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  • Uremia Wikipedia
    Lippincott Williams and Wilkins. p. 268 . ^ a b c d e f g h i j k l Burtis, C.A.; Ashwood, E.R.; Bruns, D.E. ... PMID 3292814 . ^ a b c d e f g h i j k l Burket's oral medicine . Glick, Michael (12th ed.).
    PTH, VDR, SPP1, RUNX2, TNFRSF11B, NOS3, EPO, TGFB1, TGFBR1, SPARC, SGK1, VCAM1, MGP, KDR, ICAM1, BMP2, FLT1, FGF23, EDN1, MSTN, AGER, TH, ALPL, ANGPT2, NOS2, PTK2, AGL, HNRNPD, MEN1, IL6, KL, TNF, APOE, COL18A1, HPT, VEGFA, LEP, PTX3, PON1, TPMT, ADAM17, ANGPT1, STAT5B, STAT5A, TRPC6, TEK, PLA2G7, ALB, ALPP, CCN4, AGT, AOC3, MIR21, MIR204, MIR142, MYOCD, ASRGL1, NAT10, DYM, GHRL, ADIPOR1, PDLIM3, ATRNL1, CCL27, CITED2, GDF11, KEAP1, GCM2, SLPI, APOA1, ACE, SLC20A2, BTF3P11, IL15, IL2, CFB, HMOX1, HLA-DRB1, GPT, BMP7, FGF2, KRT1, FCGR3B, FCGR3A, CRP, STX2, EGFR, CTNNB1, DLX5, B2M, MB, SLC2A1, PRKCA, RET, REN, DECR1, PTH1R, FAS, PROS1, PROC, ARG1, MBL2, NOTCH1, MTHFR, MPO, MMP9, MMP2, ATHS, MC4R, RN7SL263P
  • Variant Angina Wikipedia
    PMC 6486012 . ^ a b c d e f g h i j k l m n o p q r s Ahmed B, Creager MA (April 2017). ... PMID 25858537 . ^ Kajihara H, Tachiyama Y, Hirose T, Takada A, Takata A, Saito K, Murai T, Yasui W (2013). "Eosinophilic coronary periarteritis (vasospastic angina and sudden death), a new type of coronary arteritis: report of seven autopsy cases and a review of the literature".
    NOS3, ACE, EDN1, ALDH2, SMUG1, AKR1A1, ABCC9, MED23, ADIPOQ, KL, LGI1, PON1, ABCB1, PCYT1A, KCNJ8, APOE, HTR2A, HTR1D, HTR1B, HSPA9, CSH2, CSH1, CLCN1, ERCC8, VPS51, CERNA3
  • Meconium Aspiration Syndrome Wikipedia
    Neonatology . 107 : 225–230. doi : 10.1159/000369373 . ^ a b c d Swarnam, K; Soraisham, AS; Sivanandan, S (2012). ... PMID 15634032 . ^ Bassler, D; Choong, K; McNamara, P; Kirpalani, H (2006). ... External links [ edit ] eMedicine's article about meconium aspiration syndrome Classification D ICD - 10 : P24.0 ICD - 9-CM : 770.11 , 770.12 MeSH : D008471 DiseasesDB : 7907 External resources MedlinePlus : 001596 eMedicine : ped/768 Orphanet : 70588 v t e Conditions originating in the perinatal period / fetal disease Maternal factors complicating pregnancy, labour or delivery placenta Placenta praevia Placental insufficiency Twin-to-twin transfusion syndrome chorion / amnion Chorioamnionitis umbilical cord Umbilical cord prolapse Nuchal cord Single umbilical artery presentation Breech birth Asynclitism Shoulder presentation Growth Small for gestational age / Large for gestational age Preterm birth / Postterm pregnancy Intrauterine growth restriction Birth trauma scalp Cephalohematoma Chignon Caput succedaneum Subgaleal hemorrhage Brachial plexus injury Erb's palsy Klumpke paralysis Affected systems Respiratory Intrauterine hypoxia Infant respiratory distress syndrome Transient tachypnea of the newborn Meconium aspiration syndrome Pleural disease Pneumothorax Pneumomediastinum Wilson–Mikity syndrome Bronchopulmonary dysplasia Cardiovascular Pneumopericardium Persistent fetal circulation Bleeding and hematologic disease Vitamin K deficiency bleeding HDN ABO Anti-Kell Rh c Rh D Rh E Hydrops fetalis Hyperbilirubinemia Kernicterus Neonatal jaundice Velamentous cord insertion Intraventricular hemorrhage Germinal matrix hemorrhage Anemia of prematurity Gastrointestinal Ileus Necrotizing enterocolitis Meconium peritonitis Integument and thermoregulation Erythema toxicum Sclerema neonatorum Nervous system Perinatal asphyxia Periventricular leukomalacia Musculoskeletal Gray baby syndrome muscle tone Congenital hypertonia Congenital hypotonia Infections Vertically transmitted infection Neonatal infection rubella herpes simplex mycoplasma hominis ureaplasma urealyticum Omphalitis Neonatal sepsis Group B streptococcal infection Neonatal conjunctivitis Other Miscarriage Perinatal mortality Stillbirth Infant mortality Neonatal withdrawal
    ATF2, AGT, AGTR2, NAP1L1, OPTN, NRP1, PTGS2, NOS2, GNAS, LINC01672, CPOX, DMBT1, NANOS2, ACE2, TLR8, TLR7, AMY1A, COX5A, AMY1B, SCGB1A1, TLR4, TLR1, REN, AMY1C, PTGS1, NT5E, NOS3, C5, NOS1, CD14, NR3C2, MAS1, KCNJ13, CXCL8, COX8A, GH1, GDNF, AFP
    • Meconium Aspiration Syndrome Orphanet
      Meconium aspiration syndrome is a pulmonary complication appearing in newborns with a meconium-stained amniotic fluid. Aspirated meconium can interfere with normal breathing by several mechanisms including airway obstruction, chemical irritation, infection and surfactant inactivation and induces more or less severe signs of respiratory distress at birth.
