Arterial emboli may recur even after successful treatment. [2] Complications [ edit ] Possible complications of arterial embolism depend on the site of the obstruction: In the heart it can cause myocardial infarction [2] In the brain, it can cause a transient ischemic attack (TIA), [2] and, in prolonged blood obstruction, stroke . [2] Blockage of arteries that supply arms or legs may result in necrosis and gangrene [1] Temporary or permanent decrease or loss of other organ functions [2] In septic embolism , there can be infection of the affected tissue or even septic shock, [2] Epidemiology [ edit ] In the United States , approximately 550,000 people die each year from heart-related arterial embolism and thrombosis. [1] Approximately 250,000 of these individuals are female, [1] and approximately 100,000 of all these deaths are considered premature, that is, prior to the age of average life expectancy. [1] References [ edit ] ^ a b c d e f g h i j k l m n o p q r s t u v w x y z aa ab ac ad ae af ag ah ai aj ak al am an ao ap aq ar as at au av MDGuidelines > Arterial Embolism And Thrombosis From The Medical Disability Advisor by Presley Reed, MD. Retrieved on April 30, 2010 ^ a b c d e f g h i j k l m n o p q r s t u v w x y z aa ab ac ad ae af ag ah ai aj ak al am an ao ap aq ar as at au MedlinePlus > Arterial embolism Sean O.
. ^ Peuskens J, Pani L, Detraux J, De Hert M (May 2014). "The effects of novel and newly approved antipsychotics on serum prolactin levels: a comprehensive review" . ... Courtois F, Charvier K, Leriche A, Vézina JG, Côté I, Raymond D, Jacquemin G, Fournier C, Bélanger M (Oct 2008). "Perceived physiological and orgasmic sensations at ejaculation in spinal cord injured men".
There is wide consensus that these motor complications depend on both pre- and post-synaptic disturbances of nigrostriatal DA transmission. ^ Halje, P; Tamtè, M; Richter, U; Mohammed, M; Cenci, MA; Petersson, P (21 November 2012).
Albrecht, "Encyclopedia of disability, Volume 2", Sage Publications, 2006, ISBN 0-7619-2565-1 , p. 1437 Midori , "Wild Side Sex: The Book of Kink Educational, Sensual, And Entertaining Essays", Daedalus Publishing, 2005, ISBN 1-881943-22-4 , p. 211 v t e Sexual fetishism Actions, states Aquaphilia Autassassinophilia Coprophilia Cuckold / Cuckquean Emetophilia Erotic hypnosis Erotic lactation Erotic spanking Exhibitionism Forced seduction Gaining and feeding Medical fetishism Omorashi Paraphilic infantilism (adult baby) Pregnancy Smoking Tickling Total enclosure Transvestic Tightlacing Tamakeri Urolagnia Vorarephilia Wet and messy fetishism Body parts Armpit Breast Belly Buttocks Eyeball Fat Feet Hands Height Hair Legs Navels Noses Clothing Boots Ballet boots Boot worship Thigh-high boots Clothing Corset Diapers Gloves Pantyhose Latex Rubber and PVC Shoes Spandex Underwear Uniforms Objects Balloons Dolls Latex and PVC Robots Spandex Controversial / illegal Lust murder Necrophilia Rape fantasy Zoophilia Culture / media Artists Fetish art Fetish clubs Fashion Magazines Models Race Asian sexual fetishism Ethnic pornography Sexual racism Related topics BDSM FetLife International Fetish Day Kink Leather subculture Leather Pride flag Sexual roleplay Book Category v t e Outline of BDSM Glossary Index Bondage and discipline B&D or B/D Animal roleplay Bondage hood Bondage positions and methods Bondage suit Collar Equipment Erotic sexual denial Erotic tickling Forced orgasm Head bondage Hogtie bondage Human furniture In culture and media Interrogation scene Japanese bondage Law Metal bondage Mummification Organizations Positions Predicament bondage Rope bondage Self-bondage Sensation play Spreadeagle position Suspension bondage Total enclosure Dominance and submission D&S or D/s Ageplay Bladder desperation Body worship Boot worship Dominatrix Erotic humiliation Facesitting Fear play Female submission Feminization Male dominance Male submission Master/slave Medical fetishism Rape fantasy Forced seduction Servitude Sadomasochism S&M or S/M Breast torture Caning Cock and ball torture Erotic asphyxiation Erotic electrostimulation Erotic spanking Figging Impact play Knife play Play piercing Pussy torture Temperature play Urethral sounding Violet wand Wax play In fiction Related topics Consent Dungeon monitor Edgeplay Edging Feminist views on BDSM Gorean subculture International Fetish Day Kink Leathermen Leather Pride flag Limits Munch Pegging Play Play party Risk-aware consensual kink Safeword Sexual fetishism Sexual roleplay Top, bottom, switch Writers Laura Antoniou Pauline Réage Catherine Robbe-Grillet Leopold von Sacher-Masoch John Norman Commentators and theorists Gloria Brame Patrick Califia Dossie Easton Janet Hardy Trevor Jacques Fakir Musafar Gayle Rubin Organizations FetLife Universities with BDSM clubs Category
A disorder that caused by the Junin virus (JUNV), is an acute viral hemorrhagic disease characterized by initial fever and malaise followed by gastrointestinal symptoms and in some cases hemorrhagic and neurological manifestations.
