMJD6, UBE2D1, CRTC1, CDK20, RNF19A, POLDIP2, SIGLEC7, LAMTOR2, TMED5, SLC39A10, SLC25A37, NCKIPSD, SF3B6, NANS, AHI1, SMARCAD1, VHL, SST, TYS, CUX1, DNAH8, EBF1, EDN1, EIF2S1, EIF2S3, ESR1, ETV3, G6PD, GATA1, GPI, HCLS1, HP, PRMT1, IFNG, IGF1, DAPK3, CTSL, TTR, MAPK14, ACP5, APEX1, ARG1, ARSA, ATP4A, BACH1, BSG, CAT, CCK, CDKN2A, CENPF, CLN3, SLC31A1, CRK, CRYGD, IL1A, IL2RB, CXCL8, SMAD1, RPL29, S100A9, TSPAN31, SAT1, SLC2A1, SLC11A1, SOD1, ST13, STAT3, STAT5A, STAT5B, CNTN2, TCF3, TNFRSF1B, TST, REN, PRPH2, PVALB, PDE7A, SMAD7, MMP9, MPO, NGF, NUCB2, REG3A, CFP, PTH, PPARG, MAPK1, PRNP, RELN, PSMC6, PSMD10, H3P30
  • Acrophobia Wikipedia
    Research is underway at several clinics. [18] Recent studies found that participants experienced increased anxiety not only during elevation in height, but also when they were required to move sideways in a fixed height. [19] A recombinant model of the development of acrophobia is very possible, in which learning factors, cognitive factors (e.g. interpretations), perceptual factors(e.g. visual dependence), and biological factors (e.g. heredity) interact to provoke fear or habituation. [5] Assessment [ edit ] ICD-10 and DSM-V are used to diagnose acrophobia. [20] Acrophobia Questionnaire (AQ) is a self report that contains 40 items, assessing anxiety level on a 0-6 point scale and degree of avoidance on a 0-2 point scale. [21] [22] The Attitude Towards Heights Questionnaires (ATHQ) [23] and Behavioural Avoidance Tests (BAT) are also used. [5] However, acrophobic individuals tend to have biases in self report. ... Experimental Brain Research . 150 (2): 208–221. doi : 10.1007/s00221-003-1446-0 . ISSN 0014-4819 . PMID 12677318 . ... Behavior Therapy . 8 (1): 17–23. doi : 10.1016/S0005-7894(77)80116-0 . ISSN 0005-7894 . ^ a b c Steinman, Shari A.; Teachman, Bethany A. (2011-10-01). ... Behaviour Research and Therapy . 27 (5): 561–567. doi : 10.1016/0005-7967(89)90091-0 . hdl : 2027.42/28207 . ISSN 0005-7967 .
    OPN1SW, TNC, MAOA
  • Palmer Raids Wikipedia
    . ^ a b c d e f Avrich, Paul, Sacco and Vanzetti: The Anarchist Background , Princeton University Press, ISBN 0-691-02604-1 (1991), pp. 140–143, 147, 149–156 ^ "Plotter Here Hid Trail Skillfully; His Victim Was A Night Watchman", The New York Times, 4 June 1919 ^ "Wreck Judge Nott's Home", The New York Times, 3 June 1919 ^ Hagedorn, 229–30; Coben, 211 ^ Pietruszka, 146–7 ^ Coben 217–8 ^ Coben, 207–9 ^ Coben, 214–5 ^ Coben, 219–21; Post, 28–35. ... Post, 91–2, 96, 104–5, 108, 110, 115–6, 120–1, 124, 126, 131 ^ Post, 96–147, passim ^ Post, 91–5, 96–147 ^ Coben, 230; The New York Times : "Palmer Upholds Red Repression," January 24, 1920 , accessed January 15, 2010, ^ The Washington Post , "The Red Assassins," January 4, 1920 ^ a b Coben, 232 ^ Avakov, Aleksandr Vladimirovich, Plato's Dreams Realized: Surveillance and Citizen Rights from KGB to FBI , Algora Publishing, ISBN 0-87586-495-3 , ISBN 978-0-87586-495-2 (2007), p. 36 ^ Daniels, 545–6 ^ Post, 