    • Meconium Aspiration Syndrome GARD
      Meconium aspiration syndrome is a serious condition in which a newborn breathes a mixture of meconium and amniotic fluid into the lungs around the time of delivery. This can cause breathing difficulties due to swelling (inflammation) in the baby's lungs after birth. Treatment may include suctioning the newborn's mouth as soon as the head emerges during delivery, deep suctioning of the windpipe, antibiotics to treat infection, oxygen to keep blood levels normal, and radiant heat to maintain body temperature. In severe cases, the baby may need assistance breathing (ventilator).
  • Porphyria Cutanea Tarda Wikipedia
    . ^ Sökmen, M; Demirsoy, H; Ersoy, O; Gökdemir, G; Akbayir, N; Karaca, C; Ozdil, K; Kesici, B; Calişkan, C; Yilmaz, B (2007). ... Scandinavian Journal of Clinical and Laboratory Investigation . 60 (7): 509–40. doi : 10.1080/003655100448310 . PMID 11202048 . ^ Singal, Ashwani K.; Kormos–Hallberg, Csilla; Lee, Chul; Sadagoparamanujam, Vaithamanithi M.; Grady, James J.; Freeman, Daniel H.; Anderson, Karl E. (2012).
    UROD, HFE, CYP1A2, CPOX, HAMP, IL6, CD6, PTEN, TFRC, UROS, SLC40A1, KL, HPGDS, SLCO6A1, HJV, OR10A4, TNF, BCL2, TFR2, SOAT1, SERPINA1, MMP3, GSTT1, GSTM1, GLS, FECH, EEF1A2, CYP2E1, CYP1A1, GSTK1
    • Porphyria Cutanea Tarda Orphanet
      Porphyria cutanea tarda (PCT) is the most common form of chronic hepatic porphyria (see this term). It is characterized by bullous photodermatitis. Epidemiology The prevalence in western Europe is estimated at about 1/25,000 and men are more affected than women. Clinical description The disease manifests in adulthood. PCT is acquired (75% of cases) or familial (25% of cases). Generally manifestations of the disease appear earlier in familial cases. Some risk factors can precipitate symptoms: excessive consumption of alcohol, hepatitis C, estrogen, and mutations of the genes that control iron metabolism, leading to iron overload (hemochromatosis).
    • Porphyria Cutanea Tarda, Type I OMIM
      Description De Verneuil et al. (1978) classified porphyria cutanea tarda (PCT), the most common type of porphyria, into 2 types: type I, or 'sporadic' type, associated with approximately 50% level of uroporphyrinogen decarboxylase (UROD; 613521) in liver (Elder et al., 1978; Felsher et al., 1982), and type II, or 'familial' type (176100), characterized by 50% deficient activity of the same enzyme in many tissues (Kushner et al., 1976; Elder et al., 1980). Type I is the most common form of PCT, comprising 70 to 80% of cases. The causes of the deficiency are often unclear and are probably multifactorial (review by Lambrecht et al., 2007). Clinical Features Among 10 families containing 17 patients with PCT, Roberts et al. (1988) found that 4 were of type I. Each of these 4 families contained at least 2 subjects with overt PCT; all members of these families, including 7 patients with overt PCT, had normal red cell uroporphyrinogen decarboxylase immunoreactive concentrations and enzyme activities.
    • Porphyria Cutanea Tarda OMIM
      A number sign (#) is used with this entry because porphyria cutanea tarda type II, or familial PCT, is caused by heterozygous mutation in the gene encoding uroporphyrinogen decarboxylase (UROD; 613521). Hepatoerythropoietic porphyria (HEP) is caused by homozygous or compound heterozygous mutation in the UROD gene. Description Porphyria cutanea tarda (PCT) is characterized by light-sensitive dermatitis and the excretion of large amounts of uroporphyrin in urine (Elder et al., 1980). De Verneuil et al. (1978) and others classified porphyria cutanea tarda, the most common type of porphyria, into 2 types: type I (176090), or 'sporadic' type, associated with approximately 50% level of uroporphyrinogen decarboxylase (UROD) in liver (Elder et al., 1978; Felsher et al., 1982), and type II, or 'familial' type, characterized by 50% deficient activity of the same enzyme in many tissues (Kushner et al., 1976; Elder et al., 1980). PCT type II is an autosomal dominant disorder with low penetrance and constitutes about 20% of cases of PCT.
    • Porphyria Cutanea Tarda GARD
      Porphyria cutanea tarda (PCT) is a form of porphyria that primarily affects the skin. People affected by this condition generally experience " photosensitivity ," which causes painful, blistering lesions to develop on sun-exposed areas of the skin (i.e. the hands and face). Skin in these areas may also be particularly fragile with blistering and/or peeling after minor trauma. In some cases, increased hair growth as well as darkening and thickening of the affected skin may occur. Liver abnormalities may develop in some people with the condition and PCT, in general, is associated with an increased risk of liver cirrhosis and liver cancer .