Cite journal requires |journal= ( help ) ^ Wilson, William C.; Grande, Christopher M.; Hoyt, David B. (2007-02-05). Trauma: Critical Care . ... ISBN 9781118889626 . Irwin, Richard S.; Rippe, James M. (2012-03-28). Manual of Intensive Care Medicine .
PMC 1835547 . PMID 2522326 . ^ Makris M, Bardhan G, Preston FE (March 1996). ... PMID 13171021 . ^ Schramm W, Spannagl M, Bauer KA, et al. (June 1993). "Treatment of coumadin-induced skin necrosis with a monoclonal antibody purified protein C concentrate".
Homocystinuria due to MTHFR deficiency is a genetic condition that results from poor metabolism of folate (also called vitamin B9), due to a lack of working enzyme called MTHFR. The gene that tells our body how to make the enzyme is also called MTHFR . At least 40 rare MTHFR gene variants have been found in people with decreased or no working enzyme. Very common gene variants (C677T and A1298C) can cause some decrease in enzyme function. People with homocystinuria due to MTHFR deficiency tend to have two rare variants or sometimes a rare variant and a common variant.
A number sign (#) is used with this entry because homocystinuria due to methylenetetrahydrofolate reductase deficiency is caused by homozygous or compound heterozygous mutation in the MTHFR gene (607093) on chromosome 1p36. Description Methylenetetrahydrofolate reductase deficiency is a common inborn error of folate metabolism. The phenotypic spectrum ranges from severe neurologic deterioration and early death to asymptomatic adults. In the classic form, both thermostable and thermolabile enzyme variants have been identified (Rosenblatt et al., 1992). Clinical Features Freeman et al. (1972) studied a 15-year-old mildly retarded black female with a 2-year history of progressive withdrawal, hallucinations, delusions, and catatonia unresponsive to psychotherapy.
Homocystinuria due to methylene tetrahydrofolate reductase (MTHFR) deficiency is a metabolic disorder characterised by neurological manifestations. Epidemiology The prevalence is unknown. Clinical description Onset usually occurs during the first year of life with severe neurological signs, recurrent apnoea, microcephaly and convulsions. There is no megaloblastic anaemia. There are some forms with onset during childhood, adolescence, or adulthood beginning with mental regression, ataxia, and, most often, common psychiatric disorders of the schizophrenic type that may be linked to cerebrovascular accidents. Other symptoms such as subacute degeneration of the spinal chord have been reported. Etiology It is caused by mutations in the MTHFR gene (1p36.3). MTHFR deficiency results in abnormal intracellular folic acid metabolism and prevents reduction of 5-10 methylenetetrahydrofolate to 5-methyltetrahydrofolate, the methyl donor for the remethylation of homocysteine into methionine.