273 ^ https://www.aclu.org/about/aclu-history ^ Report Upon the Illegal Practices of the United States Department of Justice . ... Finan, Christopher M., From the Palmer Raids to the Patriot Act: A history of the fight for free speech in America (Boston: Beacon Press, 2007) Hagedorn, Ann, Savage Peace: Hope and Fear in America, 1919 (New York: Simon & Schuster, 2007) Kennedy, David M., Over Here: The First World War and American Society (New York: Oxford University Press, 1980) Murray, Robert K., Red Scare: A Study in National Hysteria, 1919–1920 (Minneapolis: University of Minnesota Press, 1955) Pietrusza, David, 1920: The Year of Six presidents (New York: Carroll & Graf, 2007) Post, Louis F., The Deportations Delirium of Nineteen-twenty: A Personal Narrative of a Historic Official Experience (New York, 1923), reissued: ISBN 0-306-71882-0 , ISBN 1-4102-0553-3 External links [ edit ] Media related to Palmer Raids at Wikimedia Commons
  • Addictive Personality Wikipedia
    Willpower's Not Enough: Understanding and Recovering From Addictions of Every Kind . HarperCollins. pp. 59– . ISBN 978-0-06-015996-2 . [ page needed ] ^ Cox, W. ... Determinants of Substance Abuse: Biological, Psychological, and Environmental Factors . Springer. pp. 209–46 . ISBN 978-0-306-41873-0 . ^ Hurley, Katie. "Depression and Related Conditions" . ... Lexington Books. pp. 157–236 . ISBN 978-0-669-06293-9 . Lay summary – The New York Times (January 18, 1983). ^ a b c d Takao, Motoharu; Takahashi, Susumu; Kitamura, Masayoshi (2009). ... Personality and Individual Differences . 32 (7): 1185–1197. doi : 10.1016/s0191-8869(01)00080-0 . ^ a b Müller, K. W.; Koch, A.; Dickenhorst, U.; Beutel, M.
  • Bovine Malignant Catarrhal Fever Wikipedia
    John Wiley & Sons. pp. 157–. ISBN 978-0-470-34481-1 . ^ Lee Merriam Talbot; Martha H. ... Veterinary Microbiology . 42 (1): 45–52. doi : 10.1016/0378-1135(94)90076-0 . PMID 7839584 . ^ a b Berezowski, John Andrew; Appleyard, Greg D.; Crawford, Timothy B.; Haigh, Jerry; Li, Hong; Middleton, Dorothy M.; O'Connor, Brendan P.; West, Keith; Woodbury, Murray (2005). ... New York: Oxford University Press. ISBN 978-0-19-570506-5 . ^ Homewood, K. H.; Rodgers, W. ... Veterinary Virology (2nd ed.). Academic Press. ISBN 978-0-12-253056-2 . ^ O'Toole, D.; Taus, N. ... Journal of General Virology . 88 (Pt 1): 40–45. doi : 10.1099/vir.0.82285-0 . PMID 17170434 . ^ a b "Malignant Catarrhal fever" (PDF) .
  • Feline Hyperesthesia Syndrome Wikipedia
    Obsessive-compulsive disorders: diagnosis, etiology, treatment . Informa Health Care. p. 121. ISBN 0-8247-9856-2 . ^ a b Pinney CC (2003). ... McGraw-Hill Professional. pp. 351–352. ISBN 0-07-141272-7 . ^ a b Dewey CW (2003), A practical guide to canine and feline neurology , Wiley-Blackwell, p. 442, ISBN 0-8138-1249-6 ^ Rand J (2006). ... Elsevier Health Sciences. p. 1016. ISBN 0-7020-2488-0 . External links [ edit ] World Small Animal Veterinary Association – World Congress, Vancouver 2001.