  • Lactose Intolerance Wikipedia
    However, if a "D" (for "dairy") is present next to the circled "K", "U", or other hechsher , the food product likely contains milk solids, [59] although it may also simply indicate the product was produced on equipment shared with other products containing milk derivatives. ... PMID 26715083 . ^ Suchy FJ, Brannon PM, Carpenter TO, Fernandez JR, Gilsanz V, Gould JB, Hall K, Hui SL, Lupton J, Mennella J, Miller NJ, Osganian SK, Sellmeyer DE, Wolf MA (February 2010). ... Retrieved 2018-02-08 . ^ a b c Schnorr SL, Sankaranarayanan K, Lewis CM, Warinner C (December 2016). ... PMID 30183735 . ^ Tishkoff SA, Reed FA, Ranciaro A, Voight BF, Babbitt CC, Silverman JS, Powell K, Mortensen HM, Hirbo JB, Osman M, Ibrahim M, Omar SA, Lema G, Nyambo TB, Ghori J, Bumpstead S, Pritchard JK, Wray GA, Deloukas P (January 2007). ... S2CID 33253438 . ^ Oh E, Jeremian R, Oh G, Groot D, Susic M, Lee K, et al. (January 2017). "Transcriptional heterogeneity in the lactase gene within cell-type is linked to the epigenome" .
    MCM6, LCT, GLB1, CFTR, SLC26A3, PTH, DDX43
  • Nystagmus Wikipedia
    February 27, 2013 . Retrieved 2012-12-12 . ^ Saladin K (2012). Anatomy and Physiology: The Unity of Form and Function . ... PMC 1722486 . PMID 9613375 . ^ Rowe FJ, Hanna K, Evans JR, Noonan CP, Garcia-Finana M, Dodridge CS, et al.
    ATXN2, CACNA1A, VLDLR, TDP1, APTX, WWOX, COQ2, ATXN10, PIK3R5, SETX, AFG3L2, STUB1, XPA, TBP, CACNB4, SPTBN2, SLC1A3, ATXN1, RFC1, PRKCG, MRE11, ATXN3, GRID2, FGF14, SPG11, LY6E
  • Graves' Ophthalmopathy Wikipedia
    ISSN 0003-9950 . PMID 6548373 . ^ Ohtsuka, K. (October 1997). "Intraocular pressure and proptosis in 95 patients with Graves ophthalmopathy". ... Endocrine Journal . 50 (5): 595–601. doi : 10.1507/endocrj.50.595 . PMID 14614216 . ^ Bhargav, P. R. K.; Sabaretnam, M.; Kumar, S. Chandra; Zwalitha, S.; Devi, N.
    PTGS2, SCD, CTLA4, IL1B, TSHR, IL6, TNF, IGF1R, IGF1, CD40, IFNG, IL1A, MIR146A, TPO, CD40LG, CD34, TG, PPARG, IL17A, CXCL8, IL1RN, MS4A1, IL10, CCL2, KRT20, PIK3CB, BCAR1, CSE1L, NR3C1, MBTPS1, TGFB1, SST, CTNND1, CXCL10, ICAM1, PIK3CA, LINC01193, PIK3CD, PIK3CG, TMX2-CTNND1, IL23R, CCN2, MIR155, SOAT1, ADIPOQ, LEP, CD80, CD28, CASQ1, CCN1, IL4, BCL2, IL2, IL5, IL6R, AHR, PARP1, IL12B, IL13, HLA-DRB1, CD44, FOXP3, MMP9, IL22, FAM155B, NFKB1, THY1, CDK5, MMP2, SERPINE1, HLA-A, FGF2, SPAM1, STAT1, S100A8, SSTR1, VDR, ZFP36, TLR4, ST8SIA1, CCL5, VCAM1, TIMP2, CXCL11, TYRP1, SFRP1, SELE, TIMP1, SSTR2, ABCF1, HMGA2, TNFSF13, MAVS, IL21, IFIH1, BOLL, PRR3, HAVCR2, TSLP, LACRT, RBM45, HT, SERPINA13P, MIR183, MIR21, MIR27A, MIR27B, MIR96, MTCO2P12, UACA, TLR9, SHC3, EBI3, IL18R1, ST3GAL5, SPHK1, SOCS3, RNH1, KNTC1, NAMPT, EFS, IL23A, TNFSF13B, PPP1R13L, SNED1, ACOT11, PTPN22, TLR7, TNFRSF12A, S100A4, MTHFR, RBP1, PTX3, CTNNB1, CYP1A1, DEFA3, NQO1, DIO2, S1PR1, FGF1, FGFR1, FOS, GABPA, GAPDH, GDF1, CBLIF, GNAS, GNL1, GSTP1, HAS2, CRP, CRABP1, COX8A, BMP7, ACTB, AKT1, ALDH2, AIRE, APRT, ARRB1, AZGP1, CASQ2, CCR6, CD19, CD86, CD68, CEBPB, CHUK, TPP1, CLN3, HLA-B, HLA-C, HLA-DOA, PDCD1, ACHE, MYC, MYD88, NEDD9, NFE2L2, NOTCH2, OMG, PIP, CXCL9, PPARA, PRKAA1, PRKAA2, PRKAB1, MAPK1, MAPK3, PTPN12, COX2, MFAP1, HSPA1A, IL7R, HSPA1B, HSPA4, IGFBP5, IKBKB, IL2RA, IL3, IL7, IL15, MBP, IL16, IL18, ISG20, ITGAE, LDLR, LTA, LYZ, LINC02605
  • Memory Disorder Wikipedia
    Retrieved March 11, 2010, from Heart and Stroke Foundation of Ontario : http://www.heartandstroke.on.ca/site/c.pvI3IeNWJwE/b.3581869/k.8BD1/Stroke__Effects_of_a_stroke.htm ^ Rising Tide: The Impact of Dementia on Canadian Society. (2010). ... PMID 19901352 . ^ a b c d e f g h i j k Popescu, Bogdan O.; Toescu, Emil C.; Popescu, Laurenţiu M.; Bajenaru, Ovidiu; Muresanu, Dafin F.; Schultzberg, Marianne; Bogdanovic, Nenad (2009). ... Ceci Susan Clancy Hermann Ebbinghaus Sigmund Freud Patricia Goldman-Rakic Jonathan Hancock Judith Lewis Herman HM (patient) Ivan Izquierdo Marcia K. Johnson Eric Kandel KC (patient) Elizabeth Loftus Geoffrey Loftus Chris Marker James McGaugh Paul R.