See also [ edit ] Cupola sign Football sign Pneumoretroperitoneum Rigler's sign References [ edit ] ^ Necrotizing Enterocolitis Bugs, Drugs and Things That Go Bump in the Night ^ Imran, M.; Bhat, R.; Anijeet, H. (2011). "Pneumoperitoneum in peritoneal dialysis patients; one centre's experience" . ... CS1 maint: multiple names: authors list ( link ) ^ Hirsch M, Cortés C. THE LEO RIGLER SIGN: DOUBLE WALL IN PNEUMO-PERITONEUM. http://dx.doi.org/10.4067/S0717-93082009000300008 ^ Rampton JW (April 2004).
. ^ Pezzella FR, Di Rezze S, Chianese M, et al. (October 2003). "Hedonistic homeostatic dysregulation in Parkinson's disease: a short screening questionnaire". ... PMID 25093777 . ^ Pirritano D, Plastino M, Bosco D, Gallelli L, Siniscalchi A, De Sarro G (2014).
Retrieved 4 February 2010 . ^ a b Jones, G. M.; Bailey, T. L. "Understanding the Basics of Mastitis" . ... Dairy Sci. 77:2103-2112. Jones, G. M., R. E. Pearson, G. A. Clabaugh, and C.
Specialty Hematology Megaloblastic anemia is a type of macrocytic anemia that results from inhibition of DNA synthesis during red blood cell production. [1] When DNA synthesis is impaired, the cell cycle cannot progress from the G2 growth stage to the mitosis (M) stage. This leads to continuing cell growth without division, which presents as macrocytosis. ... P.; Boyer, P. J.; Love, L. A.; Burritt, M. F.; Butz, J. A.; Wolfe, G. I.; Hynan, L.
PMID 23158648 . ^ Urbina, F.; Sudy, E.; Barrios, M. (2009). "Traction folliculitis: 6 cases caused by different types of hairstyle that pull on the hair" . ... International Journal of Dermatology . 48 (8): 840–845. doi : 10.1111/j.1365-4632.2009.04129.x . PMID 19659862 . ^ a b Fu, J. M.; Price, V. H. (2009). "Approach to Hair Loss in Women of Color".
Traumatic alopecia Specialty Dermatology Traumatic alopecia is a cutaneous condition that results from the forceful pulling out of the scalp hair. [1] See also [ edit ] Traction alopecia List of cutaneous conditions References [ edit ] ^ Rapini, Ronald P.; Bolognia, Jean L.; Jorizzo, Joseph L. (2007). Dermatology: 2-Volume Set . St. Louis: Mosby. p. 1388. ISBN 978-1-4160-2999-1 . This dermatology article is a stub . You can help Wikipedia by expanding it . v t e
The primary methods of treatment involve adjustments to playing of the instrument: [3] [5] good hygiene for the affected area and for the instrument use of a clean cotton cloth that is changed frequently use of a comfortable pad to absorb sweat and reduce friction between the instrument and skin use of a shoulder rest to reduce pressure below the jaw a suitable chin rest, especially one carved or molded for the individual Covering or changing potentially allergenic materials on the instrument. shifting the chin rest to the center of the body over the tailpiece smoothing coarse surfaces to reduce abrasion for males, growing a beard to avoid folliculitis Surgery is necessary for sialolithiasis, parotid tumors, and cysts. [3] Cervical lymph nodes that are larger than 1 cm must be biopsied. [3] Connective tissue can be removed by excision when a non-inflamed mass is large, and there is generally little recurrence. [3] Infections should be treated conservatively, and causative species should be identified through smear and culture for appropriate antibiotic selection. [3] Reduction of playing time may be helpful for cases without inflammation, but in 30% of cases this did not improve the symptoms. [3] Prevalence [ edit ] Fiddler's neck does not usually form unless the musician is practicing or playing for more than a few hours each day, and only seems to develop after a few years of serious playing. [2] Thus, when not infected or otherwise problematic, fiddler's neck may be known as a benign practice mark and may be worn proudly as an indication of long hours of practice. [3] Blum & Ritter (1990) found that 62% of 523 professional violinists and violists in West Germany experienced fiddler's neck, with the percentage among violists being higher (67%) than among violinists (59%). [3] Viola players are believed to be more predisposed to developing fiddler's neck than violinists because the viola is larger and heavier, but this has not been empirically confirmed. [2] The development of fiddler's neck does not depend on preexisting skin problems, and Blum & Ritter find that only 23% of men and 14% of women in their study reported cutaneous disorders in other parts of the face (mainly acne and eczema ) that were independent of playing the violin or viola. [3] Fiddler's neck may exacerbate existing acne, but acne may also be limited solely to the lesion and not appear elsewhere. [2] Nonetheless, musicians with underlying dermatologic diseases like acne and eczema are more endangered by fiddler's neck than others. [3] Males may develop folliculitis or boils due to involvement of beard hair. [3] References [ edit ] ^ a b c d e f g h i Gambichler, T.; Boms, S.; Freitag, M. (2004). "Contact dermatitis and other skin conditions in instrumental musicians" . ... PMID 150281 . ^ a b c d e f g h i j k l m n o p q r s t Blum, Jochen; Ritter, G.