  • Croup Wikipedia
    The most common bacteria implicated are Staphylococcus aureus , Streptococcus pneumoniae , Haemophilus influenzae , and Moraxella catarrhalis . [5] Pathophysiology [ edit ] The viral infection that causes croup leads to swelling of the larynx , trachea , and large bronchi [4] due to infiltration of white blood cells (especially histiocytes , lymphocytes , plasma cells , and neutrophils ). [5] Swelling produces airway obstruction which, when significant, leads to dramatically increased work of breathing and the characteristic turbulent, noisy airflow known as stridor . [4] Diagnosis [ edit ] Westley score: Classification of croup severity [3] [13] Feature Number of points assigned for this feature 0 1 2 3 4 5 Chest wall retraction None Mild Moderate Severe Stridor None With agitation At rest Cyanosis None With agitation At rest Level of consciousness Normal Disoriented Air entry Normal Decreased Markedly decreased Croup is typically diagnosed based on signs and symptoms. [4] The first step is to exclude other obstructive conditions of the upper airway, especially epiglottitis , an airway foreign body , subglottic stenosis , angioedema , retropharyngeal abscess , and bacterial tracheitis . [4] [5] A frontal X-ray of the neck is not routinely performed, [4] but if it is done, it may show a characteristic narrowing of the trachea, called the steeple sign , because of the subglottic stenosis, which resembles a steeple in shape. ... It is primarily used for research purposes rather than in clinical practice. [5] It is the sum of points assigned for five factors: level of consciousness, cyanosis, stridor, air entry, and retractions. [5] The points given for each factor is listed in the adjacent table, and the final score ranges from 0 to 17. [13] A total score of ≤ 2 indicates mild croup. ... Missoula, Mont.: Mountain Press. p. 324 . ISBN 978-0-87842-505-1 . ^ a b c Feigin, Ralph D. (2004). ... Philadelphia: Saunders. p. 252. ISBN 978-0-7216-9329-3 . ^ a b c d "Diagnosis and Management of Croup" (PDF) . ... Elsevier Health Sciences. 2014. p. 762. ISBN 978-0-323-26373-3 . Archived from the original on 2017-09-08. ^ Vanderpool, Patricia (December 2012).
    THBS1, CD14, FLNB, CXCL8, USO1, NXF1, SEC14L2
    • Croup Mayo Clinic
      Overview Croup refers to an infection of the upper airway, which becomes narrow, making it harder to breathe. Croup also causes a cough that sounds like barking. The cough and other signs and symptoms of croup are the result of swelling and irritation around the voice box (larynx), windpipe (trachea) and bronchial tubes (bronchi). When a cough forces air through this narrowed passageway, the swollen vocal cords produce a noise like a seal barking. Taking a breath often produces a high-pitched whistling sound called stridor. Healthy airway This shows a child's healthy airway. Croup When the upper airway is infected with the virus that causes croup, tissues around the voice box (larynx) and the windpipe (trachea) will swell.
  • Lordosis Wikipedia
    Louis: Mosby, pp. 116–133, doi : 10.1016/b978-0-323-07759-0.00031-6 , ISBN 978-0-323-07759-0 , retrieved 2020-11-03 ^ "Lordosis" . ... New Zealand: Spinal Publications New Zealand, Ltd. ISBN 978-0-9876504-0-5 . ^ "Lordotic Chest Technique" . ^ Solomon, Ruth.
