The protein CD55 (also called decay-accelerating factor) helps to regulate the complement cascade , part of the innate immune system , by regulating the amplification phase.
It is also appropriate to classify by site of origin: Atrial arrhythmia [ edit ] Sinus bradycardia Premature atrial contractions (PACs) Wandering atrial pacemaker Atrial tachycardia Multifocal atrial tachycardia Supraventricular tachycardia (SVT) Atrial flutter Atrial fibrillation (Afib) AV nodal reentrant tachycardia Junctional arrhythmia [ edit ] AV nodal reentrant tachycardia Junctional rhythm Junctional tachycardia Premature junctional contraction Ventricular arrhythmia [ edit ] Premature ventricular contractions (PVCs), sometimes called ventricular extra beats (VEBs) Premature ventricular beats occurring after every normal beat are termed ventricular bigeminy PVCs that occur at intervals of 2 normal beats to 1 PVC are termed "PVCs in trigeminy" Three premature ventricular grouped together is termed a "run of PVCs" in general, runs lasting longer than three beats with an increased heart rate are referred to as ventricular tachycardia Accelerated idioventricular rhythm Monomorphic ventricular tachycardia Polymorphic ventricular tachycardia Ventricular fibrillation Torsades de pointes Arrhythmogenic right ventricular dysplasia Re-entry ventricular arrhythmia Heart blocks [ edit ] These are also known as AV blocks, because the vast majority of them arise from pathology at the atrioventricular node . ... The term sinus arrhythmia [17] refers to a normal phenomenon of alternating mild acceleration and slowing of the heart rate that occurs with breathing in and out respectively. ... Archived from the original on 3 March 2015 . Retrieved 7 March 2015 . ^ a b "How Are Arrhythmias Diagnosed?" . National Heart, Lung, and Blood Institute. ... Retrieved 7 March 2015 . ^ a b c d e f g "How Are Arrhythmias Treated?" . National Heart, Lung, and Blood Institute. ... External links [ edit ] Classification D ICD - 10 : I47 – I49 ICD - 9-CM : 427 MeSH : D001145 DiseasesDB : 15206 External resources MedlinePlus : 001101 Arrhythmia at Curlie v t e Cardiovascular disease (heart) Ischaemic Coronary disease Coronary artery disease (CAD) Coronary artery aneurysm Spontaneous coronary artery dissection (SCAD) Coronary thrombosis Coronary vasospasm Myocardial bridge Active ischemia Angina pectoris Prinzmetal's angina Stable angina Acute coronary syndrome Myocardial infarction Unstable angina Sequelae hours Hibernating myocardium Myocardial stunning days Myocardial rupture weeks Aneurysm of heart / Ventricular aneurysm Dressler syndrome Layers Pericardium Pericarditis Acute Chronic / Constrictive Pericardial effusion Cardiac tamponade Hemopericardium Myocardium Myocarditis Chagas disease Cardiomyopathy Dilated Alcoholic Hypertrophic Tachycardia-induced Restrictive Loeffler endocarditis Cardiac amyloidosis Endocardial fibroelastosis Arrhythmogenic right ventricular dysplasia Endocardium / valves Endocarditis infective endocarditis Subacute bacterial endocarditis non-infective endocarditis Libman–Sacks endocarditis Nonbacterial thrombotic endocarditis Valves mitral regurgitation prolapse stenosis aortic stenosis insufficiency tricuspid stenosis insufficiency pulmonary stenosis insufficiency Conduction / arrhythmia Bradycardia Sinus bradycardia Sick sinus syndrome Heart block : Sinoatrial AV 1° 2° 3° Intraventricular Bundle branch block Right Left Left anterior fascicle Left posterior fascicle Bifascicular Trifascicular Adams–Stokes syndrome Tachycardia ( paroxysmal and sinus ) Supraventricular Atrial Multifocal Junctional AV nodal reentrant Junctional ectopic Ventricular Accelerated idioventricular rhythm Catecholaminergic polymorphic Torsades de pointes Premature contraction Atrial Junctional Ventricular Pre-excitation syndrome Lown–Ganong–Levine Wolff–Parkinson–White Flutter / fibrillation Atrial flutter Ventricular flutter Atrial fibrillation Familial Ventricular fibrillation Pacemaker Ectopic pacemaker / Ectopic beat Multifocal atrial tachycardia Pacemaker syndrome Parasystole Wandering atrial pacemaker Long QT syndrome Andersen–Tawil Jervell and Lange-Nielsen Romano–Ward Cardiac arrest Sudden cardiac death Asystole Pulseless electrical activity Sinoatrial arrest Other / ungrouped hexaxial reference system Right axis deviation Left axis deviation QT Short QT syndrome T T wave alternans ST Osborn wave ST elevation ST depression Strain pattern Cardiomegaly Ventricular hypertrophy Left Right / Cor pulmonale Atrial enlargement Left Right Athletic heart syndrome Other Cardiac fibrosis Heart failure Diastolic heart failure Cardiac asthma Rheumatic fever Authority control GND : 4024696-6 LCCN : sh85007430 NDL : 00563779 PLWABN : 9810553796805606
Find sources: "Premature heart beat" – news · newspapers · books · scholar · JSTOR ( March 2015 ) ( Learn how and when to remove this template message ) Premature heart beat A premature ventricular contraction marked by the arrow.