    APP, PSEN1, MAPT, C9orf72, PRNP, COX2, TLR4, ABCA7, CP, GRN, FAS, PRRT2, SCN2A, FMR1, IL10, PIK3CA, SCN8A, SORL1, ATXN1, SIM1, RPS20, IL1B, ALDH18A1, SLC20A2, HLA-DQB1, HLA-B, ACHE, MSH6, SPAST, SPG7, IL12A, VAMP1, TGFBR2, TYROBP, VCP, KLRC4, FGF14, SQSTM1, EIF2B4, EIF2B3, EIF2B2, STAT4, PRKCG, PSEN2, COX1, MYD88, TRNW, TRNS2, TRNS1, TRNQ, TRNL1, TRNH, TRNF, PAH, ND6, ND5, ND4, ND1, COX3, MSH2, PSAP, PDGFB, PDGFRB, MLH1, ATXN3, MEFV, PMS1, PMS2, POLG, EPCAM, KRAS, KCNQ3, KCNQ2, PRKAR1B, EIF2B1, EIF2B5, NOTCH3, FAN1, UBAC2, CSF1R, AFG3L2, IL23R, UGT1A1, VPS13A, APOE, CAMTA1, CCR1, ARSA, SEMA4A, XPR1, MYORG, CHMP2B, BDNF, MLH3, REEP2, JPH3, BMPR1A, TWNK, TMEM106B, ERAP1, ADA2, TREM2, ACSF3, C4A, IL12A-AS1, DNMT1, SLC25A13, TOMM40, ATN1, GNE, STUB1, ECM1, CASP3, TNF, BACE1, NFE2L2, GRIN2B, ESR1, GABPA, IL6, DLG4, CAT, PERCC1, SNCA, ADRA2B, HCRT, MCIDAS, GSK3B, MAPK1, MME, ACKR3, SUCLA2, APLN, LAMC2, EPHB2, MAPK8, CTNNB1, BCL2, CDH1, FNDC5, CSF2, GPX1, CREB1, CNR1, GFAP, S100A9, TRPV1, CHRNA4, AGT, PTGS2, CDK5, PTPN5, KIF17, MIR132, LPAR2, PVALB, POMC, PPARG, GRAP2, NGFR, KAT5, AHSA1, TAC1, CXCR6, SYP, OGA, SSTR4, CPLX1, KLK8, ST3GAL4, S100B, WWC1, RAC1, SIRT1, SMUG1, RNF19A, PRKCA, POLDIP2, ACTB, PPT1, PPP5C, AIMP2, MTCO2P12, NR3C1, FANCD2, GCG, AKT1, BRS3, MAPK14, CHAT, CTSB, AGTR1, GRM5, ADRA1A, GLUL, GPR42, HMGB1, BRCA2, NR3C2, CASP1, ESR2, EPO, IGF1, NGF, IFNG, CRK, EDNRA, IDE, HTR6, TARDBP, CRTC1, CHRNB2, SYNM, NPY, QPCT, CHI3L1, CADM1, CNR2, LRBA, ENTPD1, CASP6, SLC17A5, REM1, FLVCR1, CLU, TPPP, COMT, CYBB, CLOCK, ARHGAP32, HDAC4, SV2A, DPYSL2, CFD, IGSF6, ACE, DAPK1, RACK1, CYP19A1, CX3CR1, DKK1, ARID3B, CSN2, CPLX2, CSF3, NMU, PPARGC1A, BTG3, COPS5, PRLH, WDHD1, CPA1, KCNIP3, CAMK4, GAL, ADIPOR1, SLCO6A1, ALOX5, PPM1K, AKT2, OPN5, STH, AHR, NEAT1, AGER, GSTK1, MIR107, MIR134, MIR137, MIR146A, MIR155, MIR204, MIR23A, MIR27A, MIR34A, MIR34C, TRIM72, MIR383, MIR448, ZGLP1, ECT, PARP1, ADA, LRG1, APC, FTMT, APMAP, CAPN1, HOMER1, CALML5, CALB1, KCNK10, C5AR1, CCHCR1, C3AR1, KLF9, TMED9, PGPEP1, HEATR3, BMP1, PLXNA4, NDRG2, CCND1, BCHE, NLRC4, SNX6, ATP2A1, PINK1, EHMT1, RNF34, TMX1, TTBK1, ATF4, SLC25A27, SELENBP1, GSTO1, LCN2, PRKAA1, PRKAA2, PRKAB1, KCNK2, KCNA1, PRKCB, PRKCD, ITPR1, PRKG1, ITGB2, MAPK3, ITGAM, MAP2K7, EIF2AK2, IRS1, PROP1, PROS1, RELN, IRAK1, INSR, INPPL1, PTGS1, IL17A, CXCL8, IL4, LAG3, LEP, NTN1, LGALS1, NRGN, NT5E, NTF3, NUCB2, OPRM1, PAEP, RNR2, PAM, PCSK1, PDE1B, MS, PDK1, CFP, KMT2A, PIK3CB, PIK3CD, PIK3CG, PKM, PLA2G4A, PLAT, MECP2, MAP2, MAOB, LGALS3, PPARD, IL1A, RAP1GDS1, RARRES2, IGFBP7, TIAM1, TLR2, TRPM2, TTC3, TUBA4A, GLP1R, GJA1, VGF, WNT1, SLC30A3, GAD1, FYN, PLA2G6, PDE5A, BECN1, RIPK1, FKBP5, CDK5R1, PTK2B, EZH2, SLC29A1, EGR1, LGI1, ADIPOQ, DYRK1A, TH, GPT, TFAM, HNF4A, S100A1, SORT1, IFNB1, CCL3, CX3CL1, SDHD, SHBG, HTR2A, HRES1, SLC6A4, SLC8A3, SOD1, TERT, HDAC2, GSTT1, GSTM1, SPRR2A, GRP, STAT1, STAT3, CDKL5, GRM2, GRIA1, TCN1, PRCP