    SGCA, CAPN3, ACP5, SPEG, HERC1, BAZ1B, HACD1, TRIP11, EIF2AK3, GTF2IRD1, CUL7, KLHL41, PPM1D, AGPAT2, COLEC10, POP1, RAB3GAP1, SYNE2, EXOC6B, SYNGAP1, COLQ, SYNE1, TPM2, ACTA1, SCN9A, SGCG, TBX6, TFAP2A, TGFB1, TPM3, DYSF, TRPS1, TTN, TWIST1, VCP, CLIP2, NAA10, BICD2, SMCHD1, RMRP, PLEKHG5, TRAPPC11, MTMR14, WNK1, COLEC11, FKRP, TMEM43, CCDC8, SLC10A7, GNPTG, KLC4, CANT1, B3GALT6, AMER1, CAVIN1, KY, TRPV4, SELENON, OBSL1, LMOD3, RAB3GAP2, BSCL2, TBL2, POMT2, SMARCAL1, NSDHL, INPP5K, MAP3K20, RETREG1, DYM, BCOR, SETD5, POMGNT1, IARS2, ZC4H2, RYR1, SCN4A, RFC2, GLB1, FRG1, FN1, FLNB, FHL1, FGFR2, FGFR3, FKTN, EMD, ELN, TOR1A, DNM2, DYNC1H1, DNA2, DMD, DES, CTSK, CRKL, COMP, COL11A2, COL5A2, COL5A1, COL2A1, COL1A2, COL1A1, CFL2, CAV1, BCR, KIF1A, BIN1, GALNS, FOS, DUX4, IHH, MYL2, MYH7, MMP13, LMNA, LIMK1, LAMA2, L1CAM, NPR2, PCYT1A, ITGA7, IGHMBP2, NOTCH3, PPARG, HSPG2, HNRNPH2, MAPK1, HNRNPA2B1, MASP1, HNRNPA1, NECTIN1, GTF2I, RET, NEB, GPER1, ESR1, ESR2, FTH1, ROM1, CHPT1, ADAMTS1, CRPPA, AMH, LIAS, ARSD, GH1, KISS1R, CD6, POMC, GNRH1, DHDDS, PNOC, PGR, PDE4A, OPRL1, OPRK1, PROK2, CRP, CYP19A1
  • X-Linked Ichthyosis Wikipedia
    . ^ Brcic L, Underwood JFW, Kendall KM et al. (2020) Medical and neurobehavioural phenotypes in carriers of X-linked ichthyosis-associated genetic deletions in the UK Biobank. J. Med. Genet. 0:1-7 PMID:32139392 doi:10.1136/jmedgenet-2019-106676 URL: https://jmg.bmj.com/content/early/2020/03/05/jmedgenet-2019-106676 ^ Chatterjee S, Humby T, Davies W (2016) Behavioural and psychiatric phenotypes in men and boys with X-linked ichthyosis: evidence from a worldwide online survey. ... Fitzpatrick's Dermatology in General Medicine . (6th ed.). McGraw-Hill. ISBN 0-07-138076-0 . ^ James, William; Berger, Timothy; Elston, Dirk (2005). Andrews' Diseases of the Skin: Clinical Dermatology . (10th ed.). Saunders. ISBN 0-7216-2921-0 . External links [ edit ] Classification D ICD - 10 : Q80.1 ICD - 9-CM : 757.1 OMIM : 308100 MeSH : D016114 DiseasesDB : 29136 External resources eMedicine : derm/191 Orphanet : 461 v t e Congenital malformations and deformations of integument / skin disease Genodermatosis Congenital ichthyosis / erythrokeratodermia AD Ichthyosis vulgaris AR Congenital ichthyosiform erythroderma : Epidermolytic hyperkeratosis Lamellar ichthyosis Harlequin-type ichthyosis Netherton syndrome Zunich–Kaye syndrome Sjögren–Larsson syndrome XR X-linked ichthyosis Ungrouped Ichthyosis bullosa of Siemens Ichthyosis follicularis Ichthyosis prematurity syndrome Ichthyosis–sclerosing cholangitis syndrome Nonbullous congenital ichthyosiform erythroderma Ichthyosis linearis circumflexa Ichthyosis hystrix EB and related EBS EBS-K EBS-WC EBS-DM EBS-OG EBS-MD EBS-MP JEB JEB-H Mitis Generalized atrophic JEB-PA DEB DDEB RDEB related: Costello syndrome Kindler syndrome Laryngoonychocutaneous syndrome Skin fragility syndrome Ectodermal dysplasia Naegeli syndrome / Dermatopathia pigmentosa reticularis Hay–Wells syndrome Hypohidrotic ectodermal dysplasia Focal dermal hypoplasia Ellis–van Creveld syndrome Rapp–Hodgkin syndrome / Hay–Wells syndrome Elastic / Connective Ehlers–Danlos syndromes Cutis laxa ( Gerodermia osteodysplastica ) Popliteal pterygium syndrome Pseudoxanthoma elasticum Van der Woude syndrome Hyperkeratosis / keratinopathy PPK diffuse : Diffuse epidermolytic palmoplantar keratoderma Diffuse nonepidermolytic palmoplantar keratoderma Palmoplantar keratoderma of Sybert Meleda disease syndromic connexin Bart–Pumphrey syndrome Clouston's hidrotic ectodermal dysplasia Vohwinkel syndrome Corneodermatoosseous syndrome plakoglobin Naxos syndrome Scleroatrophic syndrome of Huriez Olmsted syndrome Cathepsin C Papillon–Lefèvre syndrome Haim–Munk syndrome Camisa disease focal : Focal palmoplantar keratoderma with oral mucosal hyperkeratosis Focal palmoplantar and gingival keratosis Howel–Evans syndrome Pachyonychia congenita Pachyonychia congenita type I Pachyonychia congenita type II Striate