Women have a higher probability to experience depression than men, with the prevalences of 19.2% and 13.5% respectively. [15] Risk factor [ edit ] In childhood, boys and girls appear to be at equal risk for depressive disorders; during adolescence, however, girls are twice as likely as boys to develop depression, there is more pressure for girls to fit into society, meet the societal standard for how a girl should conduct herself and how she should behave and once girls cannot live up to certain standards they end up feeling bad about themselves. ... After adjusting for each individual's baseline body mass index (calculated as the weight in kilograms divided by the square of height in meters), depressed late adolescent girls were at a greater than 2-fold increased risk for obesity in adulthood compared with their non-depressed female peers (relative risk, 2.32; 95% confidence interval, 1.29-3.83). ... During cognitive behavioural therapy, children and adolescents with depression work with therapists to learn about their diagnosis, how to identify and reshape negative thought patterns, and how to increase engagement in enjoyable activities. [44] CBT-trained therapists work with individuals, families, and groups. ... Typically, the therapy will focus on one of the four specific problems, grief, social isolation, conflicts about roles and social expectations, or the effect of a major life change. [16] Family therapy [ edit ] The principles of group dynamics are relevant to family therapists who must not only work with individuals, but with entire family systems. [46] Family counseling can help families understand how a child's individual challenges may affect relations with parents and siblings and vice versa. [41] Therapists strive to understand not just what the group members say, but how these ideas are communicated (process). ... Virginia Satir expanded on the concept of how individuals behave and communicate in groups by describing several family roles that can serve to stabilize expected characteristic behavior patterns in a family.
Please help improve it by rewriting it in an encyclopedic style . ( April 2012 ) ( Learn how and when to remove this template message ) This article may be confusing or unclear to readers . ... There might be a discussion about this on the talk page . ( April 2012 ) ( Learn how and when to remove this template message ) ( Learn how and when to remove this template message ) Personality is the makeup of an individuals thinking, feeling, and behavior and is bound to change over the period of a lifetime. ... They modified the old, more monolithic construct by measuring how people differ across situations. ... Hence inconsistency is quantifiable much like a trait and constitutes an index of and enhances the fit of psychological models. ... ISBN 978-0-12-809846-2 . ^ Gurven, Michael; von Rueden, Christopher; Massenkoff, Maxim; Kaplan, Hillard; Lero Vie, Marino (2013). "How universal is the Big Five? Testing the five-factor model of personality variation among forager–farmers in the Bolivian Amazon" .
. ^ Thomas, Elizabeth (11 June 2018). "Be wary of Dr Google" . The Asian Age . ^ Oxford English Dictionary #rd Ed. (2003) ^ "The Future Use of the Internet in 4 Countries in Relation to Prescriptions, Physician Communication and Health Information" (PDF) .
Lexikon Psychiatrie, Psychotherapie, Medizinische Psychologie . Dipsomania entry at Google Books. ^ Wiley.com journal ^ NLA record ^ Tracy, Sarah W. (2005).
Although variations in several of these genes have been associated with alcohol use disorder, it is unclear how these genetic changes influence the way in which the nervous system responds to alcohol.
Thus, the proper question is not ‘whether a person is dependent on alcohol’, but ‘how far along the path of dependence has a person progressed’. ... Sulfonic acids : Acamprosate Religion and alcohol Christian views on alcohol alcohol in the Bible Islam and alcohol History Bratt System Related Index of alcohol-related articles Austrian syndrome Ban on caffeinated alcoholic beverages Brief intervention Gateway drug effect Last call Mood disorder Non-alcoholic fatty liver disease Self-medication Spins Sober companion Sober living houses Sobering center Town drunk Category
Patients also differ in durations of RA (how long they can't recall information) and durations of what is forgotten (past time frame for which information is unavailable). ... Amnesic patients with damage to the hippocampus are able to demonstrate some degree of unimpaired semantic memory , despite a loss of episodic memory , due to spared parahippocampal cortex . [33] In other words, retrograde amnesics "know" about information or skill, but cannot "remember" how they do. The diencephalon and the surrounding areas' role in memory is not well understood. ... The effect of the memory loss was the same no matter how long it had been after from the injury. [38] Brain abnormalities can be measured using magnetic resonance imaging (MRI), computed tomography scan (CT) and electroencephalography (EEG), which can provide detailed information about specific brain structures. ... When he saw his family and friends, he was shocked at how old they looked because he remembered them from 5 years earlier. ... This was a classic case of PRA. [57] GC was a 38 year old accountant that was found in a town square unable to remember anything about himself and unaware of where he was and how he got there. He was eventually able to recall basic information about himself and his family, but could not recall emotionally charged autobiographical events pertaining to the last 7 years of his life.