World Journal of Hematology . 3 (1): 1–7. ^ a b c d e Frieri M (June 2018). "Mast Cell Activation Syndrome". ... Retrieved 27 April 2020 . ^ Sonneck K, Florian S, Müllauer L, Wimazal F, Födinger M, Sperr WR, Valent P (2007). "Diagnostic and subdiagnostic accumulation of mast cells in the bone marrow of patients with anaphylaxis: Monoclonal mast cell activation syndrome".
All had similar side effects, but PEG was more effective at treating constipation. [11] [12] Osmotic laxatives are recommended over stimulant laxatives. [13] Epidemiology [ edit ] There is wide variation in the rates of constipation as reported by research in various countries. [14] The variation in research data makes it challenging to describe the true global situation. [14] Approximately 3% of children have constipation, with girls and boys being equally affected. [4] With constipation accounting for approximately 5% of general pediatrician visits and 25% of pediatric gastroenterologist visits, the symptom carries a significant financial impact upon our healthcare system. [1] Society and culture [ edit ] Constipation is often emotionally stressful for children and their caregivers. [15] It is common for parents to bring their children to doctors for this condition. [15] The experience of going to a doctor for this can be stressful. [15] Too often, children at doctors receive unnecessary health care when they get medical imaging for constipation. [16] Children should only get tests when there is an indication . [16] References [ edit ] ^ a b c d Colombo, Jennifer M.; Wassom, Matthew C.; Rosen, John M. (2015-09-01).
Overview Constipation in children is a common problem. A constipated child has infrequent bowel movements or hard, dry stools. Common causes include early toilet training and changes in diet. Fortunately, most cases of constipation in children are temporary. Encouraging your child to make simple dietary changes — such as eating more fiber-rich fruits and vegetables and drinking more water — can go a long way toward alleviating constipation. If your child's doctor approves, it may be possible to treat a child's constipation with laxatives. Symptoms Signs and symptoms of constipation in children may include: Less than three bowel movements a week Bowel movements that are hard, dry and difficult to pass Pain while having a bowel movement Stomach pain Traces of liquid or pasty stool in your child's underwear — a sign that stool is backed up in the rectum Blood on the surface of hard stool If your child fears that having a bowel movement will hurt, he or she may try to avoid it.
. ^ Bradley DJ, Fischbach PS, Law IH, Serwer GA, Dick M (August 2001). "The clinical course of multifocal atrial tachycardia in infants and children". ... Retrieved 2013-04-24 . ^ a b c d e f g Custer, Adam M.; Yelamanchili, Varun S.; Lappin, Sarah L. (2020).
Multifocal atrial tachycardia is a rare supraventricular arrhythmia in neonates and young infants that is characterized by multiple P waves with varying P wave morphology and is usually asymptomatic. Epidemiology It is a very rare condition occurring in around 1 per 150,000 live births. Clinical description 'The disease mainly affects newborn infants (or those younger than 6 months of age) with a normal heart and no other underlying illness. Most infants are asymptomatic but some may show shortness of breath or respiratory distress. Less often, the disorder may occur in children with heart malformations (such as hypertrophic cardiomyopathy, tetralogy of Fallot, or atrioventricular canal defect; see these terms) or in those having recently undergone an open-heart surgery.