palmoplantar keratoderma Tyrosinemia type II punctate : Acrokeratoelastoidosis of Costa Focal acral hyperkeratosis Keratosis punctata palmaris et plantaris Keratosis punctata of the palmar creases Schöpf–Schulz–Passarge syndrome Porokeratosis plantaris discreta Spiny keratoderma ungrouped: Palmoplantar keratoderma and spastic paraplegia desmoplakin Carvajal syndrome connexin Erythrokeratodermia variabilis HID / KID Other Meleda disease Keratosis pilaris ATP2A2 Darier's disease Dyskeratosis congenita Lelis syndrome Dyskeratosis congenita Keratolytic winter erythema Keratosis follicularis spinulosa decalvans Keratosis linearis with ichthyosis congenita and sclerosing keratoderma syndrome Keratosis pilaris atrophicans faciei Keratosis pilaris Other cadherin EEM syndrome immune system Hereditary lymphedema Mastocytosis / Urticaria pigmentosa Hailey–Hailey see also Template:Congenital malformations and deformations of skin appendages , Template:Phakomatoses , Template:Pigmentation disorders , Template:DNA replication and repair-deficiency disorder Developmental anomalies Midline Dermoid cyst Encephalocele Nasal glioma PHACE association Sinus pericranii Nevus Capillary hemangioma Port-wine stain Nevus flammeus nuchae Other/ungrouped Aplasia cutis congenita Amniotic band syndrome Branchial cyst Cavernous venous malformation Accessory nail of the fifth toe Bronchogenic cyst Congenital cartilaginous rest of the neck Congenital hypertrophy of the lateral fold of the hallux Congenital lip pit Congenital malformations of the dermatoglyphs Congenital preauricular fistula Congenital smooth muscle hamartoma Cystic lymphatic malformation Median raphe cyst Melanotic neuroectodermal tumor of infancy Mongolian spot Nasolacrimal duct cyst Omphalomesenteric duct cyst Poland anomaly Rapidly involuting congenital hemangioma Rosenthal–Kloepfer syndrome Skin dimple Superficial lymphatic malformation Thyroglossal duct cyst Verrucous vascular malformation Birthmark v t e X-linked disorders X-linked recessive Immune Chronic granulomatous disease (CYBB) Wiskott–Aldrich syndrome X-linked severe combined immunodeficiency X-linked agammaglobulinemia Hyper-IgM syndrome type 1 IPEX X-linked lymphoproliferative disease Properdin deficiency Hematologic Haemophilia A Haemophilia B X-linked sideroblastic anemia Endocrine Androgen insensitivity syndrome / Spinal and bulbar muscular atrophy KAL1 Kallmann syndrome X-linked adrenal hypoplasia congenita Metabolic Amino acid : Ornithine transcarbamylase deficiency Oculocerebrorenal syndrome Dyslipidemia : Adrenoleukodystrophy Carbohydrate metabolism : Glucose-6-phosphate dehydrogenase deficiency Pyruvate dehydrogenase deficiency Danon disease/glycogen storage disease Type IIb Lipid storage disorder : Fabry's disease Mucopolysaccharidosis : Hunter syndrome Purine–pyrimidine metabolism : Lesch–Nyhan syndrome Mineral : Menkes disease / Occipital horn syndrome Nervous system X-linked intellectual disability : Coffin–Lowry syndrome MASA syndrome Alpha-thalassemia mental retardation syndrome Siderius X-linked mental retardation syndrome Eye disorders: Color blindness (red and green, but not blue) Ocular albinism ( 1 ) Norrie disease Choroideremia Other: Charcot–Marie–Tooth disease (CMTX2-3) Pelizaeus–Merzbacher disease SMAX2 Skin and related tissue Dyskeratosis congenita Hypohidrotic ectodermal dysplasia (EDA) X-linked ichthyosis X-linked endothelial corneal dystrophy Neuromuscular Becker's muscular dystrophy / Duchenne Centronuclear myopathy (MTM1) Conradi–Hünermann syndrome Emery–Dreifuss muscular dystrophy 1 Urologic Alport syndrome Dent's disease X-linked nephrogenic diabetes