Epidemiology The prevalence of multiple endocrine neoplasia type 4 is unknown, but the syndrome is very rare. To date, 12 index cases have been reported. Prevalence is thought to be less than 1/million.
A number sign (#) is used with this entry because of evidence that multiple endocrine neoplasia type IV (MEN4) is caused by heterozygous mutation in the CDKN1B gene (600778) on chromosome 12p13. For a phenotypic description and a discussion of genetic heterogeneity of multiple endocrine neoplasia, see MEN1 (131100). Clinical Features Pellegata et al. (2006) reported a 3-generation family with what appeared to be MEN1-related tumors. The proband was a 48-year-old Caucasian female who developed acromegaly and had a 3-cm pituitary tumor removed at age 30. Histology revealed an invasive pituitary adenoma with growth hormone hyperproduction, high mitotic activity, and cell atypia.
Moses et al. (1982) reported a family in which 2 euthyroid persons had an elevated total T4, an elevated or normal T3 resin uptake, and an increased free T4 index. Free T4 and total and free T3 concentrations were normal.
It is characterised by preferential flow of blood towards the brain at the expense of the other vital organs, and it occurs as a haemodynamic adaptation in foetuses which have placental insufficiency . [1] The underlying mechanism is thought to be vasodilation of the cerebral arteries . [2] Cerebral redistribution is defined by the presence of a low middle cerebral artery pulsatility index (MCA-PI). Ultrasound of the middle cerebral artery to examine the Doppler waveform is used to establish this. [3] Although cerebral redistribution represents an effort to preserve brain development in the face of hypoxic stress, it is nonetheless associated with adverse neurodevelopmental outcome. [1] The presence of cerebral redistribution will be one factor taken into consideration when deciding whether to artificially deliver a baby with placental insufficiency via induction of labour or caesarian section .
Contents 1 Tumors occupying specific organs 2 Tumors occupying intra-organ spaces 3 Complications 4 References Tumors occupying specific organs [ edit ] Bladder cancer Prostate cancer Rectal cancer Anal cancer Ovarian cancer Uterine cancer Sacrococcygeal teratoma Tumors occupying intra-organ spaces [ edit ] Presacral space : Teratoma Sacral space (in approximate order of prevalence): Teratoma Lipoma Ganglioneuroma Myxopapillary ependymoma Primitive neuroectodermal tumor Aneurysmal bone cyst Ewing's sarcoma Metastases from brain stem tumors ( medulloblastoma , ependymoma , high-grade astrocytoma ) Complications [ edit ] Urinary incontinence Fecal incontinence References [ edit ] v t e Overview of tumors , cancer and oncology Conditions Benign tumors Hyperplasia Cyst Pseudocyst Hamartoma Malignant progression Dysplasia Carcinoma in situ Cancer Metastasis Primary tumor Sentinel lymph node Topography Head and neck ( oral , nasopharyngeal ) Digestive system Respiratory system Bone Skin Blood Urogenital Nervous system Endocrine system Histology Carcinoma Sarcoma Blastoma Papilloma Adenoma Other Precancerous condition Paraneoplastic syndrome Staging / grading TNM Ann Arbor Prostate cancer staging Gleason grading system Dukes classification Carcinogenesis Cancer cell Carcinogen Tumor suppressor genes / oncogenes Clonally transmissible cancer Oncovirus Carcinogenic bacteria Misc. Research Index of oncology articles History Cancer pain Cancer and nausea This article about a neoplasm is a stub .
Heberden's node Heberden's nodes on the 2nd (index) finger of the right hand Specialty Rheumatology Heberden's nodes are hard or bony swellings that can develop in the distal interphalangeal joints (DIP) (the joints closest to the end of the fingers and toes). [1] They are a sign of osteoarthritis and are caused by formation of osteophytes (calcific spurs ) of the articular (joint) cartilage in response to repeated trauma at the joint. [2] Heberden's nodes typically develop in middle age, beginning either with a chronic swelling of the affected joints or the sudden painful onset of redness, numbness, and loss of manual dexterity .