  • Excoriation Disorder Wikipedia
    PMID 19890232 . ^ a b c d e f g h i j k l m n o p q r s t u v w x y z aa ab ac ad ae af ag ah ai aj ak al am an ao ap aq ar as at au av Odlaug BL, Grant JE (September 2010). ... PMID 16960655 . ^ a b c d e f g h i j k l m n o p q r s t u Lang R, Didden R, Machalicek W, Rispoli M, Sigafoos J, Lancioni G, Mulloy A, Regester A, Pierce N, Kang S (2010).
    HTR2A, SLC6A4, KMT2E, NAA15, HPD, SMS, TBX4, SEPHS1, PDSS1, DLGAP3
  • Adenomyosis Wikipedia
    PMID 12351554 . ^ a b c d e f g h i j k l m n o p q r s t u v w x y Struble, Jennifer; Reid, Shannon; Bedaiwy, Mohamed A. (2016). ... PMID 16564227 . ^ Kitawaki J, Obayashi H, Ishihara H, Koshiba H, Kusuki I, Kado N, Tsukamoto K, Hasegawa G, Nakamura N, Honjo H (January 2001). ... PMID 19291813 . S2CID 9986561 . ^ Kepkep K, Tuncay YA, Göynümer G, Tutal E (2007). ... PMID 17659649 . S2CID 26333703 . ^ Sakhel K, Abuhamad A (May 2012). "Sonography of adenomyosis".
    MUC16, PGR, ESR1, PIK3CA, BCL2, MTCO2P12, PIK3CB, PTGS2, COX2, MMP9, IL10, CXCL8, IL6, VEGFA, PIK3CG, CYP19A1, PIK3CD, ANXA2, CRH, CCL2, TIMP2, COMT, CXCR4, HOXA10, STAT3, GPER1, MMP2, STIP1, TNF, F3, FGF2, CD68, CTNNB1, NOS3, PTEN, AMH, PAK1, CYP2B6, SYT1, TLR4, TGM2, PERCC1, TGFB1, ZEB1, TCF3, TBX1, RELA, MAPK1, ROCK1, MAPK3, PRL, SOD2, MIR21, SELL, MAP3K2-DT, CXCL12, LINC-ROR, SPP1, VWF, MIR17, IL37, CRB3, IL33, MUL1, IL17F, WNK1, IL25, GORASP1, LPAR5, GKN1, TUG1, NDUFA13, PDCD4, FMNL2, SIGLEC7, MIR10B, LPAR3, HECW1, PSIP1, TCFL5, PAK4, CBLL2, ROCK2, LPAR2, EBAG9, BECN1, KHSRP, IL31, TNFRSF1B, MED12, ACTA1, PECAM1, DNMT3A, MSTN, GALT, GABPA, FOXO1, FGF1, EZH2, EPHB4, EPHB2, EGR1, EFNB2, LPAR1, DUSP6, DNMT3B, CYP17A1, GSTM1, CYP1A1, CUX1, CCN2, CNR2, CGA, CEBPB, CDKN2A, CDK2, ENTPD2, CD34, CCK, RHOA, ALOX5, LPAR4, GSTM2, PRKN, LPA, PAPPA, NOTCH1, NINJ1, NHS, AKT1, NGF, NFE2L2, MSH2, MMP7, MMP1, MME, MLH1, MET, LIFR, HCLS1, LIF, LCN2, EIF3E, ILK, IL17A, IL2RB, IL1B, CCN1, IGFBP1, IFNG, HPRT1, HOXA11, HGF, NGFR
    • Adenomyosis Mayo Clinic
      Overview Adenomyosis (ad-uh-no-my-O-sis) occurs when the tissue that normally lines the uterus (endometrial tissue) grows into the muscular wall of the uterus. The displaced tissue continues to act normally — thickening, breaking down and bleeding — during each menstrual cycle. An enlarged uterus and painful, heavy periods can result. Doctors aren't sure what causes adenomyosis, but the disease usually resolves after menopause. For women who have severe discomfort from adenomyosis, hormonal treatments can help. Removal of the uterus (hysterectomy) cures adenomyosis Adenomyosis With adenomyosis, the same tissue that lines the uterus (endometrial tissue) is present within and grows into the muscular walls of your uterus.