insipidus Bone / tooth AMELX Amelogenesis imperfecta No primary system Barth syndrome McLeod syndrome Smith–Fineman–Myers syndrome Simpson–Golabi–Behmel syndrome Mohr–Tranebjærg syndrome Nasodigitoacoustic syndrome X-linked dominant X-linked hypophosphatemia Focal dermal hypoplasia Fragile X syndrome Aicardi syndrome Incontinentia pigmenti Rett syndrome CHILD syndrome Lujan–Fryns syndrome Orofaciodigital syndrome 1 Craniofrontonasal dysplasia v t e Inborn errors of steroid metabolism Mevalonate pathway HMG-CoA lyase deficiency Hyper-IgD syndrome Mevalonate kinase deficiency To cholesterol 7-Dehydrocholesterol path: Hydrops-ectopic calcification-moth-eaten skeletal dysplasia CHILD syndrome Conradi-Hünermann syndrome Lathosterolosis Smith–Lemli–Opitz syndrome desmosterol path: Desmosterolosis Steroids Corticosteroid (including CAH ) aldosterone : Glucocorticoid remediable aldosteronism cortisol / cortisone : CAH 17α-hydroxylase CAH 11β-hydroxylase both: CAH 3β-dehydrogenase CAH 21-hydroxylase Apparent mineralocorticoid excess syndrome/11β-dehydrogenase Sex steroid To androgens 17α-Hydroxylase deficiency 17,20-Lyase deficiency Cytochrome b 5 deficiency 3β-Hydroxysteroid dehydrogenase deficiency 17β-Hydroxysteroid dehydrogenase deficiency 5α-Reductase deficiency Pseudovaginal perineoscrotal hypospadias To estrogens Aromatase deficiency Aromatase excess syndrome Other X-linked ichthyosis Antley–Bixler syndrome
    STS, MDM2, TP53, FLG, EGFR, AKT1, SMUG1, CRP, PDGFRA, CDKN2A, PTEN, VCX3A, TNF, VEGFA, CD274, ANOS1, HIF1A, ABCB1, BRCA1, IGF1, SERPINE1, HEATR3, EWSR1, VIM, PARP1, MSH2, MMP9, RASSF1, MLH1, HSPB1, HSPB2, HSPB3, KDR, KIT, ABCC1, PDCD1, DMD, EGF, PECAM1, CTNNB1, CD44, MUC5B, CHEK1, BCL2, CREG1, LGR5, STAT3, MLRL, PUDP, TNFSF10, TGFB1, STAR, SS18, SSX2, SPP1, TERT, TIMP1, HMGA2, TGM1, THBD, FOSL1, TIMP2, TOP2A, WNT5A, BEST1, VIP, TST, TYMS, EZR, VEGFC, FBP2, RAB40B, CFLAR, MIR215, RTN4R, IL33, PRIMA1, SUMF1, LPP-AS2, MIRLET7I, MIR152, MIR210, MIR34B, MIB1, VCX3B, MIR429, PRB2, SSX2B, MIR1226, PSC, EMSLR, LOC110806263, HIF3A, NLGN4X, PROM1, MAPK8IP2, PCLAF, GAB2, TACC3, HPSE, SHBG, SPINK5, CHEK2, TBC1D9, VCX, DEPDC1B, HPGDS, SETD2, PYCARD, FOXP3, UBASH3A, MIEF1, KRT20, PIWIL2, SMARCB1, PIK3CD, CX3CL1, CSPG4, TIMM8A, ELANE, EPAS1, STOM, EPHB2, EPHB4, ERBB2, ERCC1, ERCC2, ERCC5, PTK2B, FGFR1, FMR1, FOS, MTOR, GLB1, GSTA1, GADD45A, CSF2, CFH, COL11A2, ALB, ALK, ANXA5, ARSA, ATF1, BMP2, BRAF, BRCA2, CA9, CASP3, CD8A, MS4A1, CD47, CD68, CDK1, CDC25C, COL7A1, HCCS, HPGD, CCL7, NRAS, SERPINA5, ABCB4, PIK3CA, PIK3CB, PIK3CG, PIK3R2, PLAGL1, POLD1, PRKCB, MAPK1, MAPK8, KLK7, PSD, RARA, RASGRF1, BRD2, CCL2, PEBP1, NME1, HRAS, MYC, HTC2, IRF8, IDS, IGF2, IL1B, IL6, LAG3, LAMB1, LGALS1, MDM4, MGMT, MITF, MKI67, MMP2, MRC1, MTAP, MUC1, H3P10
    • Recessive X-Linked Ichthyosis Orphanet
      Recessive X-linked ichthyosis (RXLI) is a genodermatosis belonging to the Mendelian Disorders of Cornification (MeDOC) and characterized by generalized hyperkeratosis and scaling of the skin. Epidemiology RXLI affects almost exclusively males. It is the second most common type of ichthyosis with an estimated prevalence of 1 / 2,000 to 1 / 6,000 males. Clinical description Onset may occur within the first days of life with the development of generalized non-erythematous, polygonal, loosely adherent scales. These scales later evolve into grayish or blackish adherent scales that are pronounced on the trunk, the extensor and flexor sites of the extremities, and the neck (giving the ''dirty-neck'' appearance). Skin's folds, palms and soles, are normally spared. Scaling improves with age and during summer.