The first cases of buried bumper syndrome were reported in 1988 and 1989. [11] The term "buried bumper syndrome" was first used in 1990. [8] See also [ edit ] Percutaneous endoscopic gastrostomy References [ edit ] ^ Pinho, J; Libânio, D; Pimentel-Nunes, P; Dinis-Ribeiro, M (April 2018). "The Challenging Acute Buried Bumper Syndrome: A Case Report" . ... PMID 12297784 . ^ Cyrany, J; Rejchrt, S; Kopacova, M; Bures, J (14 January 2016). "Buried bumper syndrome: A complication of percutaneous endoscopic gastrostomy" .
PMID 7194471 . S2CID 31143908 . ^ Urioste M, Rodriguez JL, Bofarull J, Toran N, Ferrer C, Villa A (1997). ... PMID 4063680 . ^ a b Lu J, Lian G, Lenkinski R, De Grand A, Vaid RR, Bryce T, Stasenko M, Boskey A, Walsh C, Sheen V (2007).
Boomerang dysplasia is a disorder that affects the development of bones throughout the body. Affected individuals are born with inward- and upward-turning feet (clubfeet ) and dislocations of the hips, knees, and elbows. Bones in the spine, rib cage, pelvis, and limbs may be underdeveloped or in some cases absent. As a result of the limb bone abnormalities, individuals with this condition have very short arms and legs. Pronounced bowing of the upper leg bones (femurs) gives them a "boomerang" shape.
A number sign (#) is used with this entry because of evidence that boomerang dysplasia (BOOMD) is caused by heterozygous mutation in the FLNB gene (603381) on chromosome 3p14. Clinical Features Kozlowski et al. (1981), Tenconi et al. (1983), and Kozlowski et al. (1985) each described 1 patient with a disorder termed boomerang dysplasia because of the unusual shape of the long bones of the legs. All 3 patients died in the neonatal period. They had dwarfism with short, bowed, rigid limbs and characteristic facies. In particular, the nose had a broad root and severe hypoplasia of the nares and septum. Radiographically, the radii and fibulae were absent, while the remaining long bones had the boomerang configuration.
Boomerang dysplasia (BD) is a rare lethal skeletal dysplasia characterized by severe short-limbed dwarfism, dislocated joints, club feet, distinctive facies and diagnostic x-ray findings of underossified and dysplastic long tubular bones, with a boomerang-like bowing. Epidemiology The prevalence of BD is unknown. Clinical description Affected neonates are stillborn or die rapidly after birth and present clinically with severe short-limbed dwarfism, dislocated hip, knee and elbow joints, club feet and proviso born alive have severe cardio respiratory failure. Facial dysmorphism includes midface hypoplasia and cleft palate. Boomerang dysplasia clinically differs from AOI and AOIII because of the boomerang shaped bowing of the femur and occasionally observed encephalocele and omphalocele. Etiology BD results from missense mutations or small in-frame deletions in the FLNB gene reported in exons 2-5, normally expected to translate full length but biochemically abnormal filamin B protein. Diagnostic methods Diagnosis can be confirmed from skeletal radiographs, chondro-osseous histopathology and genetic testing.
Eosinophilic fasciitis is a very rare condition in which muscle tissue underneath the skin, called fascia, becomes swollen and thick. Rapid swelling can occur in the hands, arms, legs, and feet. People with this condition have a buildup of eosinophils, a type of white blood cell, in the affected fascia and muscles. The exact cause of this condition is unknown. Corticosteroids and other immune-suppressing medications are used to relieve the symptoms. Eosinophilic fasciitis is similar in appearance to scleroderma . However, in contrast with systemic sclerosis, internal organ involvement in eosinophilic fasciitis is generally absent. Some researchers believe that eosinophilic fasciitis may be a variant of morphea (localized scleroderma).
A rare idiopathic inflammatory myopathy that is characterized by inflammation and thickening of the fascia, usually associated with peripheral eosinophilia. It presents during adulthood with symmetrical and painful swelling of mainly the extremities that progressively become indurated. Fatigue, disabling cutaneous fibrosis, myositis and arthritis may also be observed.
Thomson et al. (1989) described a brother and sister, aged 33 and 38 years, respectively, who developed eosinophilic fasciitis within a period of 6 months. They were found to have identical HLA-A, -B, -DR, and -DQ antigens. No common environmental factors close to the time of onset were identified. Muscle - Eosinophilic fasciitis Inheritance - Autosomal recessive ▲ Close