    • Adenomyosis OMIM
      Description Adenomyosis is characterized by the presence of endometrial glands and stroma within the myometrium. Abnormal uterine bleeding and dysmenorrhea are the most characteristic symptoms, occurring in approximately 65% of cases (Arnold et al., 1995). Inheritance Emge (1962) noted a possible hereditary factor in the occurrence of adenomyosis because of his finding of 7 mother-daughter pairs who had undergone surgery for adenomyosis over a 15-year period. Arnold et al. (1995) reported a family with adenomyosis in 3 successive generations; 2 sisters, their mother, and their maternal grandmother were affected. Either autosomal or X-linked dominant inheritance is possible. GU - Adenomyosis Lab - Endometrial glands and stroma within the myometrium - Abnormal uterine bleeding - Dysmenorrhea Inheritance - Autosomal vs.
  • Hemophilia A GeneReviews
    Therapies under investigation : Ongoing clinical trials of longer-acting factor VIII concentrates, bypassing agents, and gene therapy. Other : Vitamin K does not prevent or control bleeding in hemophilia A; cryoprecipitate contains factor VIII but does not undergo viral inactivation so is no longer used to treat hemophilia A. ... Combined (multiple) deficiencies are usually acquired disorders, although a few families have hereditary deficits of the vitamin K-dependent factors, often resulting from deficiency of gamma-carboxylase. ... Search ClinicalTrials.gov in the US and EU Clinical Trials Register in Europe for access to information on clinical studies for a wide range of diseases and conditions. Other Vitamin K does not prevent or control bleeding in hemophilia A.
    F8, F9, TFPI, F10, AK3, F2, VWF, COX8A, ST14, F5, F3, PROC, IL10, CCR5, AAVS1, HLA-DRB1, TNF, MS4A1, CCND1, GP1BA, G6PD, LMAN1, RBM45, KRT20, ABO, CPB2, VIPR1, PROCR, ISG20, IFNA2, IL2, TGFB1, IL2RA, SELP, IL4, IFNL3, MAPK9, PF4, HAL, PROS1, FOXP3, STATH, PRNP, ADAMTS5, WDHD1, MTHFR, F7, AMBP, ABR, DMD, ALB, GH1, ARTN, DKK1, APOH, BPI, BRCA1, ABCB6, CD163, FCGR2C, TNFRSF11A, HAP1, TRBV16, IKBKG, SLC14A2, LAP, BTK, C4BPA, VEGFA, TPT1, C6, THBD, NT5C2, HFE, TRBV7-9, CALR, MIR1246, C20orf181, C4orf3, CA13, ASPG, AGRP, IL33, MCFD2, CARD14, FUNDC2, UPF3B, HPSE2, IMPACT, SCLY, SERPINA10, SOST, APC, ICOS, TRBC1, CA6, TRBV20OR9-2, PRDX2, FUT1, LTF, LRP1, F11, KRT7, KIT, FCGR2A, FCGR2B, CXCL8, IL6, UTS2R, HLA-A, IL1B, IFNG, GPT, ICAM1, HNF4A, HMOX1, GUSB, HLA-DRA, HLA-B, MBL2, MRC1, NFYA, NHS, TRB, TAL1, CD28, CD68, SPRR2A, SLC4A1, SELPLG, CLTC, CCL3L1, REST, PSMA7, CSF1R, CSF2, CTLA4, SERPINE2, DPP4, SERPINA5, SERPINE1, TNFRSF11B, MIR4521
    • Hemophilia A Orphanet
      Hemophilia A is the most common form of hemophilia (see this term) characterized by spontaneous or prolonged hemorrhages due to factor VIII deficiency. Epidemiology Prevalence is estimated at around 1 in 6,000 males. Hemophilia primarily affects males, but a symptomatic form of hemophilia A in female carriers (see this term) has also been described with a generally milder clinical picture. Clinical description In general, onset of the bleeding anomalies occurs when affected infants start to learn to walk. The severity of the clinical manifestations depends on the extent of the factor VIII deficiency. If the biological activity of factor VIII is below 1%, the hemophilia is severe and manifests as frequent spontaneous hemorrhage and abnormal bleeding as a result of minor injuries, or following surgery or tooth extraction (severe hemophilia A; see this term).
    • Moderately Severe Hemophilia A Orphanet
      Moderately severe hemophilia A is a form of hemophilia A (see this term) characterized by factor VIII deficiency leading to abnormal bleeding as a result of minor injuries, or following surgery or tooth extraction. Epidemiology Moderately severe hemophilia A accounts for around 20% of all cases of hemophilia A. Clinical description The biological activity of factor VIII is between 1% and 5%. Spontaneous hemorrhages are rare. Etiology The disorder is caused by mutations in the F8 gene (Xq28) encoding coagulation factor VIII. Genetic counseling Transmission is X-linked recessive.
    • Hemophilia A GARD
      Hemophilia A is an inherited bleeding disorder in which the blood does not clot normally. People with hemophilia A will bleed more than normal after an injury, surgery, or dental procedure. This disorder can be severe, moderate, or mild. In severe cases, heavy bleeding occurs after minor injury or even when there is no injury (spontaneous bleeding). Bleeding into the joints, muscles, brain, or organs can cause pain and other serious complications. In milder forms, there is no spontaneous bleeding, and the disorder might only be diagnosed after a surgery or serious injury.
    • Haemophilia A Wikipedia
      . ^ Rangarajan, Savita; Walsh, Liron; Lester, Will; Perry, David; Madan, Bella; Laffan, Michael; Yu, Hua; Vettermann, Christian; Pierce, Glenn F.; Wong, Wing Y.; Pasi, K. John (16 December 2017). "AAV5–Factor VIII Gene Transfer in Severe Hemophilia A" .