  • White Dog Shaker Syndrome Wikipedia
    Textbook of Veterinary Internal Medicine (4th ed.). W.B. Saunders Company. ISBN 0-7216-6795-3 . ^ "What is shaker dog syndrome?"
  • Endocapillary Proliferative Glomerulonephritis Wikipedia
    Nephrol . 7 (4): 290–5. doi : 10.1007/s10157-003-0244-0 . PMID 14712359 . Kano K, Nishikura K, Kojima M, et al.
    CFH
  • Tight Hymenal Ring Wikipedia
    Lippincott Williams & Wilkins. p. 459. ISBN 978-0-7817-6805-4 . External links [ edit ] Classification D ICD - 10 : N89.6 ICD - 9-CM : 623.3 v t e Female diseases of the pelvis and genitals Internal Adnexa Ovary Endometriosis of ovary Female infertility Anovulation Poor ovarian reserve Mittelschmerz Oophoritis Ovarian apoplexy Ovarian cyst Corpus luteum cyst Follicular cyst of ovary Theca lutein cyst Ovarian hyperstimulation syndrome Ovarian torsion Fallopian tube Female infertility Fallopian tube obstruction Hematosalpinx Hydrosalpinx Salpingitis Uterus Endometrium Asherman's syndrome Dysfunctional uterine bleeding Endometrial hyperplasia Endometrial polyp Endometriosis Endometritis Menstruation Flow Amenorrhoea Hypomenorrhea Oligomenorrhea Pain Dysmenorrhea PMS Timing Menometrorrhagia Menorrhagia Metrorrhagia Female infertility Recurrent miscarriage Myometrium Adenomyosis Parametrium Parametritis Cervix Cervical dysplasia Cervical incompetence Cervical polyp Cervicitis Female infertility Cervical stenosis Nabothian cyst General Hematometra / Pyometra Retroverted uterus Vagina Hematocolpos / Hydrocolpos Leukorrhea / Vaginal discharge Vaginitis Atrophic vaginitis Bacterial vaginosis Candidal vulvovaginitis Hydrocolpos Sexual dysfunction Dyspareunia Hypoactive sexual desire disorder Sexual arousal disorder Vaginismus Urogenital fistulas Ureterovaginal Vesicovaginal Obstetric fistula Rectovaginal fistula Prolapse Cystocele Enterocele Rectocele Sigmoidocele Urethrocele Vaginal bleeding Postcoital bleeding Other / general Pelvic congestion syndrome Pelvic inflammatory disease External Vulva Bartholin's cyst Kraurosis vulvae Vestibular papillomatosis Vulvitis Vulvodynia Clitoral hood or clitoris Persistent genital arousal disorder This article about a disease of the genitourinary system is a stub .