    • Symptomatic Form Of Hemophilia A In Female Carriers Orphanet
      Symptomatic hemophilia A in female carriers is a form of hemophilia A (see this term) that manifests in some women with mutations in the F8 gene (Xq28), encoding coagulation factor VIII. Epidemiology Prevalence is unknown but this form of hemophilia is very rare. Clinical description Symptoms include abnormal bleeding as a result of minor injuries, or following surgery or tooth extraction. Spontaneous hemorrhages may occur occasionally. Genetic counseling Transmission is X-linked recessive.
    • Hemophilia MedlinePlus
      Hemophilia is a bleeding disorder that slows the blood clotting process . People with this condition experience prolonged bleeding or oozing following an injury, surgery, or having a tooth pulled. In severe cases of hemophilia, continuous bleeding occurs after minor trauma or even in the absence of injury (spontaneous bleeding). Serious complications can result from bleeding into the joints, muscles, brain, or other internal organs. Milder forms of hemophilia do not necessarily involve spontaneous bleeding, and the condition may not become apparent until abnormal bleeding occurs following surgery or a serious injury.
  • Alveolar Capillary Dysplasia Wikipedia
    It is likely an under-recognized cause of death shortly after birth because diagnosis requires microscopic examination of lung tissue or specialized genetic testing, or death can be attributed to severe heart or intestinal congenital abnormalities which frequently occur in ACD. [1] History [ edit ] Congenital alveolar dysplasia was first described by MacMahon in 1948. [7] [8] The seminal case first describing ACD was by Janney and colleagues in 1981. [5] Transmission from a carrier parent to a child was first reported in 1994. [9] References [ edit ] ^ a b c d e f g h i j k l m n o p q r s t u v Bishop, Naomi B.; Stankiewicz, Pawel; Steinhorn, Robin H. (15 July 2011).
    FOXF1, STRA6, ABCA3, FOXC2, SFTPB, STAT3, ANKRD11
    • Congenital Alveolar Capillary Dysplasia Orphanet
      Congenital alveolar capillary dysplasia (ACD) is a rare and fatal developmental lung disease characterized by respiratory distress in neonates due to refractory hypoxemia and severe pulmonary arterial hypertension.
    • Alveolar Capillary Dysplasia With Misalignment Of Pulmonary Veins OMIM
      A number sign (#) is used with this entry because of evidence that congenital alveolar capillary dysplasia with misalignment of pulmonary veins (ACDMPV) is caused by heterozygous mutation in the FOXF1 gene (601089) on chromosome 16q24. Description Congenital alveolar capillary dysplasia with misalignment of pulmonary veins (ACDMPV) is characterized histologically by failure of formation and ingrowth of alveolar capillaries that then do not make contact with alveolar epithelium, medial muscular thickening of small pulmonary arterioles with muscularization of the intraacinar arterioles, thickened alveolar walls, and anomalously situated pulmonary veins running alongside pulmonary arterioles and sharing the same adventitial sheath. Less common features include a reduced number of alveoli and a patchy distribution of the histopathologic changes. The disorder is associated with persistent pulmonary hypertension of the neonate and shows varying degrees of lability and severity (Boggs et al., 1994). Affected infants present with respiratory distress resulting from pulmonary hypertension in the early postnatal period, and the disease is uniformly fatal within the newborn period (Vassal et al., 1998).
  • Gunther Disease Wikipedia
    Hematology . 2012 : 19–27. doi : 10.1182/asheducation.v2012.1.19.3795678 . PMID 23233556 . ^ De, AK; Das, K; Sil, A; Joardar, S (Sep 2013). "A Case of Congenital Erythropoietic Porphyria without Hemolysis" .
    UROS, GATA1, HMBS
  • Freckle Wikipedia
    Philadelphia: Elsevier Saunders. p. 1232. ISBN 0-8089-2302-1 . ^ Hanson, K. M; Gratton, E; Bardeen, C. J (2006).