  • Retiform Parapsoriasis Wikipedia
    St. Louis: Mosby. ISBN 1-4160-2999-0 . External links [ edit ] Classification D ICD - 10 : L41.5 ICD - 9-CM : 696.2 v t e Papulosquamous disorders Psoriasis Pustular Generalized pustular psoriasis ( Impetigo herpetiformis ) Acropustulosis / Pustulosis palmaris et plantaris ( Pustular bacterid ) Annular pustular psoriasis Localized pustular psoriasis Other Guttate psoriasis Psoriatic arthritis Psoriatic erythroderma Drug-induced psoriasis Inverse psoriasis Napkin psoriasis Seborrheic-like psoriasis Parapsoriasis Pityriasis lichenoides ( Pityriasis lichenoides et varioliformis acuta , Pityriasis lichenoides chronica ) Lymphomatoid papulosis Small plaque parapsoriasis ( Digitate dermatosis , Xanthoerythrodermia perstans ) Large plaque parapsoriasis ( Retiform parapsoriasis ) Other pityriasis Pityriasis rosea Pityriasis rubra pilaris Pityriasis rotunda Pityriasis amiantacea Other lichenoid Lichen planus configuration Annular Linear morphology Hypertrophic Atrophic Bullous Ulcerative Actinic Pigmented site Mucosal Nails Peno-ginival Vulvovaginal overlap synromes with lichen sclerosus with lupus erythematosis other: Hepatitis-associated lichen planus Lichen planus pemphigoides Other Lichen nitidus Lichen striatus Lichen ruber moniliformis Gianotti–Crosti syndrome Erythema dyschromicum perstans Idiopathic eruptive macular pigmentation Keratosis lichenoides chronica Kraurosis vulvae Lichen sclerosus Lichenoid dermatitis Lichenoid reaction of graft-versus-host disease This dermatology article is a stub .
    • Poikiloderma Vasculare Atrophicans Wikipedia
      Poikiloderma vasculare atrophicans Other names Parapsoriasis variegata [1] or Parapsoriasis lichenoides [2] Typical skin changes and discoloration described as poikiloderma vasculare atrophicans Specialty Dermatology Poikiloderma vasculare atrophicans ( PVA ), is a cutaneous condition ( skin disease ) characterized by hypo- or hyperpigmentation (diminished or heightened skin pigmentation , respectively), telangiectasia and skin atrophy . [3] [4] [5] Other names for the condition include prereticulotic poikiloderma and atrophic parapsoriasis . [6] The condition was first described by pioneer American pediatrician Abraham Jacobi in 1906. [7] PVA causes areas of affected skin to appear speckled red and inflamed, yellowish and/or brown, gray or grayish-black, with scaling and a thinness that may be described as "cigarette paper". [3] On the surface of the skin, these areas may range in size from small patches, to plaques (larger, raised areas), to neoplasms (spreading, tumor-like growths on the skin). [3] [6] Mycosis fungoides, a type of skin lymphoma , may be a cause of PVA. The condition may also be caused by, associated with or accompany any of the following conditions or disorders: other skin lymphomas, dermatomyositis , lupus erythematosus , Rothmund–Thomson syndrome , Kindler syndrome , dyskeratosis congenita , and chronic radiodermatitis . [4] Rare causes include arsenic ingestion, and the condition can also be idiopathic . [1] [3] [5] PVA may be considered a rare variant of cutaneous T-cell lymphoma , a non-Hodgkin's form of lymphoma affecting the skin. [7] It may also be included among a number of similar conditions that are considered as precursors to mycosis fungoides . PVA is believed to be a syndrome closely associated with large-plaque parapsoriasis and its cohort retiform parapsoriasis ; including PVA, all three conditions fit within an updated view of the once ambiguous classification scheme known as parapsoriasis . [5] Contents 1 Presentation 2 Cause 3 Diagnosis 3.1 Classification 4 Management 5 See also 6 References 7 External links Presentation [ edit ] The layers of the epidermis (left). Melanocytes (rlght), located in the bottom epidermal layer, produce melanin. PVA can be characterized by speckled, combined hyper- and hypopigmentation in the plaques or patches of affected skin. [5] Hyperpigmentation is excess coloration, or darkening of the skin, [8] while hypopigmentation is a diminished or pallid coloring to the skin.
  • Pseudocyst Of The Auricle Wikipedia
    Andrews' Diseases of the Skin: Clinical Dermatology . Saunders Elsevier. ISBN 978-0-7216-2921-6 . ^ Glamb, Roman; Kim, Robert (1984).
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