    ALDH2, GPR143, PPP1CB, PRKAR1A, PTPN11, RAF1, MAP3K7, TERT, TRIO, TYR, TYRP1, XPA, XPC, BAP1, TP63, ABCB6, POT1, SOX18, TERF2IP, BNC2, UVSSA, OCA2, NF1, BRAF, NBN, CDH3, CDK4, CDKN2A, CDKN2B, ERCC8, DDB2, ERCC2, ERCC3, ERCC4, ERCC5, ERCC6, FANCA, HPS1, IRF4, EIF6, KIT, MC1R, MGMT, MITF, ACD
    • Melanosis Wikipedia
      Melanosis Specialty Dermatology Melanosis is a form of hyperpigmentation associated with increased melanin . [1] It can also refer to: Melanism Ocular melanosis Smoker's melanosis Oral melanosis Riehl melanosis See also [ edit ] Look up melanosis in Wiktionary, the free dictionary. List of cutaneous conditions References [ edit ] ^ Melanosis at the US National Library of Medicine Medical Subject Headings (MeSH) External links [ edit ] Classification D MeSH : D008548 v t e Pigmentation disorders / Dyschromia Hypo- / leucism Loss of melanocytes Vitiligo Quadrichrome vitiligo Vitiligo ponctué Syndromic Alezzandrini syndrome Vogt–Koyanagi–Harada syndrome Melanocyte development Piebaldism Waardenburg syndrome Tietz syndrome Loss of melanin / amelanism Albinism Oculocutaneous albinism Ocular albinism Melanosome transfer Hermansky–Pudlak syndrome Chédiak–Higashi syndrome Griscelli syndrome Elejalde syndrome Griscelli syndrome type 2 Griscelli syndrome type 3 Other Cross syndrome ABCD syndrome Albinism–deafness syndrome Idiopathic guttate hypomelanosis Phylloid hypomelanosis Progressive macular hypomelanosis Leukoderma w/o hypomelanosis Vasospastic macule Woronoff's ring Nevus anemicus Ungrouped Nevus depigmentosus Postinflammatory hypopigmentation Pityriasis alba Vagabond's leukomelanoderma Yemenite deaf-blind hypopigmentation syndrome Wende–Bauckus syndrome Hyper- Melanin / Melanosis / Melanism Reticulated Dermatopathia pigmentosa reticularis Pigmentatio reticularis faciei et colli Reticulate acropigmentation of Kitamura Reticular pigmented anomaly of the flexures Naegeli–Franceschetti–Jadassohn syndrome Dyskeratosis congenita X-linked reticulate pigmentary disorder Galli–Galli disease Revesz syndrome Diffuse/ circumscribed Lentigo / Lentiginosis : Lentigo simplex Liver spot Centrofacial lentiginosis Generalized lentiginosis Inherited patterned lentiginosis in black persons Ink spot lentigo Lentigo maligna Mucosal lentigines Partial unilateral lentiginosis PUVA lentigines Melasma Erythema dyschromicum perstans Lichen planus pigmentosus Café au lait spot Poikiloderma ( Poikiloderma of Civatte Poikiloderma vasculare atrophicans ) Riehl melanosis Linear Incontinentia pigmenti Scratch dermatitis Shiitake mushroom dermatitis Other/ ungrouped Acanthosis nigricans Freckle Familial progressive hyperpigmentation Pallister–Killian syndrome Periorbital hyperpigmentation Photoleukomelanodermatitis of Kobori Postinflammatory hyperpigmentation Transient neonatal pustular melanosis Other pigments Iron Hemochromatosis Iron metallic discoloration Pigmented purpuric dermatosis Schamberg disease Majocchi's disease Gougerot–Blum syndrome Doucas and Kapetanakis pigmented purpura / Eczematid-like purpura of Doucas and Kapetanakis Lichen aureus Angioma serpiginosum Hemosiderin hyperpigmentation Other metals Argyria Chrysiasis Arsenic poisoning Lead poisoning Titanium metallic discoloration Other Carotenosis Tar melanosis Dyschromia Dyschromatosis symmetrica hereditaria Dyschromatosis universalis hereditaria See also Skin color Skin whitening Tanning Sunless Tattoo removal Depigmentation This dermatology article is a stub . You can help Wikipedia by expanding it . v t e
  • Aluminium Phosphide Poisoning Wikipedia
    . ^ a b Gurjar, Mohan; Baronia, Arvind K; Azim, Afzal; Sharma, Kalpana (2011).
  • Alcohol And Weight Wikipedia
    PMID 7977278 . Männistö S, Uusitalo K, Roos E, Fogelholm M, Pietinen P (May 1997).
  • Trophoblastic Neoplasm Wikipedia
    .; Xiang, Yang; Golfier, François; Sekharan, Paradan K.; Lurain, John R.; Massuger, Leon (October 2018).
    CD274, TP53, LGALS1, EGF, RASA2, DKK1, HSPB3, VIM, TNF, CCL2, SALL1, RASA1, GH2, PDCD1, CITED1, KRT7, INHA, IDO1, HSPB2, HSPB1, HSD3B1, GAL
  • Arthritis Mutilans Wikipedia
    . ^ a b c d Rodgers, M; Epstein, D; Bojke, L; Yang, H; Craig, D; Fonseca, T; Myers, L; Bruce, I; Chalmers, R; Bujkiewicz, S; Lai, M; Cooper, N; Abrams, K; Spiegelhalter, D; Sutton, A; Sculpher, M; Woolacott, N (February 2011).
  • Hypernatremia Wikipedia
    ., chlorthalidone ) in congestive heart failure or corticosteroids in nephropathy also can be used. [19] See also [ edit ] Hyponatremia Salt poisoning References [ edit ] ^ a b c d e f g h i j k l m n o p Reynolds, RM; Padfield, PL; Seckl, JR (25 March 2006).
    POMC, AQP2, FGFR1, ARNT2, MAGEL2, AVPR2, DSG1, REN, AGT, HSD11B2, AVP, NR3C2, RNU1-4, AGTR1, SGK1, PLG, SLC5A2, SLC12A3, LEP, ATM, SLC33A1, PGR-AS1, PLAU, SLC12A1, FGF23, SLC9A3, SLC9A1, ADD1, SLC12A2, ZIC2, NPHS2, CCL4, KL, ATP6AP2, EGLN1, MIR429, SDC1, OPRM1, SCNN1A, GH1, AQP3, CASR, CRH, CRP, ACE, DRD1, EDN1, EDNRA, GHSR, ACSM3, HIF1A, IL17A, KLKB1, LRP2, MME, COX2, ADM, PTGS2, MTCO2P12
  • Obstetrical Bleeding Wikipedia
    . ^ a b patient.info » PatientPlus » Antepartum Haemorrhage Last Updated: 5 May 2009 ^ The Royal Women’s Hospital > antepartum haemorrhage Archived 2010-01-08 at the Wayback Machine Retrieved on Jan 13, 2009 ^ Soyama H, Miyamoto M, Ishibashi H, Takano M, Sasa H, Furuya K (2016). "Relation between Birth Weight and Intraoperative Hemorrhage during Cesarean Section in Pregnancy with Placenta Previa" .
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